APPROACH TO WIDE QRS COMPLEX TACHYCARDIA

Slides:



Advertisements
Similar presentations
Cardiac Arrhythmias A Guide For Medical Students
Advertisements

Interesting Case Rounds
Texas Tech University Health Sciences Center
Differential Diagnosis of Wide QRS Complex Tachycardia
Differential Diagnosis of Tachycardias
Differential diagnosis of broad complex tachycardia
Management of the Patient Presenting with Wide Complex Tachycardia
ECG TRAINING MODULE 4 BY BRAD CHAPMAN RCT.
Jason Ryan, MD Intern Report
By Dr.Ahmed Mostafa Assist. Prof. of anesthesia & I.C.U.
UNC Emergency Medicine Medical Student Lecture Series
Chapter Page, 12-Lead ECG for Acute and Critical Care Providers © 2006 by Pearson Education, Inc. Upper Saddle River, NJ 6 Wide Complex Tachycardia.
ECG in Ventricular arrhythmias
Management of Supraventricular Tachycardias
European Resuscitation Council
DIFFERENTIAL DIAGNOSIS OF WIDE COMPLEX TACHYCARDIA
Upper Level Conference UNC Internal Medicine 10/21/09
Supraventricular Tachycardia: Making the diagnosis
What to do if called for an arrhythmia
Cardiac Arrhythmia. Cardiac Arrhythmia Definition: The pumping action of the heart is coordinated by an electrical system within the heart tissue.
Arrhythmias: The Good, the Bad and the Ugly
WIDE QRS TACHYCARDIA - BEDSIDE DIAGNOSIS
Bradycardia & Tachycardia
WIDE COMPLEX TACHYCARDIA
Jay Green Emergency Medicine Resident, PGY-3 July 24, 2008.
Arrhythmias Principles of long and short term management of arrythmias.
WIDE COMPLEX TACHYCARDIA
Pediatric Dysrhythmias Board Review
Ventricular Arrhythmias Terry White, RN, EMT-P. Analyze the Rhythm.
Arrhythmias Medical Student Teaching Tuesday 24 th January 2012 Dr Karen Jones, SpR Emergency Medicine.
Arrhythmia recognition and treatment
Atrial & Junctional Dysrhythmias
Cardiac Arrhythmias A Guide For Medical Students
Bradycardia Risk of asystole? History of asystole Mobitz II AV block Any pause  3 s Complete heart block, wide QRS Adverse signs? Clinical evidence of.
Supraventricular Arrhythmias Claire B. Hunter, M.D.
Leonard Steinberg, MD Timothy Knilans, MD The Heart Center Children’s Hospital Medical Center Cincinnati, OH The diagnosis and management of supraventricular.
EKG Interpretation: Arrhythmias Mustafa Salehmohamed, D.O. Assistant Clinical Instructor Department of Medicine N.Y. College of Osteopathic Medicine October.
Good Morning 20 August Anesthetic Considerations in Patients With Cardiac Arrhythmias 麻醉科 林子富.
By Dr. Zahoor CARDIAC ARRHYTHMIA.
Tachyarrhythmia Gaurav Panchal. Arrhythmogenesis Impulse formation –Automaticity – inappropriate Tachy / brady; accelerated Ventricular rate after MI.
ADVANCED CONCEPTS IN EMERGENCY CARE (EMS 483)
WIDE COMPLEX TACHYCARDIA Puja Chopra, PGY-1 Emergency Medicine May 19, 2011.
Tachyarrhythmia, Cardioversion and Drugs. Learning outcomes At the end of this workshop you should: Be able to recognise types of tachyarrythmia, defined.
SCN EKG Review and Strip
2  Unstable :  Altered mental status  Ischemic chest discomfort  Acute heart failure  Hypotension  Other signs of shock  Symptomatic:  Palpitations.
Arrhythmias.
1 Case 9 Stable Tachycardias © 2001 American Heart Association.
Thank you for this difficult ECG
Tachyarrhythmia Approach to management: 1. Determine if there is a pulse – If no pulse, initiate management for pulseless arrest 2. Determine if patient.
The normal ECG. Normal sinus rhythm –Each p wave followed by a QRS –Normal P waves –P wave rate bpm.
ARRHYTHMIA Objectives At the end of this session students should be able to:  Distinguish the normal from abnormal rhythms.  Understand the pathophysiologic.
Date of download: 5/27/2016 Copyright © The American College of Cardiology. All rights reserved. From: ACC/AHA/ESC guidelines for the management of patients.
IN THE NAME OFGODIN THE NAME OFGOD SVTS.SAYAH.  All cardiac tachyarrhythmias are produced by: 1/disorders of impulse initiation :automatic 2/abnormalities.
ARRHYTHMIAS Jamil Mayet. Arrhythmias - learning objectives –Mechanisms of action of antiarrhythmic drugs –Diagnosis To differentiate the different types.
Palpitations and Common Arrhythmias J. Philip Saul, M.D. West Virginia University Morgantown, WV.
Pediatric emergency case conference Presented by R3 李智晃.
EKG REVIEW Dr. Srikanth Seethala MD,MPH. RBBB: 1.QRS duration more than 120 msec 2.rsr′, rsR′, or rSR′ in leads V1 or V2. The R′ or r′ deflection.
Tachykardie / bradykardie
Assessing and treating tachyarrhythmias Workshop
المحتوى غير شامل لكل ما تحتويه المحاضرات
Supra Ventricular Tachycardia (SVT)
David Sanders Mini-Lecture 2017
Differential Diagnosis of Wide QRS Complex Tachycardia
Differential Diagnosis of Wide Complex Tachycardias
ARRHYTHMIA DR MANSOUR ALQURASHI
Narrow complex tachycardia
Broad complex tachycardia
Interventional cardiologist & internist
Terrifying Tachycardias
Presentation transcript:

APPROACH TO WIDE QRS COMPLEX TACHYCARDIA Dr HA TUAN KHANH Dr DAVID TRAN

Content Definition Causes of WCT Diagnosis criteria Clinical history Physical examination ECG criteria: Brugada criteria, other criteria, findings favoring SVT, VT vs AVRT criteria Management Unstable hemodynamic Stable hemodynamic

Stewart RB. Ann Intern Med 1986 Definition Wide QRS complex tachycardia is a rhythm with a rate of more than 100 b/m and QRS duration of more than 120 ms SVT (20%) VT (80%) Stewart RB. Ann Intern Med 1986

Causes of wide QRS complex tachycardia Supraventricular tachycardia - with prexsisting BBB - with BBB due to heart rate (aberrant conduction) - antidromic tachycardia in WPW syndrome Ventricular tachycardia

SVT vs VT Clinical history Age - ≥ 35 ys → VT (positive predictive value of 85%) Underlying heart disease Previous MI → 98% VT Pacemakers or ICD Increased risk of ventricular tachyarrhythmia Medication Drug-induced tachycardia → Torsade de pointes Diuretics Digoxin-induced arrhythmia → [digoxin] ≥2ng/l or normal if hypokalemia

SVT vs VT Physical examination Physical findings that indicate presence of AV dissociation (cannon A waves, variable-intensity S1,variation in BP unrelated to respiration) if present are useful Termination of WCT in response to maneuvers like Valsalva, carotid sinus pressure, or adenosine is strongly in-favor of SVT but there are well-documented cases of VT responsive to these

SVT vs VT ECG criteria: Brugada algorithm Brugada P. Ciculation 1991

Step 1

Step 2

Step 3

Step 4: LBBB - type wide QRS complex SVT VT R wave >40ms small R wave notching of S wave V1 fast downslope of S wave > 70ms V6 Q wave no Q wave

Step 4: RBBB - type wide QRS complex SVT VT rSR’ configuration monophasic R wave qR (or Rs) complex V1 or R/S > 1 R/S ratio < 1 QS complex V6 or

Step 4: RBBB morphology

Step 4: LBBB morphology

Other ECG criteria North - west QRS axis deviation Negative or positive concordance Fusion beats, capture beats Ventriculoatrial conduction with block RBBB morphology with LAD > - 300 LBBB morphology with RAD > + 900 Previous ECG show MI or previous ECG show that during sinus rhythm, bifascular block is present, which changes in configuration during tachycardia

Concordance and Northwest Axis

Fusion beat and capture beat

Ventriculoatrial conduction with block

RBBB morphology with LAD

LBBB morphology with RAD

Previous MI

Previous LBBB

Findings favoring SVT Triphasic pattern in V1 and V6 Rabbit’s ear Previous ECG: Preexistent BBB or preexcitation

Triphasic pattern

Rabbit’s ear

Wide complex SVT from preexisting RBBB

Wide complex SVT from preexisting LBBB

VT vs AVRT ECG criteria Brugada P. Ciculation 1991

Wide complex SVT from bypass tract

Summary : diagnosis evaluation ACC/AHA/ESC guidelines for management of pt with SVT. Circulation 2003

Management – Hemodynamic compromise Unstable patient, but still responsible with a discernible BP and/or pulse: - Emergent synchronized cardioversion - If the QRS complex and T wave cannot be distinguished accurately → immediate defibrillation Unstable patient, unresponsive or pulseless → standard ACLS resusciation algorithms

ACLS pulseless arrest algorithm AHA Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. Ciurculation 2005

Management – Stable hemodynamic VT or WCT of uncertain etiology: Any associated conditions (cardiac ischemia, heart failure, electrolyte abnormalities or drug toxicities) Class I and III antiarrhythmic drugs - Amiodarone: 150mg IV/10mins followed by an infusion of 1mg/min for 6 hours, then 0,5mg/min - Procainamide: 15-18mg/kg infusion over 25-30mins, followed by 1-4mg/min by continuous infusion - Lidocaine: 1-1,5mg/kg IV/2-3mins followed by an infusion of 1-4mg/min Urgent or elective cardioversion

Management – Stable hemodynamic SVT Vagal maneuvers: carotid sinus pressure (if no carotid bruits) or Valsava maneuver Adenosine: 6mg over 1-2 seconds. If the initial dose is ineffective, a 12mg dose may be given and repeated once if necessary Calcium channel blocker (Verapamil 2.5 to 5mg IV) or beta blokers (Metoprolol 5 to 10 mg IV) Cardioversion

Acute management hemodynamically stable and regular tachycardia ACC/AHA/ESC guidelines for management of pt with SVT. Circulation 2003

ACC/AHA/ESC guidelines for management of pt with SVT. Circulation 2003 Recommendation acute management hemodynamically stable and regular tachycardia ACC/AHA/ESC guidelines for management of pt with SVT. Circulation 2003

Tachycardia algorithm AHA Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. Ciurculation 2005

Tachycardia algorithm

Thank you for your attention