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Spine and spinal cord injuries. Spine and spinal cord injuries mechanisms of spine injuries flexion injury extension injury axial loading rotation injury.

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Presentation on theme: "Spine and spinal cord injuries. Spine and spinal cord injuries mechanisms of spine injuries flexion injury extension injury axial loading rotation injury."— Presentation transcript:

1 Spine and spinal cord injuries

2 Spine and spinal cord injuries mechanisms of spine injuries flexion injury extension injury axial loading rotation injury

3 Spine and spinal cord injuries mechanism flexion injury Cervical and thoracic spine thoracolumbar junction Vertebral body fracture with anterior wedging extension injury Cervical spine ! Fractures of posterior elements (posterior column), with ligaments disruption subluxations and spodylolisthesis NOTE! In this mechanism the spinal cord injury and root compression may occur without evident osseous changes visible on plain films

4 Spine and spinal cord injuries mechanism axial loading C spine – diving L-S spine – jumping compression („burst”) fractures of vertebral bodies lateral element (processes fractures) and articular pillar fractures rotation injury Usually in combination with flexion-extension injury Lateral mass fractures, luxations, uncovertebral dislocations

5 Spine and spinal cord injuries effects oseous changes, fractures posttraumatic instabilities and spondylolistheses posttraumatic disc herniations spinal cord contusion spinal cord compression vascular spinal cord injury

6 Spine and spinal cord injuries Osseous changes – most important sings atlantooccipital dislocation common, but not always fatal on lateral plain film: dens – basion distance > 12,5 mm most common to C1 is a bilateral vertical fracture through the neural arch = a Jefferson fracture = a burst fracture that involves both anterior and posterior arches

7 Posterior arch C1 fracture Jefferson fracture – of C1 anterior and posterior arches : The burst mechamism causes the lateral displacement of the lateral parts of C1

8 Jefferson fracture

9 C2 fracture, is often combined with luxation of C1 – may efect with spinal cord compression; CT with MPR and 3D reconstructions is advisable Odontoid – dense fracture

10 Flexion-extension mechanism: avulsion fracture C5 spinous process fracture C7 C 5C 7 C 2 on CT

11 rotation mechanism: C1- posterior arch, body of C2 and odontoid fractures - luxation

12 Spine and spinal cord injuries Osseous changes – most important sings Odontoid (dens) fractures Usually at the base of dens – poorly visible on axial CT – MPRs and 3Ds necessary! Hangman’s fracture bilateral transpedicle with the separation of neural arch from the vertebral body and spondylolisthesis, dens and ligaments are usually intact

13 Transverse C2 body fracture – Hangman’s fracture - with bilateral arch-body separation – the result is instability

14 C2 dens apex fracture unvisible on plain film!

15 C2 dens apex fracture unvisible on axial CT!

16 C2 dens apex fracture visible on MPR!

17 C2 dens apex fracture visible on 3D SSD! Spinal canal stenosis can be evaluated

18 Spine and spinal cord injuries Osseous changes – most important sings C1-C2 luxations in all methods: plain films, CT and MR: normal distance between anterior arch of C1 and dens of C2 is 1 to 3 mm distances between dens and lateral masses of C1 should be equal luxation – spondylolisthesis is important when larger than 3-4 mm NOTE ! Arches and processes fractures are common at C6, i C7 and in L-S spine

19 C1-C2 luxation (Rheumatoid arthritis) malum suboccipitale C1-C2 luxation plain film (Rheumatoid arthritis) malum suboccipitale > 3 mm

20 C1-C2 luxation (Rheumatoid arthritis) malum suboccipitale C1-C2 luxation MR T1w (Rheumatoid arthritis) malum suboccipitale

21 C1-C2 luxation (Rheumatoid arthritis) malum suboccipitale C1-C2 luxation MR T2w (Rheumatoid arthritis) malum suboccipitale

22 Body and arches of C2 + arches of C3, luxation (CT MPR and 3D SSD)

23 spondylolisthesis

24 Transpedical fracture with spondylolisthesis C6-C7

25 Transpedical fracture with spondylolisthesis C6-C7 (CT MPR)

26 Spine and spinal cord injuries Osseous changes – most important sings Compression („burst”) and multifragment fractures of vertebral bodies may cause free osseuos fragments protrusion to the spinal canal, resulting with spinal cord damage or/and compression free or protruding osseous fragments are well seen on CT especially with MPRs and 3D SSD reconstructions Spiral or multislice CT can be used to evaluate the spinal canal stenosis caused by fractures or spondylolistheses and precisely define the location of the most threatening fragments

27 Compression „burst” fracture (C5)

28 Burst fracture L-S 3D SSD

29 Spine and spinal cord injuries Posttraumatic spinal cord compression is caused by: · haematoma - epidural - or inside the cord · disc herniation · spondylolisthesis or luxation · osseous fragments

30 Spine and spinal cord injuries  CT visualize - osseous changes - spinal canal stenosis - disc herniation - rarely haematoma

31 Spine and spinal cord injuries · MR visualize - cord oedema - haematoma, contusion, vascular injury - disc damage and herniation - osseous changes are poorly visualized on MR - use CT! - Instead MR perfectly visualizes spinal cord displacement, compression and focal lesions !

32 Posttraumatic cord oedema

33 Spine and spinal cord injuries Spinal cord contusion · bleeding foci within the cord grey matter - best visible on MR - on CT visible while massive - on CT hyperdense foci inside the cord (60 – 70 HU) - on MR image is phase dependent

34 Spine and spinal cord injuries Spinal cord contusion MR # acute phase 1- 4 days – slightly hyperintense on T2w, hypointense w T1w # subacute phase 2-4 days up to 3 weeks – slightly hyperintense on T2w, hyperintense on T1w # chronic phase more than 3 weeks – strongly hyperintense on T1 & T2w, accompanied by: - oedema – high signal on T2w - malacia – inhomogenous, high signal on T2w # residual – glial scar and myelomalacia

35 Spine and spinal cord injuries vascular spinal cord injury - caused by compression, vasospasm or damage of the spinal vessels - resulting ischaemia leeds to necrosis and myelomalacia - Well seen on MR: hypointense on T1w and hyperintense on T2w

36 Contusion, oedema, bleeding, disc herniation

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