Ppt on atrial septal defect

Congenital Heart Disease Dr. Raid Jastania. Congenital Heart Disease 8 per 1000 live birth Could be minor defect or major defect Cause – unknown –Genetic:

-to-Right Shunt Most common ASD, VSD, PDA May be asymptomatic at birth Cyanosis is not early feature Atrial Septal Defect 75% ostium secondum ASD 15% ostium primum ASD 10% sinus venosus ASD Atrial Septal Defect –Asymptomatic early in life –Diagnosed usually in adulthood –In adult: atrial arrhythmia –Early : Left-Right shunt Middiastolic rumbling tricuspid murmur Midsystolic pulmonary ejection murmur –Pulmonary hypertension –Late: Right-Left Shunt Diastolic/


Infective Endocarditis. Epidemiology and Prevention of Congenital Heart Defects Dr Alireza Ahmadi Assocciate Professor of Medical University of Isfahan.

Factors The exact causes of congenital heart disease are not known. However, genetic factors such as gene mutation can cause atrial septal defect (ASD), hypoplastic left heart syndrome (HLHS), rubella (German measles), viral infections such as flu, exposure to certain industrial/ risks at BMI 25 to 29. Causality not clear. Association possibly owing in part to unrecognized diabetes. Smoking Septal defects, others Relative risk between 1 and 3, but some studies are negative. In individual studies, risk present if/


Medical Genetics 17 出生缺陷 Birth Defects. Medical Genetics Birth defects are defined as abnormalities of structure, function, or body metabolism that are.

blood vessels near the heart 4.Problems with development of the heart itself Medical Genetics A.Abnormal passages in the heart or between blood vessels Atrial septal defect (ASD) Ventricular septal defect (VSD) Atrioventricular septal defect (AVSD) Patent ductus arteriosus (PDA) Medical Genetics Atrial septal defect (ASD) is a hole in the wall that separates the upper chambers (atria (AY-tree-uh)) of the heart. This causes blood to leak/


Congenital Heart Disease

, VSD, PDA May be asymptomatic at birth Cyanosis is not early feature Atrial Septal Defect 75% ostium secondum ASD 15% ostium primum ASD 10% sinus venosus ASD Atrial Septal Defect Diagnosed usually in adulthood Left to right shunt Right atrial dilatation, right ventricular hypertrophy Pulmonary hypertension Late: Cyanosis (Eisenmenger syndome) CHF                                                      Ventricular Septal Defect 70% in the membarneous region Most common congenital heart disease Many VSD close/


EFFECT OF LOW LEVEL LASER ON HEALING OF MODERATE SIZED INDUCED SEPTAL DEFECTS ON RABBITS By Hady Atef Labib Mohamed.

all of this ?!!!! Simply for those Aim of our work This study aimed to find an alternative or even adjunctive non surgical method for some congenital heart defects like septal defects whether it is ventricular or atrial, in order to avoid or decrease -at least- open heart surgeries complications, which is still till now the only method for treatment of such cases. What happened/


Congenital Heart Defects Fred Hill, MA, RRT. Categories of Heart Defects Left-to-right shunt Cyanotic heart defects Obstructive heart defects.

, RRT Categories of Heart Defects Left-to-right shunt Cyanotic heart defects Obstructive heart defects L  R: Atrial Septal Defect Small defect usually no problem Large defects  blood flow to lungs –  c. o. 2 - 4 X   workload for the heart Presentation:  h. r. & contractility, diaphoresis, huff & puff, poor suck Shortened life span if uncorrected ASD closed ~ 5 years of age L  R: Ventricular Septal Defect Large defect   R V pressures May/


CONGENITAL DEFECTS. CHIHUAHUAS, MALTESE, POODLE, POMERANIAN, SHELTIE PUPPIES COMMONLY AFFECTED.

birth, the blood will shunt from left to right resulting in overload of the right side of the heart. Atrial Septal Defect  CLINICAL SIGNS: ATRIAL SEPTAL DEFECTS  Result in overload of the right side of the heart → dilation and hypertrophy of the right-sided chambers/the heart. The right ventricle may dilate as well.  CLINICAL SIGNS: VENTRICULAR SEPTAL DEFECTS:  Animals with small defects may have minimal or no signs  Larger defects may result in acute left-sided heart failure, usually by 8 weeks of age /


A meta-analysis of percutaneous versus surgical closure of ostium secundum atrial septal defects Butera G, Biondi-Zoccai G, Abella R, Piazza L, Chessa.

Carminati M. Departement Of Pediatric Cardiology And GUCH Unit San Donato Milanese, Italy Background  Percutaneous atrial septal defect closure has became available in the last ten years  In literature there are studies comparing surgical/Milanese, Italy Methods  Databases: Pubmed, Google Scholar, Biomed Central  Search updated April 07  Search terms: ‘atrial septal defect’, ‘closure’, ‘percutaneous’, ‘surgery’, ‘device’  Inclusion criteria for studies: Comparison of surgical versus percutaneous ASD /


Congenital Heart Defects

LEFT SHUNT Diagnosis - PDA Loud grade I to grade III systolic murmur at left sternal border. Washing machine Echocardiography Treatment - PDA Restrict fluids. Diuretics Prostaglandin Inhibitors - Indomethacin Surgical closure (ligation). Atrial Septal Defect 6-10% of all births (1 of 1500 live births) 2 times more common in females than males. Types: Ostium Secundum (at or about the Foramen Ovale) Sinus Venous (at/


First Do No Harm: Management of Atrial Septal Defect in Adult Patients Jimmy Klemis, MD Morbidity & Mortality Conference April 4, 2002.

First Do No Harm: Management of Atrial Septal Defect in Adult Patients Jimmy Klemis, MD Morbidity & Mortality Conference April 4, 2002 Case Presentation 68 Female presents/AMVL  LAD, 1 st degree AVB 75% Sinus Venosus  anomalous pulm venous drainage into RA or vena cavae  junctional/low atrial rhythm Physiologic Consequences Shunt Flow  Size of defect  Relative compliance of ventricles  Relative resistance of pulmonary/systemic circulation L  R shunting results in diastolic overload of RV and increased/


Vanessa Beretta & Dan Fleming. About CHD A congenital heart defect also known as CHD is a defect in the structure of the heart and great vessels. Most.

: cyanotic (a blue discoloration caused by a relative lack of oxygen), and non-cyanotic. Cyanotic Tetralogy of Fallet Transpostion of the great Vessels Tricuspid Atresia Truncus Arteriosus Non-Cyanotic Ventricular Septal Defect Atrial Septal Defect Patent Ductus Arteriosus Aortic Stenosis Full list of CHD’s found here: http://www.congenitalheartdefects.com/typesofCHD.htmlhttp://www.congenitalheartdefects.com/typesofCHD.html Tetralogy of Fallot involves three always present/


Congenital Heart Defects

://www.youtube.com/watch?v=KRy8gfmGSxg Cardiac Defects Patent Ductus Arteriosus Atrial Septal Defect Ventricular Septal Defect Tetralogy of Fallot Transposition of the Great Arteries Coarctation of/systolic murmur at left sternal border. Washing machine Echocardiography Treatment - PDA Restrict fluids. Diuretics Prostaglandin Inhibitors - Indomethacin Surgical closure (ligation). Atrial Septal Defect 6-10% of all births (1 of 1500 live births) 2 times more common in females than males. Types: Ostium Secundum/


Congenital Heart Diseases: Atrial Septal Defect. Patient History This is a case of G.L., a 21 year old female, complaining of easy fatigability and occasional.

Congenital Heart Diseases: Atrial Septal Defect Patient History This is a case of G.L., a 21 year old female, complaining of easy fatigability/ And those with chronic emboli Volume/ Pressure Overload –Seen in patients with left to right intracardiac shunts –May passively occur in patients with left atrial hypertension and left ventricular dysfunction, mitral valve disease and hose with aortic stenosis Pulmonary Arterial Hypertension Common Features –Medial hypertrophy –Eccentric and concentric intimal /


CONGENITAL HEART DEFECTS DR. HANA OMER. CONGENITAL HEART DEFECTS D. HANA OMER.

.Truncus arteriosus. 5.Eisenmenger’s syndrome. Acyanotic Disorders With left to right shunt :- 1.Atrial septal defect (ASD). 2.Ventricular septal defect (VSD). 3.Patent ductus arteriosis. With no shunt :- 1.Coarctation of aorta. 2./TREATMENT MEDICAL TREATMENT MILD DYSPNEA : salt restriction, low doses of diuretics. SINUS RHYTHM : beta blockers. ATRIAL FIBRILLATION : beta blockers, calaium channel blocker Prphylactic antibiotics to prevent infective endocarditis. SURGICAL TREATMENT PERCUTANEOUS TRANSLUMINAL/


Atrial Septal Defects Dr Nithin P G. Preview Introduction Embryology & Types of ASD Physiology, natural history & clinical features Investigations -salient.

Atrial Septal Defects Dr Nithin P G Preview Introduction Embryology & Types of ASD Physiology, natural history & clinical features Investigations -/ for device closure, rims Investigations- salient features Bi-caval view for IVC & SVC rims [subcoastal sagittal & TEE] Investigations- salient features AV rim & Postero- superior (atrial) rim [Suboastal coronal & TEE 4C] Aortic rim [TTE PSX & TEE BSX] Investigations- salient features MRI Investigations- salient features CATH Main indication is assessment of PVR/


DR. HANA OMER CONGENITAL HEART DEFECTS. The major development of the fetal heart occurs between the fourth and seventh weeks of gestation, and most congenital.

. Truncus arteriosus. 5. Eisenmenger’s syndrome. Acyanotic Disorders With left to right shunt :- 1. Atrial septal defect (ASD). 2. Ventricular septal defect (VSD). 3. Patent ductus arteriosis. With no shunt :- 1. Coarctation of aorta. 2. /MEDICAL TREATMENT MILD DYSPNEA : salt restriction, low doses of diuretics. SINUS RHYTHM : beta blockers. ATRIAL FIBRILLATION : beta blockers, calaium channel blocker Prphylactic antibiotics to prevent infective endocarditis. SURGICAL TREATMENT PERCUTANEOUS TRANSLUMINAL/


WATCHMANTM Left Atrial Appendage Closure Device

but are not limited to: Air embolism, Airway trauma, Allergic reaction to contrast media/medications or device materials, Altered mental status, Anemia requiring transfusion, Anesthesia risks, Angina, Anoxic encephalopathy, Arrhythmias, Atrial septal defect , AV fistula , Bruising, hematoma or seroma, Cardiac perforation , Chest pain/discomfort, Confusion post procedure, Congestive heart failure, Contrast related nephropathy, Cranial bleed, Decreased hemoglobin, Deep vein thrombosis, Death, Device embolism/


The Etiology of Human Congenital Heart Defects Literature Seminar Feb 19 2009 Bernard Thienpont.

of : Position and connection of heart Tetralogy of Fallot and variants Great veins atriums and atrial septum AV valves and AV septal defect Ventricles and ventricular septum VA valves and great arteries Coronary arteries, arterial duct and pericardium Embryological/: Position and connection of heart Tetralogy of Fallot and variants Great veins atriums and atrial septum AV valves and AV septal defect Ventricles and ventricular septum VA valves and great arteries Coronary arteries, arterial duct and /


SH-230609-AD JUN2015 WATCHMAN™ Left Atrial Appendage Closure Device www.watchmandevice.com.

but are not limited to: Air embolism, Airway trauma, Allergic reaction to contrast media/medications or device materials, Altered mental status, Anemia requiring transfusion, Anesthesia risks, Angina, Anoxic encephalopathy, Arrhythmias, Atrial septal defect, AV fistula, Bruising, hematoma or seroma, Cardiac perforation, Chest pain/discomfort, Confusion post procedure, Congestive heart failure, Contrast related nephropathy, Cranial bleed, Decreased hemoglobin, Deep vein thrombosis, Death, Device embolism/


ALTERATIONS OF CARDIAC FUNCTION CONGENITAL HEART DEFECTS VALVULAR PROBLEMS ENDOCARDITIS ABDOMINAL AORTIC ANEURYSM 2009 CONGENITAL HEART DEFECTS VALVULAR.

THE Blood FLOW from & to? RWhat enlarges? RWHERE IS THE Abnormal opening? RWHERE DOES THE Blood FLOW from & to? RWhat enlarges? ATRIAL SEPTAL DEFECT RS&S: RSurgical correction: R99% survival rate postop RS&S: RSurgical correction: R99% survival rate postop VENTRICULAR SEPTAL DEFECT (VSD) RAbnormal opening between? RWhat can happen at birth? RDescribe the effects of the shunt? And where the blood flows? RS&S/


HEART DISORDERS Blood Vessels –Arteriosclerosis –Atherosclerosis Arrythmias Structural Defects –Valve damage –Congenital Infections Congestive Heart Failure.

13 HEART DISORDERS Blood Vessels –Arteriosclerosis –Atherosclerosis Arrythmias Structural Defects –Valve damage –Congenital Infections Congestive Heart Failure ARRYTHMIAS Ectopic Beats and Reentry Sinus Node Abnormalities Conduction Abnormalities Atrial Ventricular AV node Page 281 Name some causes of / right side Still 18-24, C Why is the shunt in the right-to-left direction? Blood Flow through septal defects May also be called shunts 1. Direction of flow? –why? 2. Cyanotic? –Or Acyanotic? 3. Reduced/


SH 286002 AC JUN 2015 WATCHMAN TM Left Atrial Appendage Closure Device Clinical Data Overview.

but are not limited to: Air embolism, Airway trauma, Allergic reaction to contrast media/medications or device materials, Altered mental status, Anemia requiring transfusion, Anesthesia risks, Angina, Anoxic encephalopathy, Arrhythmias, Atrial septal defect, AV fistula, Bruising, hematoma or seroma, Cardiac perforation, Chest pain/discomfort, Confusion post procedure, Congestive heart failure, Contrast related nephropathy, Cranial bleed, Decreased hemoglobin, Deep vein thrombosis, Death, Device embolism/


Transcatheter closure of Patent ductus arteriosus and Atrial septal defect without an onsite surgical backup; Two years experience in an African Community.”

Transcatheter closure of Patent ductus arteriosus and Atrial septal defect without an onsite surgical backup; Two years experience in an African Community.” Authors BA Animasahun, A Johnson, OO Ogunkunle, OA / 2.3 11/30/20156DR BA ANIMASAHUN Results contd  All had acyanotic congenital heart disease:  73% had Patent Ductus Arteriosus (PDA) while 17% had secundum atrial septal defect (ASD).  Age range for those who had Patent ductus arteriosus was lower (3-16years with a mean age in years + SD of 6.3+ 4.22/


WATCHMAN™ Left Atrial Appendage Closure Device

but are not limited to: Air embolism, Airway trauma, Allergic reaction to contrast media/medications or device materials, Altered mental status, Anemia requiring transfusion, Anesthesia risks, Angina, Anoxic encephalopathy, Arrhythmias, Atrial septal defect , AV fistula , Bruising, hematoma or seroma, Cardiac perforation , Chest pain/discomfort, Confusion post procedure, Congestive heart failure, Contrast related nephropathy, Cranial bleed, Decreased hemoglobin, Deep vein thrombosis, Death, Device embolism/


Ventricular Septal Defect Seoul National University Hospital Department of Thoracic & Cardiovascular Surgery.

al in Mayo Clinic : Intracardiac repair with pump oxygenator in 1955~1956 Lillehei : Atrial approach to VSD in 1957 Ventricular Septal Defect 1. Embryology VSD occurs at 1 st. 8 weeks of fetal life through interaction/endocardial cushions, and bulbar ridges that separate great vessels. 2. Etiology Chromosomal Familial Geographic Environmental Ventricular Septal Defect  Pathophysiology Defect in the interventricular septum permits left-to-right shunting of blood RV volume overload & pulmonary overcirculation/


Birth Defects Shun Zhu, Department of Cellular and Genetic Medicine.

the heart) to severe (such as missing or poorly formed parts of the heart)  Causes are largely unknown http://www.cdc.gov/ncbddd/heartdefects/facts.html Major Types of CHD  Atrial Septal Defect  Atrioventricular Septal Defect  Coarctation of the Aorta  Hypoplastic Left Heart Syndrome  Pulmonary Atresia  Tetralogy of Fallot  Total Anomalous Pulmonary Venous Return  Transposition of the Great Arteries  Tricuspid Atresia  Truncus Arteriosus  Ventricular/


Anterior papillary muscle septalization associated with annuloplasty as a new approach to treat functional tricuspid regurgitation Jean-Paul Couetil, MD.

- Prof. Alfieri These techniques Address TR, but are not a pathophysiological approach Areas to Treat Septalization of APM + annuloplasty to reverse the physiopatological FTR mecanism? - Tricuspid valve tethering causing tenting, /valve replacementn=817% Aortic valve replacementn=613% Yacoub interventionn=12% Coronary artery bypass graftingn=48% Surgical ablation of atrial fibrillationn=48% Ventricular septal defect closuren=24% – (1 post STEMI, 1 congenital) – Extracorporeal duration : 118 ± 37 minutes – /


Congenital Cardiac Defects

syndrome , Holt-Oram syndrome (and Alagille syndrome What are some of the most common heart defects, and how are they treated? Patent ductus arteriosus (PDA) Septal defect (ASD, VSD) Coartation of the aorta Heart valve anomalies Tetrology of Fallot (TOF) / heart. A hole in the septum between the atria is called an atrial septal defect (ASD). A hole between the ventricles is called a ventricular septal defect (VSD). These defects can cause the blood to circulate improperly, so the heart has to work/


VENTRICULAR SEPTAL DEFECT by Dr.Amarnath BR BMC. CONGENITAL HEART DISEASE (con-together,genitus-born) The majority of congenital anomalies of the heart.

III abnormal S2 abnormal ECG abnormal X-ray abnormal BP VENTRICULAR SEPTAL DEFECT most common ACHD 2 nd most common CHD(32%) SYNONYMS * Roger’s disease * Interventricular septal defect * congenital cardiac anomaly PATHOPHYSIOLOGY  primarily depends on size&/ - pulmonary plethora ELECTROCARDIOGRAPHYELECTROCARDIOGRAPHY - smallVSD ~ normal tracing -mod.VSD ~ broad,notched P wave characteristic of Lt. Atrial overload as well as LV overload,namely,deep Q waves & tall R waves in leads V5 and V6 and often/


Congenital Heart Defects Hemodynamics, Pharmacology, and Updates Amanda L. Affleck CRNA, MAE Providence Anesthesia Services.

systemic flow=hypoperfusion SHUNTLeft-to-right Pulmonary over-circulation may lead to pulm htn, and eventually pulmonary vascular obstructive disease (Eisenmenger’s Syndrome) Acyanotic Defects Ventricular Septal Defect Atrial Septal Defect Persistent Ductus Arteriosus Aortic Stenosis Coarctation of the Aorta Complete Common Atrioventricular Canal Acyanotic Defects What increases left-to-right shunt? Dramatic increase in SVR relative to PVR. Dramatic decrease in PVR relative to SVR. Cyanotic/


Physiology of Cardiac Defects (Heart Sounds, murmurs and valvular problems), Dr. shafali singh.

aneurysm Reverse flow across a heart valve, e.g., valvular insufficiency Shunting of blood, e.g., ventricular septal defect, patent ductus murmurs Ejection murmur Pansystolic murmur Late systolic murmur Early murmur Mid to late murmur Systolic murmurs 1/ Overload of left ventricle Volume overload of right ventricle Left ventricular end diastolic pressure Left atrial pressure Right Atrial Pressure Left Ventricular Peak Systolic Pressure Pulmonary Artery Pressure Auscaltation findings recorded at the left /


Anatomy and physiology of the heart Embryology of normal heart and of the T4F The congenital heart defects and the T4F.

heart and of the T4F The congenital heart defects and the T4F Anatomy and Physiology of the / of the right ventricle The muscular strap reinforcing the septal surface, extending into the apical trabecular component is named the septomarginal trabeculation (septal band in USA), which has two limbs, the /This is called systole. As the ventricles start to contract, the ventricular pressure soon exceeds the atrial pressure, causing the AV valves to close (B1 murmur). As the ventricles continue to contract, /


Truncation and microdeletion of EVC accompanied by novel EFCAB7 missense mutation in Ellis-van Creveld syndrome with atypical congenital heart defect PEDIATRIC.

frenule-+-++---+ Hypodontia--+/------ Neonatal teeth-+-+---- Others----++-- 9 cAVSD complete artio-ventricular septal defectCA common atriumPS pulmonary pAVSD partial atrio-ventricular septal defectASD atrial septal defect stenosis Case E2: two novel compound heterozygous EVC2 mutations Heterozygote 14 /-genotype relationship remains elucidated. Novel EFCAB7 variant was found in patients with atypical cardiac defect; short chordae. Short chordae has never been reported in EvC EFCAB7 knockout mice showed/


C ONGENITAL H EART D EFECTS By: Victoria Lund. W HAT ARE CONGENITAL H EART DEFECTS ? They are problems with the heart that are present at birth. They.

the heart that are present at birth. They affect the flow of blood through the heart. Defects can range from no symptoms to life threatening TYPES Holes in the heart (septal defects) Atrial Septal Defect Little to no symptoms Heals on it’s own, or with a catheter Ventricular Septal Defect Small VSD’s have no problems and heal on their own Larger VSD’s can cause the/


Copyright © 2013, Canadian Cardiovascular Society 09/09/2015 1 2012 CCS Atrial Fibrillation Guidelines Update Skanes AC, Healey JS et al., Can J Cardiol.

2012 Mar;28(2): 125-136 Copyright © 2013, Canadian Cardiovascular Society 09/09/2015 16 2012 CCS Atrial Fibrillation Guidelines Update Echocardiogram Assess ventricular size / LV wall thickness / function Evaluate left atrial size (if possible, left atrial volume) Exclude significant valvular or congenital heart disease (particularly atrial septal defects) Estimate ventricular filling pressures and pulmonary arterial pressure Skanes AC, Healey JS et al., Can J Cardiol/


Catheter Ablation in the Treatment of Atrial Fibrillation

decrease conduction velocity) Flecainide Propafenone Drug choice depends upon patient’s underlying heart disease Nonpharmacological Approaches to Atrial Fibrillation Pacemaker therapy 2. Ablation 3. Surgery RF Ablation Techniques Focal ablation of PV (Pulmonary vein/ Patients with mitral and/or aortic metallic prosthetic valves are not excluded Previous repair of atrial septal defects is not an absolute contraindication Frequently Asked Question AF ablation for asymptomatic individuals? Asymptomatic Patients/


Syndromes and Birth Defects

leading cause of death in the first year of life. Caused by genetic, environmental or unknown factors. Most Common Birth Defects Heart Lip Palate Limbs Neural Tube Defects Congenital Heart Defects Any defect of the heart that is present at birth Atrial Septal Defect (ASD) Ventral Septal Defect (VSD) 1 in 125 babies are born with CHD Multiple causes and is often a characteristic of a syndrome but can/


Pathophysiology of atrial fibrillation and associated stroke Module 2 Developed and funded by DBG1780 | June 2013.

2013 Conditions associated with AF include: Symptomatic heart failure Valvular heart disease Cardiomyopathies Atrial septal defect Coronary artery disease Diabetes mellitus COPD Sleep apnoea Chronic renal disease ESC Guidelines for the management of atrial fibrillation 2010. Accessed June 2013 DBG1780 | June 2013 Causes of atrial fibrillation DBG1780 | June 2013 Causes of AF: atrial fibrosis Fibrotic tissue does not conduct electrical signals efficiently, thereby disrupting the heart/


The Relationship Between infertility Treatment and Birth Defects. Where We Are, Where Do We Want to Go, and How Can We Get There Michael Davies, MPH, PhD.

in the appendix of the annual reports contained ate the following web site. http://www.wch.sa.gov.au/services/az/other/phru/documents/2002_sabdr_annual_report.pdf CARDIOVASCULAR DEFECTS (74500 - 74799) Transposition of Great Vessels (74510 - 74519) Tetralogy of Fallot (74520) Ventricular Septal Defect (74540 - 74549) Atrial Septal Defect (74551 - 74559) Hypoplastic Left Heart Syndrome (74670) Patent Ductus Arteriosus (74700) Coarctation of Aorta (74710 - 74719) URO-GENITAL/


HEART DISEASE IN PREGNANCY. Mortality associated with specific cardiac lesions 1. Low risk of maternal mortality (less than 1%). (a) Septal defects. (b)

murmurs of severe intensity (grade 3). 3.Unequivocal enlargement of heart (X-ray). 4.Presence of severe arrythmias, atrial fibrillation or flutter The indications for Termination of pregnancy: Because of high maternal risks, MTP is indicated in: 1.Eisenmenger/ hypertension and hypervolemia avoid pulmonary edema should not put weight on the belly. Also, when a narrow pulmonary artery, septal defects, OAD. To not develop right ventricular failure and there was no discharge of blood from right to left. With /


Cryoballoon ablation for atrial fibrillation

in the CS and SVC via the femoral vein and a trans-septal puncture was performed. Pts were anticoagulated with either warfarin prior, IV / 3 Pleuritic CP persisting beyond procedure 1 Oesophageal ulceration 1 Transient visual field defect (TIA) 2 Vasovagals requiring medical Tx <2% uncomplicated haematomas 1 Right /and clinical effects of cryoballoon pulmonary vein isolation in patients with symptomatic paroxysmal and persistent atrial fibrillation. Europace (2008) 10, 1277–1280. 14. Packer DL, Irwin JM, /


Congenital Heart Defects and the Need for Transitional Services Brandon Howell Christine Haltiwanger Introduction to Health Promotion and Wellness 13 April.

syndrome has extra 18 th chromosome Maternal risks – Toxins or poisons – Existing medical conditions – Medications Medical advances increase survival Medications for mild conditions Catheters can be used to repair – Atrial septal defect (ASD) – Ventricular septal defect (VSD) – Other defects Open heart surgery for complex cases Heart transplants for very severe conditions Survival Present Children have a 90% chance of surviving to adulthood even with a severe/


Birth Defects Taryn Ballmann. Cleft Lip & Cleft Palate O The tissue that forms the lips or roof of the mouth do not join completely before birth O Can.

O Sometimes heart defect cannot be fully repaired Types of heart defects O Atrial Septal Defect O Coarctation of the Aorta* O Ebstein anomaly* O Single ventricle* O Tetralogy of Fallot* O Truncus Arteriosus* O Ventricular Septal Defect Down Syndrome Trisomy /under control O See health care professional regularly References O Centers for Disease Control and Prevention. (2014). Birth defects. Retrieved from http://www.cdc.gov/ncbddd/birthdefects/in dex.html http://www.cdc.gov/ncbddd/birthdefects/in dex/


Birth Defects Taryn Ballmann. Cleft Lip & Cleft Palate O The tissue that forms the ____or _____ of the mouth do not ____ ___________before birth O Can.

O Sometimes heart defect __________ be fully repaired Types of heart defects O Atrial Septal Defect O Coarctation of the Aorta* O Ebstein anomaly* O Single ventricle* O Tetralogy of Fallot* O Truncus Arteriosus* O Ventricular Septal Defect Down Syndrome Trisomy /under control O See health care professional regularly References O Centers for Disease Control and Prevention. (2014). Birth defects. Retrieved from http://www.cdc.gov/ncbddd/birthdefects/in dex.html http://www.cdc.gov/ncbddd/birthdefects/in dex/


COMMON LEARNING ISSUES PBL TEST 1 2012. ALPHA FETOPROTEIN  USED AS A SCREENING MARKER INDICATING INCREASED RISK FOR BIRTH DEFECTS (NEURAL TUBE, BODY.

flow: direction and velocity of blood flow within heart and great vessels for valve function in regards to regurgitation  Septal defects, perfusion, valvular heart disease, prolapse, stenosis, subaortic stenosis, tumors, aneurysm  Perflutren (definity or optison) /13.5 second  INR: 0.8-1.4  Higher with mechanical heart valves or history of thromboembolitic disease or atrial fibrillation  INR is now the standard measure reported CAUSES OF POSITIVE VALUES ON UA  Bilirubin: Jaundice, hepatitis, fecal/


Atrial and Ventricular Enlargement

T wave inversion (reason unknown) Right axis deviation Right Ventricular Hypertrophy Causes of RVH congenital heart disease atrial septal defects others Emphysema may mask signs of RVH Left Ventricular Hypertrophy With LVH, the electrical balance is tipped even/(not specific indicator). R wave > 11-13 mm in aVL ST segment changes Left axis deviation Left atrial abnormality ST strain patterns Left Ventricular Hypertrophy Causes: Hypertension Aortic stenosis Risks of LVH congestive heart failure arrhythmias /


Congenital Heart Defects Functional Overview Dr. Yasser Salem.

across Left to right shunt Factors affecting shunt flow Atrial level Ventricular or Great artery level Relative compliance Right vs Left ventricle Blood viscosity Size of defect Pressure gradient between chambers or arteries Ratio of PVR /CNTRACTILITYCARDIAC OUTPUT OXYGEN DELIVERY Afterload Heart rate OXYGEN extractionVenous saturation Arterial O 2 content preload Angels Demons Septal aneurysm Persistent left SVC Interrupted IVC Restrictive VSD High pressure gradient across VSD Law pressure gradient across/


9th October 2013 Dr Julian Tomkinson

with no evidence of any specific underlying cause). Hypertension (especially with associated left ventricular hypertrophy). Coronary artery disease. Valvular heart disease, especially mitral valve stenosis. Cardiac surgery. Myocarditis. Atrial septal defect. Atrial myxoma. Sick sinus syndrome. Pre-excitation syndromes, eg WPW Dilated and hypertrophic cardiomyopathy. Pericardial disease, eg pericardial effusion, constrictive pericarditis. Hyperthyroidism. Acute infections (especially pneumonia in the elderly/


Amplatzer® Septal Occluder

I will present the FDA summary for the Amplatzer ASO submission. This device is a transcathter septal defect closure device used in the treatment of atrial septal defects and Fenestrated Fontans. Today you will be asked to discuss and make recommendations on the sponsor’s/pivotal studies has been provided in the Panel packs. First is pivotal data set from the Phase IIB registry for atrial septal defect -ASD closure. A total of 459 patients were enrolled in this registry. Second is the pivotal data set for/


HEART FAILURE. FUNCTIONS OF HEART : Pumping blood. Secret atrial Natriuretic polypeptide (BNP).

HEART FAILURE FUNCTIONS OF HEART : Pumping blood. Secret atrial Natriuretic polypeptide (BNP). HEART FAILURE definition: HF in ability of the heart to pump adequate/disease (cor pulmonale)  pulmonary hypertension(PE &MVD).  tricuspid valve disease  pulmonary valve disease  CHD with left-to-right shunts (e.g. atrial or ventricular septal defects)  isolated right ventricular cardiomyopathy CLINICAL FEATURES  Left side heart failure  SOB on exertion.(SOB at rest in severe HF).  Orthopnea is dyspnea, /


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