sub 1 유전자를 벼에 넣었을 때의 효과 (127일을 40초 동안 압축)

황금쌀(Golden rice) 눈에 매우 좋은 성분

황금쌀 표현형을 이용한 생명공학 기술 선진화 (성과) 관련 기술의 지적재산권 확보: 국내 특허 등록 및 국제 특허 출원

다중유전자 동시발현 신기술로 베타카로틴 생성 황금쌀 개발 “07국가연구우수성과 100선에 선정” 다중유전자 동시발현 신기술로 베타카로틴 생성 황금쌀 개발 (농촌진흥청 국립농업과학원 하선화 박사) (성과) 관련 기술의 지적재산권 확보: 국내 특허 2건 및 국제 PCT 출원 1건 4

β-PAC 도입된 콩 종자 수확 (T2종자) (동아대 정영수) Wild type (광안콩) line7 line13 line16 E.V. Wild type (광안콩) line13 동아대학교 온실에서 수확한 T2종자 Line #7-4 #7-5 #13-2 #13-5 #13-6 #13-7 #13-9 #13-10 #16-1 #16-2 #16-3 #16-4 #16-5 Pink 20 16 30 18 3 15 25 4 24 62 38 yellow 45 63 48 58 22 29 35 1 121 합계 65 79 78 76 44 53 5 159

Quantitative traits Quantitative traits refer to phenotypes (characteristics) that vary in degree and can be attributed to polygenic effects, i.e., product of two or more genes, and their environment. Quantitative trait loci (QTLs) are stretches of DNA containing or linked to the genes that underlie a quantitative trait. Mapping regions of the genome that contain genes involved in specifying a quantitative trait is done using molecular tags such as AFLP or, more commonly SNPs . This is an early step in identifying and sequencing the actual genes underlying trait variation.

Quantitative traits Polygenic inheritance, also known as quantitative or multifactorial inheritance refers to inheritance of a phenotypic characteristic (trait) that is attributable to two or more genes, or the interaction with the environment, or both. Unlike monogenic traits, polygenic traits do not follow patterns of Mendelian inheritance (separated traits). Instead, their phenotypes typically vary along a continuous gradient depicted by a bell curve.[1]

Multifactorial traits in general Generally, multifactorial traits outside of illness contribute to what we see as continuous characteristics in organisms, such as plant height[5], and weight[7]. All of these phenotypes are complicated by a great deal of interplay between genes and environment[5]. The continuous distribution of traits such as height and weight described above reflects the action of genes that do not quite show typical patterns of dominance and recessiveness. Instead the contributions of each involved locus are thought to be additive. Writers have distinguished this kind of inheritance as polygenic, or quantitative inheritance[8]. Thus, due to the nature of polygenic traits, inheritance will not follow the same pattern as a simple monohybrid or dihybrid cross[6]. Polygenic inheritance can be explained as Mendelian inheritance at many loci[5], resulting in a trait which is normally-distributed. If n is the number of involved loci, then the coefficients of the binomial expansion of (a + b)2n will give the frequency of distribution of all n allele combinations. For a sufficiently high n, this binomial distribution will begin to resemble a normal distribution. From this viewpoint, a disease state will become apparent at one of the tails of the distribution, past some threshold value. Disease states of increasing severity will be expected the further one goes past the threshold and away from the mean[8].