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Secondary genetic lesions in acute myeloid leukemia with inv(16) or t(16;16): a study of the German-Austrian AML Study Group (AMLSG) by Peter Paschka, Juan Du, Richard F. Schlenk, Verena I. Gaidzik, Lars Bullinger, Andrea Corbacioglu, Daniela Späth, Sabine Kayser, Brigitte Schlegelberger, Jürgen Krauter, Arnold Ganser, Claus-Henning Köhne, Gerhard Held, Marie von Lilienfeld- Toal, Heinz Kirchen, Mathias Rummel, Katharina Götze, Heinz-August Horst, Mark Ringhoffer, Michael Lübbert, Mohammed Wattad, Helmut R. Salih, Andrea Kündgen, Hartmut Döhner, and Konstanze Döhner Blood Volume 121(1):170-177 January 3, 2013 ©2013 by American Society of Hematology
Pie chart illustrating the genetic heterogeneity and coexistence of the distinct secondary genetic abnormalities in AML with inv (16). Paschka P et al. Blood 2013;121:170-177 ©2013 by American Society of Hematology
Impact of trisomy 22 on RFS and OS. (A) RFS. (B) OS. Paschka P et al. Blood 2013;121:170-177 ©2013 by American Society of Hematology
Impact of trisomy 8 on OS. Paschka P et al. Blood 2013;121:170-177 ©2013 by American Society of Hematology
Impact of KIT mutations on RFS. Paschka P et al. Blood 2013;121:170-177 ©2013 by American Society of Hematology
Impact of FLT3 mutations on OS. Paschka P et al. Blood 2013;121:170-177 ©2013 by American Society of Hematology
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