Presentation on theme: "Assessment and Treatment of Low Back Pain Steven Stanos, DO Medical Director Center for Pain Management Rehabilitation Institute of Chicago Asst. Professor,"— Presentation transcript:
Assessment and Treatment of Low Back Pain Steven Stanos, DO Medical Director Center for Pain Management Rehabilitation Institute of Chicago Asst. Professor, Dept. PM&R Northwestern University Medical School Feinberg School Of Medicine
Goals Individualized yet comprehensive Efficient Comfortable for patient Comfortable for clinician Build rapport Educate and prepare patient for treatment Monitor for inconsistencies
Anatomy of LumboSacral Spine Bogduk N, Clinical Anatomy Lumbar Spine and Sacrum, 3 rd. Ed. Churchill Livingstone, 1999.
Lumbar Facets: zygapophysial joints “z-joint” Bogduk N, Clinical Anatomy Lumbar Spine and Sacrum, 3rd. Ed. Churchill Livingstone, 1999.
Definitions Somatome: field of somatic and autonomic innervation based on embryologic segmental origin of somatic tissues three basic elements: 1. Dermatome: cutaneous structures 2. Myotome: skeletal musculature 3. Sclerotome: bones, joints, and ligaments 8 Inman VT, Saunders J. J Nerv Ment Dis 1944;99:
Spinal “stability” Neural Control Unit Spinal Column Spinal Muscles Vertebral Position Spinal Loads Spinal Motions Muscle Activation Patterns Panjabi MM. J Electromyography Kinesiology 2003:12:371-9
“Core” muscle groups –Abdominals (Front) –Paraspinals and gluteals (Back) –Diaphragm (Roof) –Pelvic floor and hip muscles (Bottom) Richardson C, et al.Therapeutic exercise for spinal stabilization and low back pain. Edinburgh (Scotland): Churchill Livigstone1999.
Gait Balance Base of support Arm swing/ trunk and shoulder rotation Cadence Leg: cicumduction, stance time, position Pain behavior
Static Stance Assessment (J. Rittenberg. Photos from practice & personal files used with permission) L4-L5 PSIS
Flexion Based Muscular Ligamentous Compression Fracture Discogenic Extension Based Stenosis Facet Spondylosis Central Disc Transitional Spondylolisthesis Sacroiliac Facet Differential Diagnosis
Facet Arthropathy Zygapophyseal (z-joint) Poor correlation with history and exam 1 Commonly pain with extension & rotation Referral patterns 2 1.Schwarzer AC, et al. Spine 1994;19: Slipman, C. Arch PM&R 81: , 2000.
Myofascial Trigger Points Travell JG, Simons DG: Myofascial Pain and Dysfunction: The Trigger Point Manual, Volume 2. Williams & Wilkins, Baltimore, 1992.
“Muscle pain is not skin pain” Jay Shah, MD
Myofascial Trigger Points (MTrPs) Active – cause a clinical pain complaint or other abnormal sensory symptoms Latent – show all the other characteristics of active MTrPs, except that they’re pain free
Muscle Pain Aching and cramping Difficult to localize and refers to other deep somatic tissues (fascia, muscle, joints) Muscle nociceptive activity is processed differently in the CNS Inhibited more strongly by descending pain-modulating pathways than cutaneous pain
Symptoms Local & referred pain Pain with iso contraction Stiffness, limited ROM Muscle weakness Paresthesia & numbness Propriocpetive disturbance Autonomic dysfunction Physical Findings Local Tenderness Single or multiple muscles Palpable nodules Firm or Taut Bands “twitch response” (LTR) Jump sign Muscle shortening Limited joint motion Muscle Weakness
Motor Strength Testing 5 = Normal, full ROM vs. gravity, max resistance 4 = Good, full ROM vs. gravity, moderate resistance 3 = Fair, full ROM vs. gravity, no resistance 2 = Poor, full ROM, gravity eliminated 1 = Trace 0 = No activity
Integral Components of SIJ motion Form closure: joint surfaces congruently fit together Force closure: muscles & ligaments provide force to withstand load Motor control: timing & sequencing of muscle activation & release Emotion & awareness: emotions can influence motor control Vleeming A, et al. Spine 1990;15:133-5
SIJ Assessment (J.Rittenberg. Photos from practice & personal files used with permission)
Sacroiliac Joint Provocative Tests: SIJ border tenderness Patrick’s test Gaenslen’s test Prone hip extension Compression testing Fortin J, et al, Spine 1994;19:
Sacroiliac Joint Injections Bogduk N, MJA 2004;19:79-83.
Lumbar Spinal Stenosis: Posture Akuthota, V. Pathogenesis of lumbar spinal stenosis pain. Phys Med Rehab Clin N Am 14:17-28, With permission. J. Rittenberg. Used with permission.
BI-Level Central Neurovascular Claudication Porter RW. Spine 1996;21: Onset with walking “Heavy” sensation Variability Attempt to increase flexion Stooped posture
Lumbar Spinal Stenosis: Simian Stance Posterior pelvic tile Hips, knees flexed Hands face backwards Hip and psoas tight Gluteus and piriformis inhibited Gait: lumbar flexion
Geraci, M. Rehabilitation of the hip and pelvis. In: Kibler WB. Functional Rehab Sports Musculoskeletal Med; Aspen Publishers,1998. With permission. Weak and Inhibited Muscles
Finding Balance UnderactiveOveractiveShortened StabiliserSynergistAntagonist Glut MediusTFL, QL, PiriformisThigh adductors Glut MaximusIliocast, HamstringIliopsoas, Rec Fem Lower TrapeziusLevator ScapulaePectoralis Major Upper trapezius Geraci, M. Rehabilitation of the hip and pelvis. In: Kibler WB. Functional Rehab Sports Musculoskeletal Med; Aspen Publishers,1998. With permission.
trapezius and cercival spine
Cervical & Scapular Dysfunction (Janda 2002)
APS: LBP Guidelines Categorize the condition –Nonspecific low back pain? –Back pain associated with neurologic deficits, radiculopathy or spinal stenosis? –Back pain associated with an alternate cause? Identify patients who require urgent surgical evaluation Chou R, et al. Ann Intern Med. 2007;147:
Acute Low Back Pain ‘Red Flags’ Cauda equina syndrome? Cancer? Infection? Fracture? –Confirmation of red flag conditions may require Lab testing [complete blood count (CBC)/erythrocyte sedimentation rate (ESR)/C-reactive protein (CRP)/ urinalysis (UA) and PSA when appropriate] Medical imaging [lumbosacral (LS) radiographs/computed tomography (CT)/magnetic resonance imaging (MRI)] Test results may indicate need for emergent surgical referral Chou R, et al. Ann Intern Med. 2007;147: Chou R, et al. Lancet. 2009;373:
Pharmacologic Interventions Acute Low Back Pain DrugNet benefit Level of evidence AcetaminophenSmall to moderateFair NSAIDsModerateGood Skeletal muscle relaxants Moderate (for acute LBP only)Good Chou R, et al. Ann Intern Med. 2007;147:
Chou R, et al. Ann Intern Med. 2007;147: Guideline Highlights 1.Conduct a focused history and physical examination –Assess severity of baseline pain and functional deficits 2.Evaluation of psychosocial risk factors is essential to predict the risk for chronic, disabling low back pain 3.Limit use of diagnostic imaging and testing –Except in patients with signs of severe or progressive underlying disease or those with neurologic deficits
Recommendation 6 ACP/APS Guidelines 2007 Clinicians should consider the use of medications with proven benefits in conjunction with back care information and self-care. Clinicians should assess the severity of baseline pain and functional deficits, potential benefits, risks, and relative lack of long-term efficacy and safety data before initiating therapy. For most patients, first-line medication options are acetaminophen or NSAIDs. (Strong recommendation, moderate-quality evidence) Chou R, et al. Ann Intern Med. 2007;147:
Pharmacologic Interventions DrugNet benefitLevel of evidence AcetaminophenSmall to moderateFair NSAIDsModerateGood Skeletal muscle relaxants Moderate (for acute LBP only)Good Tricyclic antidepressants Small to moderate (for chronic LBP only) Good Opioids and tramadolModerateFair BenzodiazepinesModerateFair Antiepileptic medications Small ( for gabapentin in patients with radiculopathy only) Unable to estimate topiramate Fair for gabapentin to poor for topiramate Systemic steroidsNo benefitGood Chou R, et al. J Pain. 2009;10:
Summary Comprehensive, but focused Efficient Exam should be easy on you and the patient Great opportunity to initiate a therapeutic relationship and dialogue Use a “good” exam to improve outcomes and identify deficits or impairments