Ppt on atrial septal defect in infants

Congenital Heart Defects Fred Hill, MA, RRT. Categories of Heart Defects Left-to-right shunt Cyanotic heart defects Obstructive heart defects.

) Fail to gain weight  thin, large stomachs Develop pulm edema & die L  R: Patent Ductus Arteriosus Common in premature infants < 33 weeks Medical management –First 48 hours Fluid restriction Lasix (furosemide) –Indocin (indomethacin) –NeoProfen (ibuprofen lysine) Surgical: ligation of ductus L  R: Endocardial cushion defect Atrial & ventricular septal defect Common A-V valve Large L  R shunt Operation at 8 weeks 50% of Down’s babies/


Embryology Review.

Tx: Holes in the heart=surgery Tetralogy of Fallot= actually 5 things: VSD, PDA, pulmonary stenosis, RV hypertrophy, overriding aorta Cardiovascular Defects ASD (Atrial-septal defect) If foramen /infant (2-6w) with non-bilious post prandial vomiting, failure to thrive Dx: palpable olive-sized mass in the epigastric region Increased prevalence in males, non-blacks (dominant polygenic inheritance) GI defects II Annular pancreas: wraps duodenum, results from bilobed ventral pancreatic bulb Results in/


Atrial septal defects David M. Chaky, MD. Terminology ► ASD = defect in the atrial septum of the heart which can be isolated anomaly or associated with.

Atrial septal defects David M. Chaky, MD Terminology ► ASD = defect in the atrial septum of the heart which can be isolated anomaly or associated with other congenital heart lesions ► 10% of congenital heart lesions in children yet 30% of congenital heart lesions in adults Types of ASD ► Foramen ovale is nl interatrial communication that in utero allows flow from IVC to enter the left atrium ► Secundum ASD is oval defect bordered/


Ostium Secundum Atrial Septal Defect Closure Thomas Hoy February 11, 2004 Department of Biomedical Engineering BME 272 Senior Design.

disease causes left to right shunting across interatrial septum Why should we care? Why should we care? Isolated secundum atrial septal defects of significance occur in approximately 70,000-100,000 individuals today. OSASD constitutes approx. 7-10% of all cases of CHD. 32,000 infants (one out of every 125 to 150) Current Status Have updated website Have updated website http://vubme.vuse.vanderbilt/


EFFECT OF LOW LEVEL LASER ON HEALING OF MODERATE SIZED INDUCED SEPTAL DEFECTS ON RABBITS By Hady Atef Labib Mohamed.

of babies with a CHD have a critical CHD. Infants with critical CHDs generally need surgery or other procedures in their first year of life (Schelbert, 2010). How are pediatric congenital heart defects treated? There are two main options: treatment with a/study aimed to find an alternative or even adjunctive non surgical method for some congenital heart defects like septal defects whether it is ventricular or atrial, in order to avoid or decrease -at least- open heart surgeries complications, which is still /


Blood and Blood Vessels

disease Second pregnancy with Rh– mother and Rh+ infant Subsequent pregnancy with Rh+ infant can allow maternal anti-Rh antibodies to cross placental /2 108 CLINICAL MODULE 17.21: Fetal circulation and defects Congenital cardiac defects Ventricular septal defects Openings in interventricular septum Patent foramen ovale Passageway remains open Left/18.6 1 130 Module 18.6: Heart valves Valve action during atrial relaxation and ventricular contraction AV valves Closed Blood pressure from contracting ventricles /


Tetralogy of fallot.

or bronchial artery collaterals, were documented in TOF with PS.: Stenosis of LPA in 40% Associated cardiac abnormalities PFO or a true atrial defect in 83% of hearts with TOF…/BT shunt[1945]…SCAPA on side opposite AA Modified BT… esp in small infants <6 months…side to side anastom with interposition graft of PTFE or/ require no additional support in the neonatal period and go on to elective repair later in infancy. The surgical repair….. in addition to ventricular septal defect closure and right ventricular/


Percutaneous interventions in CHD Shunt lesions. ASD device closure.

,tamponade] : Incidence 0.1% Vulnerable sites : anterosup atrial wall & adjacent aorta Increased risk in pts with deficient aortic &/or superior rims; oversized devices /to the left atrium if needed in the future. VSD CLOSURE Recommendation for Device Closure of M-VSDs In infants >5 kg, children and /for Cardiac Catheterization and Intervention in Pediatric Cardiac Disease … AHA scientific statement.. Circulation 2011 Transcatheter Closure of Perimembranous Ventricular Septal Defects Butera et al JACC 2007/


VENTRICULAR SEPTAL DEFECT by Dr.Amarnath BR BMC. CONGENITAL HEART DISEASE (con-together,genitus-born) The majority of congenital anomalies of the heart.

deficient supracristal,subpulmonary,infundibular or conoseptal SEPTAL DEFICIFNCY SEPTAL DEFICIFNCY –AVseptal defect (AVcanal) CLINICAL FEATURES  Race : no particular racial predilection  Sex :no particular sex preference infants  Age :infants– difficult in postnatal period,although ccf during first 6mths/ normal tracing -mod.VSD ~ broad,notched P wave characteristic of Lt. Atrial overload as well as LV overload,namely,deep Q waves & tall R waves in leads V5 and V6 and often AF -large VSD ~RVH with rt./


Percutaneous interventions in CHD Shunt lesions

formation over the device : Incidence 0.2% Post-procedure AF and persistent atrial septal aneurysm were significant predictors of thrombus formation ASO less thrombogenic J Ayub Med Coll Abbottabad 2009;21(3) Several studies have shown outcomes from transcatheter device closure of secundum ASD to be comparable to surgical outcome in carefully selected adult and pediatric patients. Surgery versus percutaneous intervention. The largest/


Atrial Septal Defects Dr Nithin P G. Preview Introduction Embryology & Types of ASD Physiology, natural history & clinical features Investigations -salient.

Atrial Septal Defects Dr Nithin P G Preview Introduction Embryology & Types of ASD Physiology, natural history & clinical features Investigations -salient features Management References 1.Joseph Perloff. The clinical recognition of congenital heart disease. Fifth Edition 2003 2.Abraham M. Rudolf. Congenital diseases of the heart. Third Edition 2009 3.Moss & Adams’ Heart diseases in infants, children & Adolescents. Seventh Edition 2008 4.Nadas Pediatric Cardiology. Second Edition/


Nonrheumatic carditis in children. Cardiomyopathy. Prof. Pavlyshyn H.A.

– may delineate the fibrotic endomyocardial surface. Infants in whom valvular lesions or associated congenital cardiovascular defects are predominant die in the 1 st mo of life. Clinical/ ECG – combination of atrial enlargement, varying degrees of left or right ventricular hypertrophy; EchoCG in patient with DCMP Echo-CG/sudden death with implantable defibrillator is efficacious Surgery treatment – ventricular septal myotomy (disabling angina, syncope associated with LV outflow obstruction) – /


Evidence-based recommendations for psychotropic medications in pregnancy and lactation Dr Angelika Wieck Consultant in Perinatal Psychiatry September 2015.

palate Atrial septal defects Polydactyly Craniosynostosis OR: 12.7 (CI : 7.7 to 20.7) Carbamazepine 2 Spina bifida Cardiovascular anomalies ? OR: 2.6 (CI : 1.2 to 5.3) Lamotrigine 3-7 1 study found increase in oral clefts from 7 : 10,000 to 7 :1,000, subsequent studies did not confirm. Low teratogenicity compared with valproate but few comparisons with untreated infants yet/


CARDIOVASCULAR SYSTEM: CONGENITAL ABNORMALITIES JAMIE DAVIS, RN, BSN, CEN IN LIEU OF MSNED.

pressure to proximally to head and UE, and decrease pressure distally to body and LE  increased BP and bounding pulses in arms and weak or absent femoral pulses, cool LE TX: balloon aortoplasty or resection with anastomosis of aorta CYANOTIC CHD RIGHT/ Septal defect MUST BE PRESENT to allow saturation & unsaturated blood to mix (PDA/PFO). Infant dependent for survival S/S: CHF and cyanosis; CXR – egg heart on a string TX: prostaglandin E will allow mixing of blood to maintain sats of 75%; balloon atrial /


The Infant of the Diabetic Mother Maureen L. Tate LTC MC February, 2003.

Atrial septal defect l Hypertrophic Cardiomyopathy Congenital Malformations Renal l Hydronephrosis l Renal agenesis l Ureteral duplication Congenital Malformations GI l Duodenal atresia l Anorectal atresia l Small left colon syndrome Mrs. J is 32 YO, G1P0 who is currently in / initial steps of resuscitation including 45 sec. of FM PPV the infant responds and is transferred to the baby suite for further evaluation. Which baby is the infant of the diabetic mother? A B What perinatal and neonatal complications /


The Human Heart and Blood Flow.  Located in the Thoracic Cavity, between the two lungs and slightly to the left  About the size of a clenched fist.

Ventricular Septal Defect  There is a hole in the muscular wall (septum) separating the top two chambers of the heart and doesn’t close properly.  Oxygen rich blood from the left atrium flows into the right atrium. Atrial Septal Defect  /goes to the lungs. Patent Ductus Arteriosus Complications & Treatment  Heart failure  Endocarditis Treatment  Infants it can be closed with medications  In older children and adults, surgery is performed to close the vessel. Patent Ductus Arteriosus Pulmonary Valve /


Palliative Care in the Pediatric CVICU Case Studies Priya Bhat, MD, MS Pediatric Palliative Care Conference May 2, 2012.

or Sano Modification) Hybrid Stage 1- PDA stent, bilateral PA bands, atrial septostomy Diagnosis List Hypoplastic Left Heart Syndrome Severe tricuspid regurgitation Fetal Alcohol /in outcome measures. Barriers to PPC education = lack of time, lack of knowledgeable faculty and mentors. Jan 2010: 74 active palliative care fellowships. Role for specially trained, board-certified palliative care NPs. Case 2 Sara is a 4mo female infant with prenatally suspected chromosomal trisomy and unbalanced AV septal defect/


Vanessa Beretta & Dan Fleming. About CHD A congenital heart defect also known as CHD is a defect in the structure of the heart and great vessels. Most.

formed improperly. Some types of congenital heart defects can be related to an abnormality of an infants chromosomes (5% to 6%), single gene defects (3% to 5%), or environmental factors (2%). In 85 to 90 percent of cases, /pulmonary trunk are never properly divided. Ventricular Septal Defect is a defect in the wall dividing the ventricles (ventricular septum). This abnormal flow of blood between ventricles causes a murmur in some cases. Atrial Septal Defect enables blood flow between atriums through the /


Congenital Heart Defects

ductus arteriosus Pink Baby (L  R shunt) Diagnosing L  R shunts depends on: 1. Examination findings: Non-cyanotic infant in resp distress. Crackles, widely-fixed second heart sound, elevated JVP, cor pulmonale. 2. CXR: Increased pulmonary vasculature (suggestive of/Indomethacin Surgical closure (ligation). Atrial Septal Defect 6-10% of all births (1 of 1500 live births) 2 times more common in females than males. Types: Ostium Secundum (at or about the Foramen Ovale) Sinus Venous In 1950 most children with /


Congenital Cardiac Defects

during exercise. Infants may experience these difficulties during feeding, sometimes resulting in poor weight gain. Affected infants and children also may have swelling of the legs or abdomen or around the eyes. Certain heart defects can cause cyanosis/Septal defect: A hole in the septum that divides the right and left sides of the heart. A hole in the septum between the atria is called an atrial septal defect (ASD). A hole between the ventricles is called a ventricular septal defect (VSD). These defects/


Lupus in Pregnancy Darren Farley, MD Clinical Assistant Professor

cause NLE 30% of SLE pt have antissa ab and 15-20% have antissb ab Incidence of CHB in infants of SLE mothers is 2% Recurrence risk 5-25%; reports of twins discordant for NLE Therefore antissa /1:16) + SSA and + SSB Maternal HLA DR3 Structural heart defects most commonly associated with complete heart block Left atrial isomerism Transposition of the great arteries Atrioventricular septal defect Pulmonic atresia Anomalous pulmonary venous connection Double outlet right ventricle Atrioventricular discordance/


Diagnosis and Early Management of the Infant with Suspected Congenital Heart Disease.

heart abnormalities Evaluation (Continued) Hyperoxia test: baseline pre-ductal ABG when infant in room air, then repeat on 100% FiO2 Reason for ABG and/defect Ventricular Septal Defect Atrial Septal Defect Atrioventricular Canal Patent Ductus Arteriosus Initial Stabilization ABC’s: Volume resuscitation, ionotorpic support, correction of metabolic acidosis, r/o sepsis Intubate if needed, titrate Fi02 to keep Sp02 80%- 85% to prevent pulmonary overcirculation Placement of umbilical lines Infants who present in/


Sequelae of Congenital Heart Disease in the Adult Paul Reynolds, MD Uma and Sujit Pandit Professor and Chief of Pediatric Anesthesiology The University.

Cyanosis in Fontan Patients Patent Surgical Fenestration Baffle Leak (deoxygenated blood from systemic veins contaminates left atrial pulmonary venous blood) Systemic venous collateralization to left side Pulmonary AVM’s Hepatic veins to Coronary sinus or LA Intrinsic pulmonary pathology Diaphragm paralysis Driscoll DJ, Long-Term Results of the Fontan Operation. Pediatric Cardiol 2007, 28:438-442. Left to Right Shunts Ventricular Septal Defect (VSD) Atrial Septal Defect/


Ventricular Septal Defect Seoul National University Hospital Department of Thoracic & Cardiovascular Surgery.

description in 1879 Eisenmenger: Autopsy finding in 1897 Lillehei, Varco: Repair using controlled cross circulation in 1954 DuShane et al in Mayo Clinic : Intracardiac repair with pump oxygenator in 1955~1956 Lillehei : Atrial approach to VSD in 1957 Ventricular Septal Defect 1/septal type Malalignment Anterior (TOF) Posterior (COA, IAA) Rotational (Taussig-Bing) Morphologic Types of VSD VSD MO type VSD PMOE type VSD PMIE type VSD PMIE Type Associated Lesions with VSD 1. PDA 6% of all ages, 25% of infants in/


Hypoglycemia in Newborn Infants Incidence: Difficult to define: The more you test the more you have 8% of large for gestational age (LGA) infants 15%-20%

septal defect Coarctation of the aorta Atrial septal defect Cardiomegaly Renal anomalies Hydronephrosis Renal agenesis Ureteral duplication Gastrointestinal Duodenal atresia Anorectal atresia Small left colon Others Single umbilical artery Maternal obesity, gestational diabetes and central nervous system malformations Anderson JL et al, Epidemiology 2005;16:87–92 Overall OR for congenital defects (cardiovascular defects, renal/urinary anomalies, spine/rib alterations, holoprosencephaly) in infants/


Infant of the Diabetic Mother Sunhwa Kim, MD Loma Linda University Children Hospital.

in 10.3% of 155 case mothers versus 1.4% of 854 control mothers Multivariate-adjusted odds ratios (CI): Diabetes6.3 (2.7 -1 4.9) (Werler et al., Birth Defects Research 70:258, 2004) Diabetic Embryopathy – Cardiac anomalies u Transposition of great vessels u Coarctation of the aorta u Atrial & Ventricular septal defects/ u Formulas: only when medically indicated or mother has given informed consent u Keep mother and infant together continuously u Support mothers to nurse often (10-12 times per day) IDM- Long /


Birth Defects Shun Zhu, Department of Cellular and Genetic Medicine.

out to the rest of the body  Vary from mild (such as a small hole in the heart) to severe (such as missing or poorly formed parts of the heart)  Causes are largely unknown http://www.cdc.gov/ncbddd/heartdefects/facts.html Major Types of CHD  Atrial Septal Defect  Atrioventricular Septal Defect  Coarctation of the Aorta  Hypoplastic Left Heart Syndrome  Pulmonary Atresia  Tetralogy of Fallot  Total/


Encephalocoele with Congenital Heart Disease.  1. To present a case of a term infant with Encephalocoele and Congenital Heart disease  2. To know how.

Encephalocoele with Congenital Heart Disease  1. To present a case of a term infant with Encephalocoele and Congenital Heart disease  2. To know how to manage a / Disease,Taussig Bing Anomaly (Double outlet right ventricle, Malposition of great arteries, Ventricular septal defect, Atrial septal defect)  Neural tube defect  Sac-like protrusions of the brain  Due to failure of neural tube to close completely  Occur in 1:5000 births worldwide  Ethnicity  Genetics  Environmental factors  Parental age /


COMMON LEARNING ISSUES PBL TEST 1 2012. ALPHA FETOPROTEIN  USED AS A SCREENING MARKER INDICATING INCREASED RISK FOR BIRTH DEFECTS (NEURAL TUBE, BODY.

velocity of blood flow within heart and great vessels for valve function in regards to regurgitation  Septal defects, perfusion, valvular heart disease, prolapse, stenosis, subaortic stenosis, tumors,/ and auditory canal (rectal in infants) Usually above 99 degrees until after 3 years May approach 101 in normal children in late afternoon after vigorous activity/in blue top tube  PT: 11.5-13.5 second  INR: 0.8-1.4  Higher with mechanical heart valves or history of thromboembolitic disease or atrial/


BACH and Transitioning: Preparing Adolescents with CHD for Self-Care in Adulthood Susan M. Fernandes, MPH / Michael J. Landzberg, MD Boston Adult Congenital.

2 indicates Myocardial Oxygen Consumption; CCTGA, Congenitally Corrected Transposition of the Great Arteries; SD, Septal Defect; ANOVA, Analysis of the Variance; VO 2, Volume of Oxygen 12 Bouchardy J, et al. Circulation 2009 20 year risk of AA: 15% (> 3x higher) AA in ACHD: > 50%  mortal risk Atrial Arrhythmias (AA) in ACHD ACHD Arrhythmic “Phenotype” 15 Who is providing the care? Many being seen by/


HEART FAILURE IN NEONATE AND INFANT

and necrosis CHF beyond second week of life Most common cause of CHF in infants is VSD Presents around 6-8 weeks of age. Left to right / missing entirely. Defects in the IVS or abnormal LV function- Adversely affect the third phase of normal RV contraction through its interdependence on normal septal function Volume overload/childhood tachyarrhythmia is SVT Often presents in the first few months of life Rarely cause heart failure Occasionally PJRT ,ectopic atrial tachycardia and VT CHRONIC BRADYCARDIAS LV/


Tips for doing well in neonatology section of Pediatric Boards Shantanu Rastogi MD, MMM Neonatologist, Maimonides Medical Center Associate Professor of.

in WBN. The statement that you are MOST likely to include in your discussion is that –A. an infant should be tested while asleep –B. intervention in/ Hypoglycemia No ketosis 1.Fatty acid oxidation defects 2. Organic acidurias Ketonuria Normal lactate 1/and jaundice Specific malformations-Hypertrophic Obstructive Cardiomyopathy d/t asymmetrical ventricular septal hypertrophy and caudal agenesis syndrome Hypocalcemia Types –Early (till 72h/1-3 Norwood for HLHS Stage 1 –Atrial septectomy –PA to Asc.aorta –BT /


SUBSTANCE ABUSE & NEWBORNS. Why is this important:  5.5% of pregnant women in the United States reported using at least one illicit drug during pregnancy.

age)  Heart defects such as ventricular septal defect (VSD) or atrial septal defect (ASD) Structural problems with the face, including:  Narrow, small eyes with large epicanthal fold  Small head  Small upper jaw  Smooth groove in upper lip  Smooth/  Temperature instability  Loose diarrheal stools  Seizures  Nasal stuffiness  Sleep Disturbances Which leads to: “Unlovable Infant… Baby Outcomes  Guilt and Denial from the mother contribute to a poor communication/ connection between mom and baby /


The Relationship Between infertility Treatment and Birth Defects. Where We Are, Where Do We Want to Go, and How Can We Get There Michael Davies, MPH, PhD.

without death or major disability National Birth Defects Prevention Study (NBDPS) 5008 control infants and 13 586 case infant Contributors to ‘risk’ Patient characteristics /in the appendix of the annual reports contained ate the following web site. http://www.wch.sa.gov.au/services/az/other/phru/documents/2002_sabdr_annual_report.pdf CARDIOVASCULAR DEFECTS (74500 - 74799) Transposition of Great Vessels (74510 - 74519) Tetralogy of Fallot (74520) Ventricular Septal Defect (74540 - 74549) Atrial Septal Defect/


International Atomic Energy Agency Radiation risks in paedriatic interventional cardiology L 10.

atrial septal defect closure). B: Moore et al. [6] (patent ductus coil occlusion). C: Moore et al. [6] (pulmonary valvuloplasty). D: Wu et al. [8] (pulmonary valvuloplasty). E: Park et al. [10] (arhythmia ablation). F: Rosenthal et al. [11] (arhythmia ablation). Radiation Protection in Cardiology Lecture 10: Radiation risks in paediatric interventional cardiology15 Occupational exposure in pediatric interventions Although pediatric cardiologists have been performing cardiac catheterizations in infants/


Dr. Miada Mahmoud Rady Pediatric emergencies. Introduction  Children differ from adults in their anatomy, physiology, and emotions and experience a range.

includes Cardiorespiratory support and monitoring. Noncyanotic Disease Examples include: 1.Atrial septal defects (ASDs) 2.Ventricular septal defects (VSDs) 3.Patent ductus arteriosus (PDA) Clinical presentation varies. Coarctation of the aorta Definition : a discrete narrowing of the thoracic aorta. Major clinical finding: 1. Difference in systolic blood pressure between upper and lower extremities. 2.Most older infants and children remain asymptomatic. Congestive Heart Failure Definition : failure of/


Neurodevelopmental and Oral Feeding Outcomes for Infants born with Single Ventricle Physiology requiring Surgery Sharon Sables-Baus, PhD, RN, MPA, PCNS-BC.

in approximately 1 in 5000 babies and accounts for 1% of all congenital heart disorders The left ventricle (the pumping chamber) is small and the Mitral and/or the Aortic valve may be narrow, blocked or not formed at all The Aorta is often small (Hypoplastic) And there is a hole (Atrial Septal Defect/ DiGeorge Alagelle Shone Complex VACTERL Trisomy 21 Turner Klinefelter Ebstein Purpose To evaluate: Changes in infants’ Bayley scores from 6 to 12 months Relationship between medical variables to 12 month /


Background and Significance C. B. C. A. B. Population Pharmacokinetics of Dexmedetomidine in Infants Following Open Heart Surgery Felice Su MD, Susan C.

09 Conclusions To define the PK and safety of DEX in infants following open heart surgery To develop a population PK model exploring sources of variability in DEX PK Study Design Patient population 36 evaluable infants post-operative from open heart surgery Inclusion Criteria ­Age:/5.1-11.2) 7.0 Gender Female Male 5757 6666 5757 16 20 Surgical procedure Two ventricle physiology Atrial septal defect repair CAVC 1 repair RV – PA 2 conduit revision Subaortic membrane resection PAPVR 3 repair Rastelli Ross-Kono /


Cardiovascular Disease In Pregnancy It is a relatively common in women of child bearing age, complicate about 1% of pregnancies Maternal mortality related.

In other cases fetal consideration predominate, for example the teratogenic effect of warfarin Risks for Maternal Mortality Caused by Various Heart Disease Cardiac Disorder Mortality % Group 1 – Minimal Risk Atrial septal defect 0 -1 % Ventricular septal defect /in women,(3/4 th of cases of mitral stenosis) _Congenital Heart Disease: many congenital heart lesions appear to be inherited as polygenic characteristic, 10% of women with congenital heart disease would give birth to similarly affected infants/


Challenging Cases in Pediatric Anesthesia

Defects in abdominal wall Diaphragmatic abonormalities: Hernia Sternal deformities Cardiac and Pericardial defects Request Cardiac Echocardiography Hypoplastic LV, coarctation of the aorta with hypoplastic transverse arch, hypoplastic anomalous left pulmonary artery branch, hypoplastic mitral valve, large atrial septal defect/. The dose of ketamine needed to prevent gross movement in infants younger than 6 months of age is four times greater than in children 6 years of age57. Bioavailability of ketamine is /


Back To The Old School.

and a continuous grade 2 murmur sited below the left clavicle. What is the most likely cardiac defect? A Ventricular septal defect (VSD) B Atrial septal defect (ASD) C Patent ductus arteriosus (PDA) D Transposition of the great vessels E Totally anomalous pulmonary/pyloric stenosis Question 1. A four week old male infant presents with difficulty feeding. Mum is worried that he vomits (forcefully) after his feeds, and this is becoming more frequent. In addition, he is constantly hungry despite feeding well /


Diabetes in Pregnancy Dr. Priyanka Kalra Resident Dr. C. P. Kachchwa Assoc. Prof. Dr. (Mrs.) H. V. Singh Prof. Dr. (Mrs.) Sumitra Bora Prof. & Head Ob.

development Congenital Abnormalities Due to GDM  Cardiac (most common): transposition of great vessels, Ventricular septal defect, Atrial septal defect  Central nervous system (7.2%): spina bifida, Anencephaly, hydrocephalus  Skeletal: cleft lip/palate/ delivery may reduce the likelihood of brachial plexus injury in the infant  Unfavorable condition of the cervix is a problem  Maintain euglycemia during labor  Maternal hyperglycemia in labor: fetal hyperinsulinemia and worsen fetal acidosis  Maintain/


Viral Infections In Which Cardiovascular Manifestations Predominate

Myocarditis - Pericarditis Clinical features: relatively rare form of heart disease in U.S., generally acute and benign  Occurrence - a disease of newborns and infants; sometimes older children, occasionally in adults Antecedent URI---1-30d before symptoms refer to heart subacute or/ for vascular endothelium: patent ductus arteriosus, atrial septal defect, ventricular septal defect, lesions of myocardial fibers, alterations in renal arteries, pulmonary artery stenosis, and also thrombocytopenic purpura


DIABETES IN PREGNANCY BY DR. SHUMAILA ZIA.

of infants of diabetic mother Skeletal and central nervous system Caudal regression syndrome Neural tube defects Microcephaly Cardiac Transposition of the great vessels Ventricular septal defects Coarctation of the aorta defects or patent ductus arteriosus Atrial septal defects Cardiomegaly/: d) Biophysical Profile: POST PARTUM CARE CARE OF THE MOTHER CARE OF THE BABY Fall in insulin requirement in the puerperium. CARE OF THE BABY RESPIRATORY SYSTEM Resp distress syndrome Transient tachypnoea of the newborn /


Birth Defects Taryn Ballmann. Cleft Lip & Cleft Palate O The tissue that forms the lips or roof of the mouth do not join completely before birth O Can.

in life Heart Defects Causes Treatment O Mostly unknown O Genetic or chromosome changes O Diabetes or obesity O Smoking O Medications O Mother’s health O Depends on type and severity O May need one or more surgeries O Cardiac catheterization O Sometimes heart defect cannot be fully repaired Types of heart defects O Atrial Septal Defect/ protected, causing it to shorten, twist, or swell. O Even after repair, infants may have problems with feeding, digestion and absorption O 1,871 babies Gastroschisis Causes /


Birth Defects Taryn Ballmann. Cleft Lip & Cleft Palate O The tissue that forms the ____or _____ of the mouth do not ____ ___________before birth O Can.

defect __________ be fully repaired Types of heart defects O Atrial Septal Defect O Coarctation of the Aorta* O Ebstein anomaly* O Single ventricle* O Tetralogy of Fallot* O Truncus Arteriosus* O Ventricular Septal Defect/O Speech, occupational, and physical therapy O ______ _____________ programs in school O Most lead _________ and _________ lives Gastroschisis O Baby/it to shorten, ______, or ________. O Even after repair, infants may have problems with ________, ________ and ___________ of nutrients O /


Drugs In Pregnancy.

carbamazepine too and a pattern of malformations similar to phenytoin Increases the risk facial dysmorphology, neural tube defects, cardiovascular defects, and urinary tract defects. ANTICONVULSANTS VALPROIC ACID: Category D It is commonly used for petit mal seizures 1 to 2 % risk of neural tube defects with use in pregnancy atrial septal defect, cleft palate, hypospadias, polydactyly, and craniosynostosis. Antidepressants Recent publications have implicated some of the SSRIs administered/


بسم اللة الرحمن الرحيم. Presented by Yasser Hamed MD Neurology In Children Stroke.

. Seizures are common in both ischemic and hemorrhagic strokes. There can be significant differences in the clinical presentation based on the child’s age. Neonatal strokes present with focal seizures or lethargy. Infants present with lethargy, apnea/a high stroke risk, should be repaired. Resection of an atrial myxoma is indicated. Surgical repair or transcatheter closure is reasonable in individuals with a major atrial septal defect. In children with a risk of cardiac embolism, it is reasonable/


CARDIOVASCULAR DISORDERS IN NEONATES Juliet Ans Biju Independent Research GT Dr. Ruth Agwuna, Pediatrician Dr. Monique Satpute, Neonatologist Dr. Kiehl.

Webb).  Cause wrong blood flow to the heart ventricles, may rupture them (Smoking Early in Pregnancy Raises the Risk of Heart Defects in Infants)  Atrial Septal Defect- opening of the upper chambers of the heart (Woolston).  Nicotine will cause Arteriosclerosis-/.Vol. 21 No. 1 1997: 21-29. SIRS Government Reporter. Web. 30 Oct 2011  "Smoking Early in Pregnancy Raises Risk of Heart Defects in Infants." CDCPressRelease. 28 Feb 2011: n.p. SIRS Government Reporter. Web. 21 Sep 2011.  Scheel, Janet. Personal/


Lecturer Of Anesthesia

endocarditis D- Ostium secundum ASD AHA guidelines for bacterial endocarditis Prophylaxis in patients with cardiac conditions Endocarditis prophylaxis not recommended Endocarditis prophylaxis recommended Negligible-risk category - Physiologic, or functional heart murmurs - Surgical repair without residua beyond 6 months : ASD, PDA,VSD - Cardiac pacemaker or - implanted defibrillator - Isolated secundum atrial septal defect - Mitral valve prolapse without reg. - Previous coronary artery bypass surgery/


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