Ppt on atrial septal defect in children

Review Physiological Integrity of Children Question and Answer.

and withhold the medication 65. When evaluating the nursing interventions and goal attainment for an infant with suspected atrial septal defect (ASD), which finding necessitates further assessment? A.Child is acyanotic at rest without tachypnea and tachycardia B.Child/.Progressive replacement of bone marrow with inefficient blood producing cells A.Inadequate iron intake, which is common in children B.Decreased red blood cell production by bone marrow C.Increased destruction of red blood cells by lymphocytes/


Infective Endocarditis. Epidemiology and Prevention of Congenital Heart Defects Dr Alireza Ahmadi Assocciate Professor of Medical University of Isfahan.

septal defects and milder valvar stenoses 1,200,000 affected babies are born worldwide every year (36,000 in the United States), of whom at least 400,000 babies are severely affected(12,000 in the United States) Prevalence among children/Factors The exact causes of congenital heart disease are not known. However, genetic factors such as gene mutation can cause atrial septal defect (ASD), hypoplastic left heart syndrome (HLHS), rubella (German measles), viral infections such as flu, exposure to certain industrial /


CARDIOVASCULAR SYSTEM: CONGENITAL ABNORMALITIES JAMIE DAVIS, RN, BSN, CEN IN LIEU OF MSNED.

)  Ventricular Septal Defect (VSD)  Patent Ductus Arteriosus (PDA) Obstruction to systemic flow  Coarctation of the Aorta (COA)  Aortic Stenosis (AS) Non-cyanotic defects:  They don’t get blue  May have trouble gaining wt  Can have CHF and pulmonary overcirculation; sometimes (to make it more confusing) they can be asymptomatic  May have exercise or exertion difficulties an opening in the atrial septum permitting free communication of/


“Dominant-Negative ALK2 Allele Associates with Congenital Heart Defects” [Smith et. al, 2009] Presented by Arron Sikka & Anita Isama.

other than 22q1.1 Heterogeneity introduces complexity What is congenital atrial septal heart defect? Congenital means existing at birth Atrial septum  thin wall of tissue that separates the left and right atria Abnormalities occur when an opening exists in this region Causes, Signs, and Symptoms Occurs during fetal development May be genetic, but for most children cause unclear Severity of symptoms determined by the size and/


Endocarditis. Epidemiology An estimated 10,000 to 15,000 new cases of infective endocarditis (IE) are diagnosed in the United States each year. Men predominate.

of underlying heart disease in children with IE varies with age: 50 to 70 percent of children less than 2 years old have no obvious underlying cardiac disease, while most older children have coexistent congenital heart /in patients with preexisting valvular lesions are usually located on the atrial surface of incompetent atrioventricular valves, or the ventricular surfaces of incompetent semilunar valves. Patients with ventricular septal defects tend to develop vegetations on the orifice of the defect/


The Chest X-ray Still common In patient Portable imaging ‘Morning Portables’ Pre-op Post-op Out patient Still effective but possibly overused.

.html http://static.wikidoc.org/3/3a/Left-atrial-enlargement-002.jpg Aortic Stenosis Narrowing of the /Septal Defect. (2014). Orphanet Journal of Rare Diseases. 9(144), 1-16 Strife Janet & Sze, Raymond. (1999). Radiographic Evaluation of the Neonate with Congenital Heart Disease. Radiologc Clinics of North America. 37(6), 1093-1107 Tumkosit M, Yingyong N, Mahayosnond A, Choo K, Goo H. (2012). Accuracy of chest radiography for evaluating significantly abnormal pulmonary vascularity in children/


ANEMIA IN PEDIATRICS. Anaemia is defined as a low Hb concentration in blood, or less often, as a low haematocrit, the percentage of blood volume that.

absent lung lobes, vascular malformation, aortic atheromas, atrial septal defect, tetralogy of Fallot, pseudotruncus, hypoplastic aorta, abnormal pulmonary drainage, double aortic arch, cardiac myopathy OTHER ANOMALIES Slow development, hyperreflexia, Bells palsy, central nervous system arterial malformation, stenosis of the internal carotid artery, small pituitary gland, absent corpus callosum Hematologic Abnormalities The first hematologic abnormalities in individuals with FA are detected at a median age/


Heart, Blood, & Lymph Disease AH 120 Atherosclerosis The buildup of fatty plaque in the arteries and arterioles. The plaque decreases the lumen of the.

: chronic, low-grade blood loss in adults; poor dietary intake in pregnant women and young children Lab: hypochromic, microcytic RBCs Treatment:/in congenital spherocytic anemia is: A. Red blood cells are too small B. Red blood cells are spherical and swell up easily C. Platelets are large and ineffective D. White blood cells attack red blood cells E. Red blood cells are fragile and half- moon shaped Select the heart defect from the following list: A. Atrial septal defect (ASD) B. Ventricular septal defect/


Dr Masood EntezariAsl INTRODUCTION  the incidence of anesthesia-related mortality and morbidity remains higher in infants than in adults and higher.

d L) 1.9-5.02.1-4.82.5-5.52.2-4.0Albumin (g/dL) Developmental Changes in Blood Gas Values in children Paco ₂ Pao ₂ pHAge 55607.26-7.291 hour 33-35707.3724 hours 3570-807.401 week 3585-907/ carina, whereas the upper esophageal segment ends blindly in the mediastinum at the level of the second or third thoracic vertebra  Approximately 20% to 25% of these infants also have a congenital heart defect (ventricular septal defect, atrial septal defect, tetralogy of Fallot, atrioventricular canal, coarctation of the/


ALTERATIONS OF CARDIAC FUNCTION CONGENITAL HEART DEFECTS VALVULAR PROBLEMS ENDOCARDITIS ABDOMINAL AORTIC ANEURYSM 2009 CONGENITAL HEART DEFECTS VALVULAR.

IN TERMS OF S&S? RWHAT TYPE OF DEFECTS: ATRIAL SEPTAL DEFECT RWHERE IS THE Abnormal opening? RWHERE DOES THE Blood FLOW from & to? RWhat enlarges? RWHERE IS THE Abnormal opening? RWHERE DOES THE Blood FLOW from & to? RWhat enlarges? ATRIAL SEPTAL DEFECT/ RHeart muscle? RUrinary elimination? RVS? REnergy? Rappetite RTemperature of skin? RHeart muscle? RUrinary elimination? CHF IN CHILDREN RSystemic venous congestion RHow does it effect? RSystemic venous congestion RHow does it effect? RWeight? RLiver? RFluid /


Percutaneous interventions in CHD Shunt lesions. ASD device closure.

,tamponade] : Incidence 0.1% Vulnerable sites : anterosup atrial wall & adjacent aorta Increased risk in pts with deficient aortic &/or superior rims; oversized devices / atrium if needed in the future. VSD CLOSURE Recommendation for Device Closure of M-VSDs In infants >5 kg, children and adolescents with /for Cardiac Catheterization and Intervention in Pediatric Cardiac Disease … AHA scientific statement.. Circulation 2011 Transcatheter Closure of Perimembranous Ventricular Septal Defects Butera et al JACC /


A meta-analysis of percutaneous versus surgical closure of ostium secundum atrial septal defects Butera G, Biondi-Zoccai G, Abella R, Piazza L, Chessa.

Carminati M. Departement Of Pediatric Cardiology And GUCH Unit San Donato Milanese, Italy Background  Percutaneous atrial septal defect closure has became available in the last ten years  In literature there are studies comparing surgical and percutaneous closure Departement Of Pediatric Cardiology And GUCH Unit San/ Port Cardiol 2002)RetrospectiveSurg: 25 Kt: 38 38 (13-67) 40 (15-72) Bialkowksi (Tex heart Int J 2004)Retyrospective Children (< 18 yrs) Surg: 44 Kt: 47 6.5 (2.3-16.9) 9.9 (2.3-17) Butera (Am/


EFFECT OF LOW LEVEL LASER ON HEALING OF MODERATE SIZED INDUCED SEPTAL DEFECTS ON RABBITS By Hady Atef Labib Mohamed.

to find an alternative or even adjunctive non surgical method for some congenital heart defects like septal defects whether it is ventricular or atrial, in order to avoid or decrease -at least- open heart surgeries complications, which is/life. 2.Healing of induced cardiac shunts. 3.Different animal models. 4.Congenital septal defects in children. 5.Long term. 6.Functional improvement in the cardiovascular measurements Applications 1.Compare between application of low level LASER application percutaneusly/


Percutaneous interventions in CHD Shunt lesions

formation over the device : Incidence 0.2% Post-procedure AF and persistent atrial septal aneurysm were significant predictors of thrombus formation ASO less thrombogenic J Ayub Med Coll Abbottabad 2009;21(3) Several studies have shown outcomes from transcatheter device closure of secundum ASD to be comparable to surgical outcome in carefully selected adult and pediatric patients. Surgery versus percutaneous intervention. The largest/


New York State Center of Excellence in Bioinformatics & Life Sciences R T U CHSS Data Center Work Weekend Ontology, Terminology, and Cardiovascular Surgery.

drugs? What about such products taken by persons that are not patients? –e.g. children mistaking tablets for candies. New York State Center of Excellence in Bioinformatics & Life Sciences R T U Cardiovascular surgery examples Systemic venous anomaly, SVC, /my blood pressure my ASD my doctor my doctor’s computer 3. Representation ‘atrial septal defect’‘W. Ceusters’‘my heart defect’ New York State Center of Excellence in Bioinformatics & Life Sciences R T U Terminology is too reductionist What concepts do/


Heart failure in children

due to pulmonary hypertension, coarctation of aorta or aortic arch interruption. CHF in first week of life An atrial or ventricular septal defect (ASD/VSD) does not lead to CHF in the first two weeks of life. An additional cause must be sought (/ AG, Berlin, Germany) biventricular support Hetzer R and Stiller B (2006) Technology Insight: use of ventricular assist devices in children Nat Clin Pract Cardiovasc Med 3: 377–386 doi:10.1038/ncpcardio0575 Berlin heart General Measures Head end elevation, Judicious /


Congenital Heart Defects

border. Washing machine Echocardiography Treatment - PDA Restrict fluids. Diuretics Prostaglandin Inhibitors - Indomethacin Surgical closure (ligation). Atrial Septal Defect 6-10% of all births (1 of 1500 live births) 2 times more common in females than males. Types: Ostium Secundum (at or about the Foramen Ovale) Sinus Venous In 1950 most children with ASD did not reach the first grade. Today, first year surgery facilitates normal growth/


Infectious Disease Board Review

with which of the following? Patent ductus arteriosus (PDA) and branch pulmonary artery stenosis Ventricular septal defect (VSD) and PDA Atrial septal defect (ASD) and PDA VSD and ASD VSD and pulmonary artery stenosis Keypoint #26 Congenital Rubella / multocida Salmonella Pseudomonas aeruginosa Hemophilus influenza type b Osteomyelitis in a neonate Osteomyelitis in children with sickle cell disease Osteomyelitis in a patient who has received a puncture would in the foot through a tennis shoe Question 36 For/


Congenital Heart Defects

Restrict fluids. Diuretics Prostaglandin Inhibitors - Indomethacin Surgical closure (ligation). Atrial Septal Defect 6-10% of all births (1 of 1500 live births) 2 times more common in females than males. Types: Ostium Secundum (at or about the Foramen Ovale) Sinus Venous (at about the point where SVC and IVC attach to Right Atrium) In 1950 most children with ASD did not reach the first grade. Today/


STROKE IN THE YOUNG Chair : Prof. Dr. B. Jayakumar.

of Sinus of Valsalva –Intracardiac defects with paradoxical embolism Patent foramen ovale, atrial septal aneurysm, atrial septal defect –Cyanotic congenital heart disease –Iatrogenic/in children. –Risk is increased with arterial hypertension, atrial fibrillation, history of phlebotomy and microcytosis. –Low Hb is associated with arterial stroke and high Hb with cerebral venous thrombosis. Paradoxical embolism –Higher rate of cerebral ischemia is reported in persistent foramen ovale and atrial septal/


Nonrheumatic carditis in children. Cardiomyopathy. Prof. Pavlyshyn H.A.

biopsy – can detect other causes of cardiomyopathy (mitochondrial defects, storage disease). Paroxismal tachycardia Premature ventricular beats ECG/ atrial enlargement, varying degrees of left or right ventricular hypertrophy; EchoCG in /in 75% –Dizziness, pre-syncope, syncope  risk of SCD in children and adolescents Clinical Manifestation The pulse can be brisk – early systolic ejection of blood from ventricle; A prominent LV lift, double apical impulse (peaked) because ejection is interrupted by septal/


CONGENITAL HEART DEFECTS DR. HANA OMER. CONGENITAL HEART DEFECTS D. HANA OMER.

defects arise during this time. resulting from an interaction between a genetic predisposition toward development of a heart defect and environmental influences. Approximately 13% of children/’s syndrome. Acyanotic Disorders With left to right shunt :- 1.Atrial septal defect (ASD). 2.Ventricular septal defect (VSD). 3.Patent ductus arteriosis. With no shunt :- 1./s B lines : which are small 1-2 cm horizontal lines present in the costohrenic angle appearing due to raised pulmonary venous pressure. ECG. /


Medical Genetics 17 出生缺陷 Birth Defects. Medical Genetics Birth defects are defined as abnormalities of structure, function, or body metabolism that are.

itself Medical Genetics A.Abnormal passages in the heart or between blood vessels Atrial septal defect (ASD) Ventricular septal defect (VSD) Atrioventricular septal defect (AVSD) Patent ductus arteriosus (PDA) Medical Genetics Atrial septal defect (ASD) is a hole in the wall that separates the / In most babies, the vessel closes within a few hours or days after birth. In some children, the vessel fails to close, resulting in PDA. Medical Genetics B. Problems with the heart valves Congenital heart defects can/


Lecture 11 General med_2nd semester

the heart and great blood vessels are relatively frequent they occur in 6 - 8 children from 1 000 at birth their etiology is not clear and consists in rather complicated development of the heart and blood vessels most of / heart and great vessels: - persistent ductus arteriosus - ventricular septal defect - tetralogy of Fallot - atrial septal defect (s) - stenosis of pulmonary trunk DEVELOPMENT OF THE SPLEEN a spleen is entirely mesodermal in origin; developmentally it has close relations to the stomach an /


Sequelae of Congenital Heart Disease in the Adult Paul Reynolds, MD Uma and Sujit Pandit Professor and Chief of Pediatric Anesthesiology The University.

squatting only seen in children CVA, cerebral /in Fontan Patients Patent Surgical Fenestration Baffle Leak (deoxygenated blood from systemic veins contaminates left atrial pulmonary venous blood) Systemic venous collateralization to left side Pulmonary AVM’s Hepatic veins to Coronary sinus or LA Intrinsic pulmonary pathology Diaphragm paralysis Driscoll DJ, Long-Term Results of the Fontan Operation. Pediatric Cardiol 2007, 28:438-442. Left to Right Shunts Ventricular Septal Defect (VSD) Atrial Septal Defect/


DR. HANA OMER CONGENITAL HEART DEFECTS. The major development of the fetal heart occurs between the fourth and seventh weeks of gestation, and most congenital.

defects arise during this time. resulting from an interaction between a genetic predisposition toward development of a heart defect and environmental influences. Approximately 13% of children/’s syndrome. Acyanotic Disorders With left to right shunt :- 1. Atrial septal defect (ASD). 2. Ventricular septal defect (VSD). 3. Patent ductus arteriosis. With no shunt :- 1./ lines : which are small 1-2 cm horizontal lines present in the costophrenic angle appearing due to raised pulmonary venous pressure. ECG/


The Human Heart and Blood Flow.  Located in the Thoracic Cavity, between the two lungs and slightly to the left  About the size of a clenched fist.

Ventricular Septal Defect  There is a hole in the muscular wall (septum) separating the top two chambers of the heart and doesn’t close properly.  Oxygen rich blood from the left atrium flows into the right atrium. Atrial Septal Defect /Ductus Arteriosus Complications & Treatment  Heart failure  Endocarditis Treatment  Infants it can be closed with medications  In older children and adults, surgery is performed to close the vessel. Patent Ductus Arteriosus Pulmonary Valve Stenosis Blood flow from the /


First Do No Harm: Management of Atrial Septal Defect in Adult Patients Jimmy Klemis, MD Morbidity & Mortality Conference April 4, 2002.

First Do No Harm: Management of Atrial Septal Defect in Adult Patients Jimmy Klemis, MD Morbidity & Mortality Conference April 4, 2002 Case Presentation 68 Female presents with 3 rd admission in past 2yrs for “CHF” exacerbation. Notes /In asymptomatic patients, ASD repair offered no benefit with regard to mortality, morbidity or progression to atrial arrhythmia Limitations: uncontrolled study, advanced pulm HTN excluded (these pt do better with surgery), 22% of original pt lost to followup Children/


Cardiac Cath and Angiocardiography SPRING 2011. Catherization Studies and Procedures Adults Children.

size of RBC’s and removed by the reticuloendothial system 27 Interventional Procedures of the Vascular System: Children Balloon Septostomy to enlarge a patent foramen ovale or preexisting atrial septal defect This allows mixing of RT and LT blood –Resulting in improved arterial oxygenation Balloon is passed through atrial septal opening into the LT atrium, inflated with contrast and pulled back through the orifice –Causes septum to/


Management Of Medical Emergencies In The Dental Office

SUPPORT (BLS) CARDIOPULMONARY RESUCITATION (CPR) 3/31/2017 SBE Prophylaxis In 2012, the guidelines were updated and now premedication is needed for fewer/ stenosis congenital (present from birth) heart conditions such as ventricular septal defect, atrial septal defect and hypertrophic cardiomyopathy 3/31/2017 SBE Prophylaxis Procedures needing prophylaxis:/ Antibiotic Prophylaxis Prophylactic Regimen for Dental Procedures AMOXCICILIN Adults 2 grams Children 50 mg/kg (not to exceed adult dosage) Orally 1 hour/


1 VALUE OF 64 SLICE MULTIDETECTOR CT IN ANOMALOUS PULMONARY VENOUS RETURN (APVR) IN NEONATES AND INFANTS H MIZOUNI, M JRAD,E MENIF Service d’Imagerie Médicale.

Experienced staff and readers were required to manage children and reading images. 14 CASES 15 CASE 1  Infant aged 14 mouths  Dyspnea  Echocardiography: - - Enlargement of right heart chambers - - Sinus venosus atrial septal defect (ASD) 16 Fig.1a et 1b: /reduced to one lobe  Absence of visualization of the right scissure 23 Dextroversion heart 24   Right pulmonary venous return in the inferior vena cava   Dextroversion heart   Hypo vascularization of the right lung   Hypoplastic right lung field/


Congenital Cardiac Defects

often can close the ductus. If that doesnt work, surgery is needed. Septal defect: A hole in the septum that divides the right and left sides of the heart. A hole in the septum between the atria is called an atrial septal defect (ASD). A hole between the ventricles is called a ventricular septal defect (VSD). These defects can cause the blood to circulate improperly, so the heart has to/


The Etiology of Human Congenital Heart Defects Literature Seminar Feb 19 2009 Bernard Thienpont.

veins atriums and atrial septum AV valves and AV septal defect Ventricles and ventricular /atrial isomerism) Twin studies Problematic MZ : 25-50% concordance DZ : 13% concordance discordance? – Postzygotic mutations – Epigenetic Δ (e.g. X inactivation) – Stochastic factors Catastrophic Chance – … Genetic causes x1.3 x4.3 x5 Population risk = 0.8% Relative risk: Genetic causes Familial aggregation Excluding BAV Genetic causes CHD Frequency in parents of CHD children Genetic causes CHD frequency in children/


Congenital Heart Diseases: Atrial Septal Defect. Patient History This is a case of G.L., a 21 year old female, complaining of easy fatigability and occasional.

Atrial Septal Defect Patient History This is a case of G.L., a 21 year old female, complaining of easy fatigability and occasional chest pain who was referred for cardiovascular evaluation prior to employment. She was diagnosed to have “heart disease” in/edema premature birth, perinatal distress, or perinatal hypoxia may be present most children with small defects remain asymptoma-tic In patients with Eisenmenger syndrome, symptoms in adult life consist of exertional dyspnea, syncope and hemoptysis R-L shunt/


Acquired valve diseases Prof. S. Smiyan. EPIDEMOLOGY ARF is mainly a disease of children aged 5–14 years. ARF is mainly a disease of children aged 5–14.

Chronic MR: Afterload reduction: ACEI Afterload reduction: ACEI Anticoagulation in case of atrial fibrillation. Anticoagulation in case of atrial fibrillation. Mitral Regurgitation Diagnosis: Diagnosis: systolic murmur at apex + LA ↑, LV ↑ + Echo systolic murmur at apex + LA ↑, LV ↑ + Echo Differential Diagnosis: Differential Diagnosis: relative MR, ventricular septal defect, relative MR, ventricular septal defect, tricuspid regurgitation, aortic stenosis tricuspid regurgitation, aortic stenosis Management/


VENTRICULAR SEPTAL DEFECT by Dr.Amarnath BR BMC. CONGENITAL HEART DISEASE (con-together,genitus-born) The majority of congenital anomalies of the heart.

swiss-cheese OUTLET SEPTUM typeIII-OUTLET SEPTUM deficient supracristal,subpulmonary,infundibular or conoseptal SEPTAL DEFICIFNCY SEPTAL DEFICIFNCY –AVseptal defect (AVcanal) CLINICAL FEATURES  Race : no particular racial predilection  Sex :no particular sex preference infants  Age :infants– difficult in postnatal period,although ccf during first 6mths is frequent,X-ray&ECG are normal. children children—after first year variable clinical picture emerges.small VSD – asymptomatic large VSD – common/


Atrial Septal Defects Dr Nithin P G. Preview Introduction Embryology & Types of ASD Physiology, natural history & clinical features Investigations -salient.

Atrial Septal Defects Dr Nithin P G Preview Introduction Embryology & Types of ASD Physiology, natural history & clinical features Investigations -salient features Management References 1.Joseph Perloff. The clinical recognition of congenital heart disease. Fifth Edition 2003 2.Abraham M. Rudolf. Congenital diseases of the heart. Third Edition 2009 3.Moss & Adams’ Heart diseases in infants, children & Adolescents. Seventh Edition 2008 4.Nadas Pediatric Cardiology. Second Edition/


Transcatheter closure of Patent ductus arteriosus and Atrial septal defect without an onsite surgical backup; Two years experience in an African Community.”

Transcatheter closure of Patent ductus arteriosus and Atrial septal defect without an onsite surgical backup; Two years experience in an African Community.” Authors BA Animasahun, A Johnson, OO Ogunkunle, OA Idowu F Bode-Thomas S Maheshwari, SI Omokhodion OF Njokanma, 11/30/2015DR BA ANIMASAHUN2 Introduction Congenital heart disease contributes significantly to the health burden of children in Nigeria. Interventions for congenital heart disease had been available/


“It is of the highest importance in the art of detection to be able to recognize out of a number of facts, which are incidental and which vital. Otherwise.

) Ventricular Septal Defect (Membranous) Ebstein’s Anomaly Patent Ductus Arteriosus Aortic pulmonary Window Eisenmenger’s Syndrome Other Data: 1). ECG sinus tach, RBBB, ? right axis; * axis provides evidence for site of lesion, particularly if ASD: right axis – secundum; left axis – primum junctional or low atrial rhythm – sinus venousus * make sure not hidden atrial flutter * negative p waves in Ebstein’s, also accessory pathways * ? ST segments/


Heart diseases in pregnancy. Cardiovascular changes during pregnancy: intravascular volume and cardiac output increase by 50%  Decrease in peripheral.

, premature birth, IUGR, low birth weight, intrauterine fetal death Left-to-right cardiac shunts: atrial septal defect, ventricular septal defect, patent ductus arteriosus  The incidence of congenital heart disease in pregnancy is increasing - beter corresctive surgery in children  Higher cardiac output does not increase shunting becouse of attenuating effect of the decrease in peripheral vascular resistance  Pregnancy, labour and delivery – well tolerated unless pulmonary hypertension exists  Risk/


Corey Bregman, M.D. Children’s Memorial Hospital Chicago, IL Pulmonary Atresia with Intact Ventricular Septum.

is tailored to this variation Anatomy of the Defect Spectrum Well formed infundibulum, and the imperforate pulmonary/ Hypoplastic but hypertrophied right ventricle Right atrial dilatation Right atrial dilatation Tricuspid valve is rarely normal/ displacement of the septal and posterior leaflets Downward displacement of the septal and posterior leaflets /-up Sources 1. Allen, Hugh D. Moss & Adams’ Heart Disease in Infants, Children & Adolescents: Including the Fetus and Young Adults, 6 th Edition. Lippincott/


Transhepatic venous cardiac catheterization David Shim, MD Division of Pediatric Cardiology The Heart Center Childrens Hospital Medical Center Cincinnati,

ablation  ± transseptal puncture (4) Shim D,et al. Cathet Cardiovasc Interv 1999;47:41-5 Transhepatic interventions  Others  atrial septal defect device occlusion (2)  Fontan fenestration device occlusion (2)  coil embolization of pulmonary artery pseudoaneurysm(2)  device retrieval (1/in a subset of children where this has been previously not possible Speculations Transhepatic access will allow larger sheaths to be used in smaller patients The transhepatic approach may allow better sheath stability in/


Children With Special Healthcare Needs 1 st Trimester March 2013 Continuing Education.

Defects Types of Acyanotic Heart Defects VSD (most common) Ventral Septal DefectDefect in wall that separates ventricles ASD Atrial Septal Defect Patent ductus arteriosus Fetal blood passage doesn’t close after birth Obstructive lesions Narrows the aorta or valves Cardiovascular Defects Cyanotic Heart Defects Blood from arteries and veins mix in/ Tube (NGT): –Catheter placed through the nose into stomach –For supplementation in children who cannot take enough by mouth –Short-term use –Can use to decompress/


International Atomic Energy Agency Radiation risks in paedriatic interventional cardiology L 10.

(II) Cumulative skin dose is well correlated with patient size and not with fluoroscopy time Radiation Protection in Cardiology Lecture 10: Radiation risks in paediatric interventional cardiology14 Radiation Exposure to Children during various interventions (III) Comparison of surface entrance doses of radiation. A: Present study (Amplatzer atrial septal defect closure). B: Moore et al. [6] (patent ductus coil occlusion). C: Moore et al. [6] (pulmonary valvuloplasty/


Ventricular Septal Defect Seoul National University Hospital Department of Thoracic & Cardiovascular Surgery.

in 1879 Eisenmenger: Autopsy finding in 1897 Lillehei, Varco: Repair using controlled cross circulation in 1954 DuShane et al in Mayo Clinic : Intracardiac repair with pump oxygenator in 1955~1956 Lillehei : Atrial approach to VSD in 1957 Ventricular Septal Defect 1/with a Dacron patch Flap Valve Double Patch Closure Flap valve double patch closure of Ventricular Septal Defects in children with Increased Pulmonary Vascular Resistance Closure of Trabecular VSD Sandwich Technique Sutures & patch can /


Congenital Heart Defects and the Need for Transitional Services Brandon Howell Christine Haltiwanger Introduction to Health Promotion and Wellness 13 April.

in genes or chromosomes – Down’s syndrome has extra 21 st chromosome – Edward’s syndrome has extra 18 th chromosome Maternal risks – Toxins or poisons – Existing medical conditions – Medications Medical advances increase survival Medications for mild conditions Catheters can be used to repair – Atrial septal defect (ASD) – Ventricular septal defect (VSD) – Other defects Open heart surgery for complex cases Heart transplants for very severe conditions Survival Present Children/


Birth Defects Shun Zhu, Department of Cellular and Genetic Medicine.

to the characteristic cat-like cry of affected children  Also known as chromosome 5p deletion syndrome  A rare genetic disorder due to a missing part (deletion) of chromosome 5 Autism 1 in 68 babies About Autism  A neurodevelopmental / as a small hole in the heart) to severe (such as missing or poorly formed parts of the heart)  Causes are largely unknown http://www.cdc.gov/ncbddd/heartdefects/facts.html Major Types of CHD  Atrial Septal Defect  Atrioventricular Septal Defect  Coarctation of the Aorta/


Palliative Care in the Pediatric CVICU Case Studies Priya Bhat, MD, MS Pediatric Palliative Care Conference May 2, 2012.

stent, bilateral PA bands, atrial septostomy Diagnosis List Hypoplastic /48%- termination. ~55% of parents of HLHS children opt for palliative care when presented with all treatment/in outcome measures. Barriers to PPC education = lack of time, lack of knowledgeable faculty and mentors. Jan 2010: 74 active palliative care fellowships. Role for specially trained, board-certified palliative care NPs. Case 2 Sara is a 4mo female infant with prenatally suspected chromosomal trisomy and unbalanced AV septal defect/


EBSTEIN’S ANOMALY First described by Wilhelm Ebstein in 1866 ; terminology coined by Alfred Arnstein 19 yr old cyanotic laborer with CHF at autopsy Ebstein.

trabecular outlet In two third cases RV gets dilated – atrialised part RVapex RVOT EBSTEIN’S ANOMALY Associated defects interatrial communication in 80 - 94/in favour of Ebsteins 2Ddetailed evaluation of the tricuspid valve apparatus Apical displcement of septal leaflet chambers associated lesions RV function R > L shunt at atrial level EBSTEIN’S ANOMALY MV closure TV closure RVdilatation ; Paradoxical septal/BSA.  15 mm / sq.m in children < 14 yrs  20 mm /sq.m in adults Diagnostic of Ebstein’s EBSTEIN’S /


بسم الله الرحمن الرحيم.

in patients with cardiac conditions Endocarditis prophylaxis not recommended Endocarditis prophylaxis recommended Negligible-risk category - Physiologic, or functional heart murmurs - Surgical repair without residua beyond 6 months : ASD, PDA,VSD - Cardiac pacemaker or - implanted defibrillator - Isolated secundum atrial septal defect/ to avoid sever bradycardia in children with  CR - Atracuruim and vecronium: have few cardiovascular side effects in children when given in recommended doses. - Pancuronuim/


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