1. “Changes in the urinary sediment” Differential diagnosis of nephropathy (pregnant, diabetic, drug) The tactics of the GP

2. The purpose of the training session:
GP various diseases associated with nefropatiyalarni (NP) at the time of their diagnosis and differential diagnosis of issues, choosing the optimal treatment strategy in patients and health care "General Practitioner qualification characteristics" in accordance with the principles of training.

3. Pedagogical task:
compatible with the NP and rural GPs (OP) or specialized outpatient treatment of diseases that can be dealt with.
NP accompanied by GPs: pregnant women, how to diagnose diabetic drug nefropatiyalarni.
GP qualifying characteristics are discussed within the framework of tactics.
The discussion of the principles of the treatment of this disease (unmedication and medication).
In the context of rural or OP to discuss the principles of observation and monitoring of patients.
these diseases in primary, secondary and tertiary prevention principles discussed.

4. GP need to know:
Diabetes, pregnancy, drug mechanism of NP damage can occur.
NP differential diagnostics of diseases associated with
Cornea.
The principles of treatment of these diseases (medication and unmedication).
In the context of rural or OP dispensary principles of supervision and monitoring of patients.
these diseases in primary, secondary and tertiary prevention principles.

5. GP should be able to:
NP associated with these diseases clinic and laboratory-instrumental investigation on the basis of the diagnosis, differentsiallashni.
proven drug selection.
Unmedication treatment counseling.
monitoring the conditions of rural or OP.

6. Diabetic nephropathic (in the form of diabetic glomerulosclerosis) is observed in type I and II diabetes, proteinuria, polyneuropathy, micro - and makroangiopatiya characterized. Renal vein thrombosis in patients with lumbar intense pain, they proteinuria, as well as eritrotsituriya (makrogematuriya level), oliguria, anuria determined by the ARD.

7. Diabetes mellitus
10-12 years glomerulosclerosis develop the disease. His first koptokchalar PU percent of the time. PU initial period, as always, will be further reduced level of selectivity
KF 45 ml / min decreased to koptokchali and kanalchali PU and observed in 60-90% of patients with AG
NS appears to be the development of atherosclerosis, the addition of a secondary infection can lead to the development of diabetic nefropatiyalarda SBE
The occurrence of persistent proteinuria koptokchalar filtration monthly decline in a minute, 1 ml.ga.

8. Kanalchali proteinuria
Genetic tubulopatiya
necrosis of the sharp kanalchalar
interstitial nephritis,
pyelonephritis,
transplantatni chronic power
metals (lead, cadmium, bismuth, rtut) contamination by salts,
some of the toxic effects of drugs.
Enough protein in the proximal part of the absorbed
 Saturated with congenital and acquired as a result of the enzyme deficiency Caudan protein
2 g per day,
Ingredients: albumin, 2 -mikroglobulin, lysozyme, immunoglobulin light chains
Kanalchali proteinuria

9. Fankoni syndrome - the proximal common types of tubulopatiya
Proximal renal kanaltsevvoy acidosis
 Bikorbonaturiya
 Tubular type of proteinuria,
 Renal glycosuria
 Fosfaturiya
 polyuria
 Osteomalyatsiya

10. Primary Fankoni syndrome pedigree disease
Secondary Fankoni syndrome amyloidosis, myelin disease giperparatireoz and other diseases as a result of the development.
Clinic: physically and mentally stunted growth colitis, hypotrophy, bones, joints, bones, rickets and specific changes in appearance, polyuria, polydipsia, glycosuria.
Clinic: PU, skeletal deformation, pain in the bones, they patolgik fracture, osteoporosis, polyuria, thirst, hypokalemia miasteniya.

11. Postrenal proteinuria
proteinuria with protein-rich inflammatory exudation with urine, urinary tract infections and inflammatory disease (prostatitis), depletion of sperm, urine appears at the long-standing
less than 1, 0 g per day

12. PROTEINURIA
Selective proteinuria, which has a small molecular weight proteins, primarily albumin determined
Non-selective proteinuria medium and high molecular weight protein seperation
Histological examination of the kidney with minimal changes
Kidney ductal heavy damage in some cases, changes occur in fibroblasts