We think you have liked this presentation. If you wish to download it, please recommend it to your friends in any social system. Share buttons are a little bit lower. Thank you!
Presentation is loading. Please wait.
Published byIvan Rodwell
Modified over 2 years ago
Gallstones Alcohol Post-ERCP Drugs Hypertriglyceridemia
intrapancreatic enzyme activation acinar cell injury intrapancreatic inflammatory reaction effects of activated proteolytic enzymes and cytokines on distant organs
Endotoxins, exotoxins, viral infections, ischemia, anoxia and direct trauma activate the zymogens. Activation is mediated by lysosomal hydrolases, such as Cathepsin B. Acinar cell injury
activation, chemoattraction, and sequestration of neutrophils intrapancreatic inflammatory reaction
Activated proteolytic enzymes digest pancreatic & peripancreatic tissues. Activation of other enzymesEnzymes digest cellular membranes. Proteolysis, edema, interstitial edema, vascular damage, coagulation & fat necrosis, and parenchymal cell necrosis
Bradykinin peptides, vasoactive substances and histamine vasodilation, increased vascular permeability and edema cellular injury and death
Normal anatomy and histology. PANCREAS PANCREATITIS ACUTE (VERY SERIOUS) CHRONIC.
Integrated Pathology Practical Normal anatomy and histology.
Acute and Chronic Inflammation. W.B. Saunders Company items and derived items Copyright (c) 1999 by W.B. Saunders Company.
Chapter 4 Injury, Inflammation, and Repair. Review of Structure and Function The body is capable of undergoing dynamic changes to carry out body functions.
This lecture was conducted during the Nephrology Unit Grand Ground by a Sub- intern under Nephrology Division, Department of Medicine in King Saud University.
INFLAMMATION PLASMA PROTEIN–DERIVED MEDIATORS Of Inflammation.
GROUP D Mamba-Medinilla. COMMON CAUSES Gallstones Alcohol Intake Hypertriglyceridemia ERCP especially after biliary manometry Blunt abdominal trauma Postoperative.
THE ACUTE INFLAMMATION AND ACUTE-PHASE RESPONSE. Innate immune mechanisms establish a state of inflammation at sites of infection.
SHOCK Background concept Shock is a severe pathological process under the effect of various types of etiological factors, characterized by acute circulatory.
1 Dr. Maha Arafah INFLAMMATION AND REPAIR Lecture 3 Monday, October 25, :00-12:00 am Dr. Maha Arafah Assistant Professor Department of Pathology.
1 INFLAMMATION AND REPAIR Lecture 3 Chemical Mediators in Inflammation and Patterns of Acute Inflammation Foundation block: pathology 2012 Dr. Maha Arafah.
Introduction to pathology Inflammation lecture 1 Dr H Awad FRCPath First trimester 2015/16.
M.Prasad Naidu MSc Medical Biochemistry, Ph.D.Research Scholar.
STARTER.... COMPLETE THE FLOW CHART AND GLOSSARY TERMS.
1 INFLAMMATION AND REPAIR Lecture 3 Chemical Mediators in Inflammation and Patterns of Acute Inflammation Foundation block: pathology INFLAMMATION AND.
The Cell-Derived Mediators of Chemical Mediators of Inflammation Presented by Sara M. Al-Shaker Wed. 5/11/2008 King Saud University Riyadh, KSA.
Chemical Mediators and Regulators of Inflammation 1 Dr. Hiba Wazeer Al Zou’bi.
Jahangir Sadeghi MD 1) Inflammation 2) Infection We approach to RED Eye through pathology.
Al-Qassim University Faculty of Medicine Phase II – Year III GIT Block (CMD332) EXOCRINE PANCREASE Lecture Dr. Gamal Hamra Wednesday 01/12/1430 (18/11/2009)
Hemodynamics & Cellular Phases Types of Exudates (Role of Chemical mediators in inflammation)
Pancreatitis Dr. Gehan Mohamed Dr. Abdelaty Shawky.
Cell injury-3. Morphology of irreversible cell injury.
Complement Synthesis and attachment of specific antibody to invading microorganisms does not directly lead to destruction. It is a "label" that.
Biology of Disease CH0576 The Inflammatory Response I Dr Stephen Todryk.
UNIVERSITY COLLEGE OF HUMANITIES Technical Lab Analysis Department. Lectures of Histopathology. INFLAMMATION NOVEMBER –
Disorders of Immune System - Hypersensitivity Reactions: Immune response to exogenous antigens - Autoimmune diseases: Immune reactions against self antigens.
Irreversible cell injury Dr Heyam Awad FRCPath. Irreversible injury Necrosis Apoptosis.
Activity… Design an emergency plan to protect people (ex. LRHS students or people attending a concert) from an attack. If an attack were to occur,
Inflammation Dr. Ahmad Hameed MBBS,DCP, M.Phil. Chemical Mediators and regulators of inflammation Chemical mediators that are responsible for vascular.
The Eight Phases of Inflammation. Injury Injury Utrastructural Changes Utrastructural Changes Metabolic (Hypoxic) Changes Metabolic (Hypoxic) Changes.
Inflammation and Cell Damage Peer Support 2014 Michael Iveson and Emily Hodgson.
CPB & Effects on the Lung Seoul National University Hospital Department of Thoracic & Cardiovascular Surgery.
Tutorial 1 Inflammation and cellular responses. Inflammation Is a protective response The body’s response to injury Interwoven with the repair process.
Lecture 36- Pancreatitis a. Define b. Discuss the causes and clinical features c. Describe the morphological changes. A.
Week 2 Cell Injury and Cell Death Dr.İ.Taci Cangül Bursa-2008.
Chemical Mediators 2 Dr. Hiba Wazeer Al Zou’bi. Complement Activation Pathways 2.
Acute and Chronic Inflammation. Introduction Injurious stimuli cause a protective vascular connective tissue reaction called “inflammation” Dilute Destroy.
Inflammation Chapter 2 Ms. Harris Pathophysiology Fall 2009.
Ultrasound Rotation Presentation Missy Purcell 3/5/10.
Chemical Mediators of Inflammation Dr. Raid Jastania.
CHEMICAL MEDIATORS OF INFLAMMATION Definition: Any messenger that acts on blood vessels, inflammatory cells, or other cells to contribute to an inflammatory.
Chapter 5 “ human body is continually exposed to conditions that result in damage” Sunlight, pollutants, trauma Infectious agents – viruses, bacteria,
Necrosis Definition: Causes:. Development of Necrosis (2 mechanisms) irreversible damage to mitochondria (failure of ATP generation) ↓ anaerobic respiration.
Section 3 Inflammatory Mediator. 1. Definition: It is the chemical substances which cause or participate in inflammation (1) Mediators originate either.
CHEMICAL MEDIATORS OF INFLAMMATION تحت اشراف أ.د/ كوكب طالب/ محمد محمود محمد مشالى عبد الحميد رقم/07274 Cairo University Faculty of Veterinary Medicine.
CHAPTER10 Biomaterial Implantation and Acute Inflammation 10.1 Introduction: Overview of Innate and Acquired Immunity Wound healing Implantation --- assault.
Inflammation The process of inflammation initiates from tissue injury or from foreign presence its initiation is triggered by the production of: a) chemokine.
Exocrine Pancreas Kimiko Suzue, MD PhD.
INFLAMMATION 1. Inflammation * Definition: Local vascular, lymphatic and cellular reactions of living tissue against an irritant. *Inflammation is a protective.
Pick up the colored cards on the cabinet: Ms. Joachim Pathophysiology.
© 2017 SlidePlayer.com Inc. All rights reserved.