2Nutritional deficiency diseasesSmall ruminantsDr Abd Elghany Hefnawy
3(not stored in tissues must have Constant supple) Wool eatingParakeratosisFallingdiseaseEnzooticataxiaGoiterWLDAnemiaWMDAnemiaSwybackZnHemoglobinueriaIronIodineCobaltSeCuPParturientparesisTrace elementsK+HypokalemiaCaMineralsNutritional elementsElectrolytesNa+HyponatremiaLactationtetanyMgVitaminsCl-HypocholermiaWater soluble(not stored in tissues must have Constant supple)Fat soluble(stored in the tissue)Non-B complexB- ComplexANight blindnessPolioencepha-lomaleciaB-1(thiamin)C (Ascorbic acid)KHypoprothrombinaemiaSkin lesionsB-2 (riboflavin)ScurvyEAnaemiaB-12(cobalamin)NMD or WMDMegaloblastanaemiaFolic acidDRickets or osteomaleciaNutritional elements and its deficiency
4Vit.& Minerals Facts 1-Vit. are essential organic nutrients. 2-Minerals are essential inorganic nutrients2-Required in small amounts.3-Vit. cannot be synthesized by the body.4-Vit. Must be obtained by outside sources like diet, rumen bacteria & sun.5-Required for growth, maintenance, reproduction and lactation.
5“Osteodystrophic diseases” “Rickets or osteomalecia Vitamin-D Deficiency“Osteodystrophic diseases”“Rickets or osteomaleciaDr/ Abd Elghany Hefnawy
6Toxicity of melybodnum Heavy infestation with GIT. Nematodes EtiologyPrimarycauseInsufficient solarirradiationSecondarycauseToxicity of melybodnumor copper chloridDeficient vit-Din food intakeHeavy infestation with GIT. Nematodesincrease carotenintake
7Etiology and predisposing factors PathogenesisClinical findings
121,25 DHCC (Active form of vit D Transportation and absorption of intestinal calciumRegulation of phosphorous absorption and metabolism specially it’s loss from the kidney1,25 DHCC(Active form of vit DMineralization of bone.Vitamin-D may have a rolein the immune sysytemSo any deficiency of vit. D leading to disturbance in calcium and phosphorus absorption and metabolism(Osteoporosis).
13Types of osteoporosis Rickets Stunted bone growth Osteomalicia Matrix osteoporosis(bony atrophy)
14Low amount of protienetious Substances (Matrix)of the bone Good Matrix osteoporosis(bony atrophy)Adult andyoungLow amount of protienetiousSubstances (Matrix)of the boneGoodmineralizationRicketsYoung orgrowingDefect in the mineralizationMatrix is presentnormallyStunted bonegrowthOsteomaliciaSimillar to rickets butaffect adult animalsDefect in the mineralization&matrix of the boneYoung
15-Dyspnoea if chest bone affected Clinical Signs-Reduced the productivity & reproductive of the animal.-Decrease in appetite and food utilization-Lameness which is more common in the forelegs accompanied in young animals by bending of long bone and enlargement of the joints.-Dyspnoea if chest bone affected-Curvature oflong bone.-Decrease thefertility-Arching of back.
16Diagnosis -Laboratory diagnosis -History and clinical signs. -low calciumandphosphorouslevel.Fecalanalysis(GI. Parasitism)-High levelofalkalinphosphatase5.7units (N-2.5)low levelof vit D
17Treatment and control Administration of vit.D Supplementation of calciumand phosphorousin the dietExposure to thesunlightTreatment and controlDry yeast isprobablysimpleand cheapsource ofvitamin DFish liver oilis goodsource ofvit-D (4,5)Sun-dried hay isgood sourceof vit-D2