Presentation on theme: "Dr/ Mervat El-Shabrawy"— Presentation transcript:
1Dr/ Mervat El-Shabrawy CataractDr/ Mervat El-ShabrawyAss. Prof. of Ophthalmology- Faculty of MedicineSuez Canal University
2What is the origin of the term Cataract? Definition:Opacity of the lens.What is the origin of the term Cataract?In the past, there is a wrong believe that the humor of the brain falling inside the eye.
4(Surgical classification) (2) According to the age(Surgical classification)SoftHardBelow 25 Y.O.After 25 Y.O.(3) According to the siteCapsularNuclearCortical
5* Congenital Cataract * N.B.:- Congenital Cataract: = Lens opacity present at birth.- Infantile Cataract: = Lens opacity that develop during the 1st year of life.- These 2 terms are fused together & some physicians prefer the term developmental cataract.
71- Anterior Polar Cataract Etiology:* Congenital- Bilateral.- Occurring at the time of the lens vesicle separation from surface ectoderm resulting in delayed formation ant. Chamber with contact between ant. Pole & cornea.* Acquired- Unilateral.- Due to small central perforation of corneal ulcer.
8* Symptoms (By parents) Clinical Picture:* Symptoms (By parents)- White dot at the center of the pupil* Signs- Small white opacity in the center of the pupil at theant. pole of the lens.- No marked visual affection (as it is far from the nodal point).
92- Posterior Polar Cataract Etiology:- Persistence of hyaloid artery.Clinical Picture:* Symptoms: Usually no symptoms.* Signs:- Disc shaped opacity at the post. Pole.- Marked visual affection due to it is near to nodal point.
103- polar Cataract Clinical Picture: Etiology:1) Hereditary.2) Malnutrition of the mother during pregnancy.Clinical Picture:* Symptoms:- White colour of the pupil.- Child doesn’t see well.
11* Signs:1) Diffuse illumination:- Central disc opacity with clear lens around it.- There may be linear extensions.2) Slit lamb examination:- Opacity arranged in one or many concentric zones.- lens substance outside & inside the affected zones areclear.3) Red Reflex:- Dim central.- Bright peripheral
124- Coronary Cataract Developmental Cataract. Appears at puberty due to defect in the growth of lens fibers at childhood.Appear clinically as a corona of oval opacities near the periphery.
136- Total Cataract 5- Punctate Cataract Developmental Cataract. Multiple small bluish dots scattered all over the lens.6- Total CataractThe lens is opaque.Etiology: Infection of the mother in the first 3 months of pregnancy by Rubella virus.
14Complications of congenital cataract: Occurs only if the opacity affects the vision:- Unilateral cataract: Squinting of the eye.- Bilateral cataract: Nystagmus.
15Management of a case of congenital Cataract EvaluationIndication of surgerySurgical techniquesCorrection of Aphakia
16A- The Eyes: (1) Evaluation: - unilateral versus Bilateral. - Density. - Morphology.- Visual functions.Q) How can you test visual acuity during infancy?- By special tests as Preferential looking.B- The patient:Examined by Pediatrician for any congenital anomaly.C- Parents:May gives clue to the etiology.
17(2) Indications of surgery: A) Bilateral advanced cataract: surgery at once.B) Bilateral & fundus can be seen: don’t require surgery.C- Bilateral & fundus seen only by indirect ophthalmoscope:- Good near vision: Postpone surgery.- Poor near vision: Surgery at once.
18(3) Surgical techniques: Old Operations:1- Needling operation.2- Needling & curette op.* Complications of old operations:- dense after cataract.- Secondary iritis.- Secondary glaucoma.- Delayed unexplained retinal detachment.
20(4) Correction of Aphakia 1- Contact lens.2- Epikeratophakia.Q) Why we don’t do ICCE???- Presence of strong zonules & strong hyaloid capsular ligament.
21Acquired Cataract 1- Senile Cataract: Def.- Bilateral progressive lens opacity affecting the old people not suffering from local or general diseases.Etiology: Unknown But theories:1- Disturbance of lens capsule permeability.2- Disturbance in lens metabolism.3- Ultraviolet rays.4- Endocrinal disturbance.5- Hereditary.
22Classification: Pathology: Hydration. Coagulation of lens proteins. - Cortical: Most common type.- Nuclear.- Cortico-nuclear.Pathology:Hydration.Coagulation of lens proteins.
23Senile Cortical Cataract C/P:- Symptoms –1) gradual painless diminution of vision.2) Fixed black spots.3) Diminution of vision at night??.4) Uniocular diplopia or polyopia.5) Index myopia??.6) Change in colour value.
24The lens is not totally opaque - Signs -hypermatureMatureImmatureThe lens is not totally opaqueLens loses water & shrinks (Typical)The whole cortex become opaque in front & behind the nucleus.Intumescent.Incipient.PathologyThe opacity inc. associated with breakdown of lens proteins.Opacity begins as sectors from the periphery of the cortex in front & behind the nucleusLens show:- Thickened caps.- White dots of Ca.- Yellow dots of cholesterol.Uniformly grayish white.Fluid vacuolesOpacities grey against black backgroundOblique illumination+ve-veIris shadowDeepNormalShallowAnt. Cham.Absent.AbsentSeen with difficultyBlack sectors against red backgroundR. Reflex.H.M.Marked decrease.Slightly dec. especially at night.V.AMay be raised.May be raisedTension
25N.B.Secondary glaucoma in intumescent cataract due to Phacomorphic glaucoma.Secondary glaucoma due to hypermature cataract is called phacolytic glaoucoma.Morgagnian cataract: one of hypermature stage. If present = Atypical hypermature.Incipient cataract & senile nuclear sclerosis = Grey pupil. Differentiated by red reflex.
26Complications Sec. glaucoma. Subluxation or dislocation of the lens due to degenerations of the zonules.Sec. uveitis.Endophthalmitis phacoanaphylactica.
27Senile Nuclear Cataract Senile nuclear sclerosis:- Physiological process.- Old lens fibers becomes compressed towards the center with loss water. So nucleus increase in size & volume but remains transparent.
28Senile nuclear cataract - Pathological senile nuclear sclerosis in which the transparency is affected.Symptoms:1) Gradual diminution of vision.2) Defective vision at day “Photopic vision”3) Index myopia.
29Signs: Do not the stages of cortical cataract. Red reflex: red ring around opacity.Due to deposition of melanin formed tyrosine, Lens may have a color:1- Black: Cataract nigra.2- Brown: Cataracta Brunescence.
30- Differential Diagnosis: 1- Gradual painless diminution of vision: 1ry optic atrophyO.A. glaucomaSenile cataractCharacter> 35 Y.O.Males more.Of the cause.> 50 Y.O.EqualField lose>50 Y.O.Equal.Fixed black spots.(1) History:1- Age:2- Sex:3- Other symptoms:Normal.Absent with dilated pupil.NormalWhite disc. shallow cupPeripheral contractionNormal outflow.PresentGlauc. Disc cupping.Glauc. Field defects.Raised.Impaired outflowGrayishAbnormalNormal outflow(2) Signs:1- Pupil color:2- D. Light reflex:3- Red reflex:4- Fundus:5- field:6- Tension:7- Tomography:2- Incipient from senile nuclear sclerosis: By R. reflex.
31Treatment I.C.C.E with or without I.O.L implantation. E.C.C.E // // // // //Phaco-emulsification with or without I.O.L implantation.
32According to the type of trauma Usually cause post. Cortical Traumatic CataractLens opacity as a result of ocular trauma.According to the type of traumaSharp traumaBlunt traumaSmall but highspeed F.BLinear opacityUsually cause post. Corticalrosette shaped Cat.Large but lowspeed F.BCloudy swollen withrupture capsule
33Complications: - Iritis. - 2ry glaucoma Treatment: 1- Medical: Immediately after trauma,* Rest * Atropine * Cortisone.2- Surgical: Later,* If light projection is good, indicating good retinal function.
34N.B.1 : In Blunt trauma we may find Vossius ring “ Brown ring on the ant. Lens capsule cue to impress of the pupillary border or the iris on the lens.N.B.2 : Electrical trauma:- Due to passage of high electric current through the body.- May occur within days or after years.- Pathology: Change in lens capsule permeability.- Morphology: Subcapsular vacuoles causing opacity.
35Complicated Cataract Lens opacity due to local disease in the eye. Etiology:- Conjunctiva.- Cornea: Sever corneal ulcer.- Sclera: Acute scleritis.- Uveal tract: Iridocyclitis.- Lens: Subluxation & dislocation.- Glaucoma.- Retina: R.D. , R.P. & retinoblastoma.
36Pathology:- Interference with lens nutrition.- Effect of toxins present in aqueous or vitreous.N.B. Why lens opacity is usually posterior cortical??Thin posterior capsule.No protective subcaosular epithelium.
37Types:1) Anterior segment.2) Posterior segment.In both types: When the whole lens become opaque, it may be difficult to differentiate from senile cataract.Treatment:Cataract operation.According to the cause.
38Pathological Cataract Lens opacity due to systemic disease.Diabetic Cataract:a- True:- Young patient with uncontrolled D.M.- Milky white dots under ant. & post. capsule.- Soon, the whole lens become opaque.b- False: The same as senile but earlier.* Treatment: Cataract extraction after control of diabetes.
392) Hypoparathyroid Cataract: - Low serum calcium which is important for lens metabolism.- Subcapsular opacities “Malformation of new lens fibers”.- Treatment: E.C.C.E & I.O.L.3) Ankylostoma Cataract: Due to:- Anemia “Malnutrition of the lens”.- Ankylostoma toxins.
40Occupational cataract Deficiency cataract- Interfere with normal lens nutrition or metabolism.a- Galactosaemia.b- Mannosidosis.c- Fabry’s disease.d- Deficiency of vit. C or D.Occupational cataract- Glass blowers cataract.
41Radiation Cataract Toxic cataract X-rays & Gamma rays Pathology: * Injury to the lens fibers directly.* Injury to the cilliary body:Abnormal aqueous Disturbance of lens nutrition Opacity in posterior cortex.Toxic cataract- Anterior subcapsular: Miotics, Gold.Posterior subcapsular: Corticosteroids“ topically or systematically”.