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PowerPoint Lecture Outlines to accompany Hole’s Human Anatomy and Physiology Tenth Edition Shier  Butler  Lewis Chapter 9 Copyright © The McGraw-Hill.

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Presentation on theme: "PowerPoint Lecture Outlines to accompany Hole’s Human Anatomy and Physiology Tenth Edition Shier  Butler  Lewis Chapter 9 Copyright © The McGraw-Hill."— Presentation transcript:

1 PowerPoint Lecture Outlines to accompany Hole’s Human Anatomy and Physiology Tenth Edition Shier  Butler  Lewis Chapter 9 Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display. 9-1

2 Chapter 9 Muscular System 9-2 Three Types of Muscle Tissues Skeletal Muscle usually attached to bones under conscious control striated Smooth Muscle walls of most viscera, blood vessels, skin not under conscious control not striated Cardiac Muscle wall of heart not under conscious control striated

3 Structure of a Skeletal Muscle 9-3 Skeletal Muscle organ of the muscular system skeletal muscle tissue nervous tissue blood connective tissues fascia tendon aponeuroses

4 Structure of a Skeletal Muscle 9-4 epimysium perimysium fascicle endomysium muscle fascicles muscle fibers myofibrils thick and thin filaments

5 Skeletal Muscle Fiber 9-5 sarcolemma sacroplasm sarcoplasmic reticulum transverse tubule triad cisterna of sarcoplasmic reticulum transverse tubule myofibril actin filaments myosin filaments sarcomere

6 Sarcomere 9-6 I band A band H zone Z line M line

7 Myofilaments 9-7 Thick Filaments composed of myosin cross-bridges Thin Filaments composed of actin associated with troponin and tropomyosin

8 Neuromuscular Junction 9-8 site where axon and muscle fiber communicate motor neuron motor end plate synaptic cleft synaptic vesicles neurotransmitters

9 Motor Unit single motor neuron all muscle fibers controlled by motor neuron 9-9

10 Stimulus for Contraction 9-10 acetylcholine (ACh) nerve impulse causes release of acetylcholine from synaptic vesicles binds to acetylcholine receptors on motor end plate generates a muscle impulse muscle impulse eventually reaches sarcoplasmic reticulum

11 Excitation Contraction Coupling 9-11 muscle impulses cause sarcoplasmic reticulum to release calcium ions into cytosol calcium binds to troponin to change its shape position of tropomyosin is altered binding sites on actin exposed actin and myosin bind

12 Sliding Filament Theory 9-12 When sarcromeres shorten, thick and thin filaments slide past one another H zones and I bands get narrower Z lines move closer together

13 Cross-bridge Cycling 9-13 actin and myosin cross-bridge bind myosin cross- bridge pulls actin ADP and phosphate released from myosin new ATP binds to myosin linkage between actin and myosin cross-bridge break ATP splits myosin cross-bridge goes back to original position

14 Relaxation 9-14 acetylcholinesterase – breaks down acetylcholine muscle impulse stops calcium moves back into sarcoplasmic reticulum myosin and actin binding prevented

15 Energy Sources for Contraction 9-15 creatine phosphate – stores energy that quickly converts ADP to ATP 1) Creatine phosphate2) Cellular respiration

16 Oxygen Supply and Cellular Respiration 9-16 Anaerobic Phase glycolysis produces little ATP Aerobic Phase citric acid cycle electron transport chain produces most ATP myoglobin stores extra oxygen

17 Oxygen Debt 9-17 oxygen not available glycolysis continues pyruvic acid converted to lactic acid liver converts lactic acid to glucose Oxygen debt – amount of oxygen needed by liver to convert lactic acid to glucose

18 Muscle Fatigue 9-18 inability to contract commonly caused from decreased blood flow ion imbalances accumulation of lactic acid cramp – sustained, involuntary contraction

19 Heat Production 9-19 by-product of cellular respiration muscle cells are major source of body heat blood transports heat throughout body

20 Muscular Responses 9-20 Threshold Stimulus minimal strength required to cause contraction Recording a Muscle Contraction twitch latent period period of contraction period of relaxation refractory period all-or-none response

21 Summation 9-21 process by which individual twitches combine produces sustained contractions can lead to tetanic contractions

22 Recruitment of Motor Units 9-22 recruitment - increase in the number of motor units activated whole muscle composed of many motor units as intensity of stimulation increases, recruitment of motor units continues until all motor units are activated

23 Sustained Contractions 9-23 smaller motor units recruited first larger motor units recruited later produces smooth movements muscle tone – continuous state of partial contraction

24 Types of Contractions 9-24 isotonic – muscle contracts and changes length concentric – shortening contraction eccentric – lengthening contraction isometric – muscle contracts but does not change length

25 Fast and Slow Twitch Muscle Fibers 9-25 Slow-twitch fibers (type I) always oxidative resistant to fatigue red fibers most myoglobin good blood supply Fast-twitch glycolytic fibers (type II) white fibers (less myoglobin) poorer blood supply susceptible to fatigue Fast-twitch fatigue- resistant fibers (type IIb) intermediate fibers oxidative intermediate amount of myoglobin pink to red in color

26 Smooth Muscle Fibers 9-26 Compared to skeletal muscle fibers shorter single nucleus elongated with tapering ends myofilaments randomly organized no striations lack transverse tubules sarcoplasmic reticula not well developed

27 Types of Smooth Muscle 9-27 Visceral Smooth Muscle single-unit smooth muscle sheets of muscle fibers fibers held together by gap junctions exhibit rhythmicity exhibit peristalsis walls of most hollow organs Multiunit Smooth Muscle fibers function separately irises of eye walls of blood vessels

28 Smooth Muscle Contraction Resembles skeletal muscle contraction interaction between actin and myosin both use calcium and ATP both depend on impulses Different from skeletal muscle contraction smooth muscle lacks troponin smooth muscle depends on calmodulin two neurotransmitters affect smooth muscle acetlycholine and norepinephrine hormones affect smooth muscle stretching can trigger smooth muscle contraction smooth muscle slower to contract and relax smooth muscle more resistant to fatigue 9-28

29 Cardiac Muscle 9-29 only in the heart muscle fibers joined together by intercalated discs fibers branch network of fibers contracts as a unit self-exciting and rhythmic longer refractory period than skeletal muscle

30 Skeletal Muscle Actions 9-30 origin – immovable end insertion – movable end prime mover (agonist) – primarily responsible for movement synergists – assist prime mover antagonist – resist prime mover’s action and cause movement in the opposite direction

31 Major Skeletal Muscles 9-31

32 Major Skeletal Muscles 9-32

33 Muscles of Facial Expression 9-33

34 Muscles of Mastication 9-34

35 Muscles of Facial Expression and Mastication 9-35

36 Muscles That Move the Head and Vertebral Column 9-36

37 Muscles That Move the Pectoral Girdle 9-37

38 Muscles That Move the Arm 9-38

39 Deep Muscles of the Back and Neck 9-39

40 Muscles of the Shoulder and Back 9-40

41 Muscles of the Anterior Chest and Abdominal Wall 9-41

42 Muscles That Move the Forearm 9-42

43 Muscles That Move the Hand 9-43

44 Muscles of the Shoulder and Arm 9-44

45 Cross Section of the Arm 9-45

46 Muscles of the Shoulder and Arm 9-46

47 Muscles of the Arm and Forearm 9-47

48 Muscles of the Arm and Forearm 9-48

49 Cross Section of the Forearm 9-49

50 Muscles of the Abdominal Wall 9-50

51 Muscles of the Abdominal Wall 9-51

52 Muscles of the Pelvic Outlet 9-52

53 Muscles of Pelvic Outlets and Urogenital Diaphragm 9-53

54 Muscles That Move the Thigh 9-54

55 Muscles That Move the Leg 9-55

56 Muscles That Move the Foot 9-56

57 Muscles of the Thigh and Leg 9-57

58 Muscles of the Thigh and Leg 9-58

59 Muscles of the Thigh and Leg 9-59

60 Cross Section of the Thigh 9-60

61 Muscles of the Leg 9-61

62 Muscles of the Leg 9-62

63 Muscles of the Leg 9-63

64 Cross Section of the Leg 9-64

65 Life-Span Changes 9-65 myoglobin, ATP, and creatine phosphate decline by age 80, half of muscle mass has atrophied adipose cells and connective tissues replace muscle tissue exercise helps to maintain muscle mass and function

66 Clinical Application 9-66 Myasthenia Gravis autoimmune disorder receptors for acetylcholine on muscle cells are attacked weak and easily fatigued muscles result difficulty swallowing and chewing ventilator needed if respiratory muscles are affected treatments include drugs that boost acetylcholine removing thymus gland immunosuppressant drugs antibodies

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