Acute Non-Q Wave Cocaine-related Myocardial Infarction Warren A. Kossowsky, M.D., Alan F. Lyon, M.D., Shyan-Yih Chou, M.D. CHEST Volume 96, Issue 3, Pages 617-621 (September 1989) DOI: 10.1378/chest.96.3.617 Copyright © 1989 The American College of Chest Physicians Terms and Conditions
Figure 1 Serial ECG tracings in a typical patient (case 13) taken on entrance to the emergency room (A), 21 h later (B), and 3 days later (C). The deep, symmetric T-wave inversion shown in (C) persisted beyond the 7th hospital day. CHEST 1989 96, 617-621DOI: (10.1378/chest.96.3.617) Copyright © 1989 The American College of Chest Physicians Terms and Conditions
Figure 2 Electrocardiogram in case 7 taken during an episode of chest pain after cocaine snorting, demonstrating ST-segment elevation in leads 1, 2, 3, aVL, aVF, and V2-6. CHEST 1989 96, 617-621DOI: (10.1378/chest.96.3.617) Copyright © 1989 The American College of Chest Physicians Terms and Conditions
Figure 3 ECG in the same patient taken 1 h later, when the chest pain subsided, demonstrating reversion of the ST-segment. CHEST 1989 96, 617-621DOI: (10.1378/chest.96.3.617) Copyright © 1989 The American College of Chest Physicians Terms and Conditions
Figure 4 ecg in the same patient taken one month later, displaying the normal variant of early repolarization. CHEST 1989 96, 617-621DOI: (10.1378/chest.96.3.617) Copyright © 1989 The American College of Chest Physicians Terms and Conditions