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Anatomy & Physiology of the respiratory system in children

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1 Anatomy & Physiology of the respiratory system in children
prof. Pavlyshyn H.A., MD, PhD

2 Respiratory system The respiratory system is divided into two parts: upper and lower respiratory tract; The border of this division is the lower edge of the cricoid cartilage. Upper respiratory tract includes the paranasal sinuses, nasal cavity, pharynx and the Eustachian tube and other parts; Lower respiratory tract includes the trachea, bronchi, bronchial and alveolar capillary.

3 Anatomical characteristics Upper respiratory tract
The nose, nasal passages (airways), sinuses infancy are comperatively narrow + Mucosa is rich for vascular tissue } → that makes child vulnerable to infection and oedema; Infection, swelling of the nasal cavity and nasal congestion contribute more narrow or blocked of nasal airways, causing difficulty in breathing and sucking. There are not inferior (lower) nasal passages (until 4 years) and as a result rarely epistaxis in infants; Nasolacrimal duct is short, the opening valve, hypoplasia valve may be the cause of conjunctivitis with upper respiratory tract infection Development sinuses of infants continued after 2 years of age and finished to 12 years; the maxillary sinuses is usually present at birth; the frontal sinuses begin to develop in early infancy; Babies can suffer from sinusitis; the ethmoid, maxillary sinuses are most vulnerable to infection.

4 Upper respiratory tract in children
Larynx is located on level the 3-4th (neck) vertebrae; Vocal and mucous membranes are rich blood vessels and lymphatic tissue, prone to inflammation, swelling, due to babies suffering from laryngitis (viral croup), airway obstruction, inspiratory dyspnea;

5 Anatomical characteristics Lower respiratory tract
The trachea is short; Tracheal and bronchial passes in children is relatively small, cartilage soft, the lack of elastic tissue LRT – vulnerable, easy to cause airway narrowing and obstruction Trachea Bronchi Tubes Right bronchus more straight, like a direct extension of the trachea (causing the right lung atelectasis or emphysema); Left bronchus is the separation from the trachea; The bronchus is divided into inter-lobe bronchus, segmental bronchus, bronchioles. Bronchioles - no cartilage, smooth muscle imperfect development, mucosa rich in blood vessels, mucous glands hypoplasia, lack of secretion of mucus, poor mucociliary movement; Bronchiole Alveoli

6 Anatomy and physiology
The ribs are cartilaginous and perpendicular relative to the vertebral column (horizontal position), reducing the movements of the rib cage. The infant chest wall is remarkably compliant and compliance decreases with increasing age. The orientation of the ribs is horizontal in the infant; by 10 years of age, the orientation is downward.

7 The mecanizm of breathing
Contraction of diaphragm: diaphragm moves downward >increases vertical dimension of thoracic cavity Contraction of external intercostal muscles > elevation of ribs & sternum > increased front- to-back dimension of thoracic cavity lowers air pressure in lungs air moves into lungs

8 Anatomy and physiology
The intercostal muscles and accessory muscles of ventilation are immature. As a result, children are more reliant on the diaphragm for inspiration. Increased respiratory effort causes subcostal and sternal recession, and the mechanical efficiency of the chest wall is reduced.

9 Summary The considerable differences in respiratory physiology between infants and adults explain why infants and young children have a higher susceptibility to more severe manifestations of respiratory diseases, and why respiratory failure is common problem in neonatal and pediatric intensive care units. The appreciation of the peculiarities of pediatric respiratory physiology is not only essential for correct assessment of any ill child, but also for correct interpretation of any pulmonary function test performed in this population.

10 An average respiratory rate at rest of the child of different age is:
newborn per minute, infant at 6 months per minute, at 1 year 30 per minute, 5 years 25 per minute, 10 years 20 per minute, 12-18 years per minute.

11 Disorders of the respiratory rate
Tachypnea is the increase of the RR (Interstitial, vascular and multitude of diseases, anxiety) Bradypnea is the decrease of the RR (Narcotics, raised intracranial tension, myxedema) Dyspnea is the distress during breathing Apnea is the cessation of breathing

12 Disorders of the respiratory depth
Hyperpnea is an increased depth. Hypoventilation is a decreased depth and irregular rhythm. Hyperventilation is an increased rate and depth.

13 Pathological respiration
Seesaw (paradoxic) respirations: the chest falls on inspiration and rises on expiration. It is usually observed in respiratory failure of third degree, RDS; Cheyne-stokes breathing: cyclical increase and decrease in depth of respiration (CHF, cerebrovascular insufficiency) Kussmaul: slow deep breathing, hyperventilation, gasping and labored respiration (Ketoacidosis) Biot's breathing: totally irregular with no pattern (CNS injury)

14 Percussion Resonant sounds are low pitched, hollow sounds heard over normal lung tissue. Flat or extremely dull sounds are normally heard over solid areas such as bones.

15 Percussion Percuss the lung fields, alternating, from top to bottom and comparing sides Percuss over the intercostals space. Keep the middle finger firmly over the chest wall along intercostals space and tap chest over distal interphalangeal joint with middle finger of the opposite hand. The movement of tapping should come from the wrist. Tap 2-3 times in a row.

16 Percussion Percuss the chest all around. Stand back, have the patient cross arms to shoulder. This maneuver will wing the scapula and expose the posterior thorax. Then, have the patient keep their hands over head and percuss axilla. Then move to the front and percuss anterior chest , clavicles and supraclavicular space.

17 Percussion The lung is filled with air (99% of lung is air).
Percussion of it gives a resonance. This step helps identify areas of lung devoid of air. Appreciate the dullness of the left anterior chest due to heart and right lower chest due to liver. Note the hyper-resonance of the left lower anterior chest due to air filled stomach. Normally, the rest of the lung fields are resonant.

18 The pathological dullness is heard in cause of
Dull or thud like sounds are normally heard over dense areas such as the heart or liver. Dullness replaces resonance when fluid or solid tissue replaces air-containing lung tissues, such as occurs with pneumonia, pleural effusions (hydro-, haemothorax), or tumors. Decreased resonance is noted with pleural effusion and all other lung diseases.

19 The hyper resonant sounds is heard in cause of
Increased resonances can be noted either due to lung distention as seen in asthma, emphysema or due to Pneumothorax. Hyper resonant sounds that are louder and lower pitched than resonant sounds are normally heard when percussing the chests of children and very thin adults. Hyper resonant (ban-box) sounds may also be heard when percussing lungs hyperinflated with air, such as emphysema of lungs, patients with COPD, asthma, asthmatic bronchitis. An area of hyper resonance on one side of the chest may indicate a pneumothorax.

20 The tympanic sounds is heard
in cause of Tympanic sounds are hollow, high, drum like sounds. Tympany is normally heard over the stomach, but is not a normal chest sound. Tympanic sounds heard over the chest indicate excessive air in the chest, such as may occur with pneumothorax.

21 Anatomy of lobes of lungs

22 Auscultation method of exam
Auscultate the lungs from the apices, middle and lower lung fields posteriorly, laterally and anteriorly. Alternate and compare sides. Listen to at least one complete respiratory cycle at each site. First listen with quiet respiration. If breath sounds are inaudible, then have him take deep breaths. First describe the breath sounds and then the adventitious sounds.

23 Auscultation method of exam
Note the intensity of breath sounds and make a comparison with the opposite side. Assess length of inspiration and expiration. Listen for the pause between inspiration, expiration. Compare the intensity of breath sounds between upper and lower chest in upright position. Note the presence or absence of adventitious sounds.

24 To assess the posterior chest, ask the patient to keep both arms crossed in front of his/her chest, if possible. Begin by auscultation the apices of the lungs, moving from side to side and comparing as you approach the bases. If you hear a suspicious breath sound, listen to a few other nearby locations and try to delineate its extent and character. It is important that you always compare what you hear with the opposite side.

25 Normal breath sounds tracheal, bronchial, broncho-vesicular and vesicular sounds. Breath sounds are described by: duration (how long the sound lasts), intensity (how loud the sound is), pitch (how high or low the sound is), and timing (when the sound occurs in the respiratory cycle).

26 Breath sounds can be divided into the following categories:
 Normal Abnormal Adventitious tracheal absent/decreased crackles (rales) vesicular bronchial wheeze rhonchi bronchovesicular stridor pleural rub mediastinal crunch (Hamman's sign)

27 Normal breath sounds Bronchial sounds are present over the large airways in the anterior chest near the second and third intercostal spaces (trachea, right sternoclavicular joints and posterior right interscapular space); These sounds are more tubular and hollow-sounding than vesicular sounds, but not as harsh as tracheal breath sounds. Bronchial sounds are loud and high in pitch with a short pause between inspiration and expiration (inspiration and expiration are equal); expiratory sounds last longer than inspiratory sounds.

28 The Bronchial Breath Sound
has the following characteristics : An I:E Ratio : 1:1 or 1:1 1/4 with a pause in between inspiration & expiration Thoracic Geography : over the manubrium of the sternum Sound Characteristics : high pitched, tubular, hollow sound Indication: that an area of consolidation exists - pneumonia, atelectasis, fluid infiltration

29 The Bronchovesicular Breath Sound
has the following characteristics : An I:E Ratio : 1:1 or 1:1 1/4 with a pause in between inspiration & expiration Thoracic Geography : They are best heard in the 1st and 2nd ICS (anterior chest) and between the scapulae (posterior chest) - over the main stem bronchi Sound Characteristics : high pitched, tubular, hollow sound Indication : an area of consolidation - pneumonia, atelectasis, fluid infiltration

30 Abnormal breath sounds include:
the absence of sound and/or the presence of sounds in areas where they are normally not heard. For example, bronchial breath sounds are abnormal in peripheral areas where only vesicular sounds should be heard. When bronchial sounds are heard in areas distant from where they normally occur, the patient may have consolidation (as occurs with pneumonia) or compression of the lung. These conditions cause the lung tissue to be dense. The dense tissue transmits sound from the lung bronchi much more efficiently than through the air-filled alveoli of the normal lung.

31 Summery Type Characteristic Intensity Pitch Description Location
Normal tracheal loud high harsh; not routinely auscultated over the trachea vesicular Soft low . most of the lungs bronchial very loud sound close to stethoscope; gap between insp & exp sounds over the manubrium (normal) or consolidated areas bronchovesicular Medium medium normally in 1st & 2nd ICS anteriorly and between scapulae posteriorly; other locations indicate consolidation Abnormal absent/decreased heard in ARDS, asthma, ateletasis, emphysema, pleural effusion, pneumothorax indicates areas of consolidation

32 The term “adventitious” breath sounds
refers to extra or additional sounds that are heard over normal breath sounds. crackles (or rales) wheezes (or rhonchi) pleural friction rubs stridor

33 Adventitious sounds Wheeze Stridor Crackles Pleural Rub

34 Crackles (or rales) Crackles are discontinuous, intermittent, nonmusical, brief, "popping" sounds that originate within the airways. are caused by fluid in the small airways or atelectasis. Crackles may be heard on inspiration or expiration. The popping sounds produced are created when air is forced through respiratory passages that are narrowed by fluid, mucus or pus. Crackles are often associated with inflammation or infection of the small bronchi, bronchioles and alveoli (pneumonia, atelectatic lung). Crackles are often described as fine (high pitched, soft, very brief), coarse (low pitched, louder, less brief). Fine crackles are soft, high-pitched, and very brief. You can simulate this sound by rolling a strand of hair between your fingers near your ear. Coarse crackles are intermittent "bubbling" sound somewhat louder, lower in pitch, and last longer than fine crackles.

35 Wheeze Lung Sounds These are continuous, high pitched, hissing,
whistling or sibilant sounds. They are caused by air moving through airways narrowed by constriction or swelling of airway or partial airway obstruction (asthma, CHF, chronic bronchitis, COPD). Wheezes are sounds that are heard continuously during inspiration or expiration, or during both inspiration and expiration. Wheezes that are relatively high pitched and have a shrill or squeaking quality may be referred to as sibilant rhonchi. These wheezes occur when airways are narrowed, such as may occur during an acute asthmatic attack. Wheezes that are lower-pitched sounds with a snoring or moaning quality may be referred to as sonorous rhonchi. Secretions in large airways, such as occurs with bronchitis, may produce these sounds;

36 Rhonchi Lung Sounds These are low pitched, continuous, musical, snore-like sounds that are similar to wheezes. They are caused by airway secretions and airway narrowing. They usually clear after coughing.

37 Pleural friction rubs are low-pitched, grating, or creaking sounds that occur when inflamed pleural surfaces rub together during respiration. More often heard on inspiration than expiration, the pleural friction rub is easy to confuse with a pericardial friction rub. To determine whether the sound is a pleural friction rub or a pericardial friction rub, ask the patient to hold his breath briefly. If the rubbing sound continues, its a pericardial friction rub because the inflamed pericardial layers continue rubbing together with each heart beat - a pleural rub stops when breathing stops.

38 Summery Type Characteristic Intensity Pitch Description Location
Adventitious crackles (rales) soft (fine crackles) or loud (coarse crackles) high (fine crackles ) or low (coarse crackles) discontinuous, nonmusical, brief; more commonly heard on inspiration; assoc. w/ ARDS, asthma, bronchiectasis, bronchitis, consolidation, early CHF, interstitial lung disease may sometimes be normally heard at ant. lung bases after max. expiration or after prolonged recumbency wheeze high expiratory continuous sounds normally heard on expiration; note if monophonic (obstruction of 1 airway) or polyphonic (general obstruction); assoc. w/ asthma, CHF, chronic bronchitis, COPD, pulm. edema can be anywhere over the lungs; produced when there is obstruction rhonchi low continuous musical sounds similar to wheezes; imply obstruction of larger airways by secretions . stridor inspiratory musical wheeze that suggests obstructed trachea or larynx; medical emergency heard loudest over trachea in inspiration pleural rub insp. & exp. creaking or brushing sounds; continuous or discontinuous; assoc. w/ pleural effusion or pneumothorax usually can be localized to particular place on chest wall mediastinal crunch not synchronized w/ respiration crackles synchronized w/ heart beat; medical emerg.; assoc. w/ pneumomediatstinum best heard w/ patient in left lateral decubitus position



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