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ER Conference Cacnio * Calimlim * Castillo * Cauilan * Causapin * Chu, D * Chu, H * Co, J * Co, V * Cosico * De Leon.

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Presentation on theme: "ER Conference Cacnio * Calimlim * Castillo * Cauilan * Causapin * Chu, D * Chu, H * Co, J * Co, V * Cosico * De Leon."— Presentation transcript:

1 ER Conference Cacnio * Calimlim * Castillo * Cauilan * Causapin * Chu, D * Chu, H * Co, J * Co, V * Cosico * De Leon

2 General Data V.N. 69 y/o Male Catholic Married Tondo, Manila Kagawad
Patient; 70%

3 Chief Complaint : Dyspnea

4 History of Present Illness
15 years PTA 11 years 8 years Occasional exertional dyspnea (SOB on walking 5 blocks Cough productive of whitish sputum (1 teaspoon full) (-) orthopnea, PND, chest pain, fever, weight loss Spontaneously resolve but would recur No consult Dyspnea, prod cough Progression of dyspnea (3 blocks) (-) other associated symptoms Consulted: Chest x-ray done (unrecalled result); Unrecalled diagnosis; unrecalled medications during symptoms relief No follow up done 15 years- cough of <1 weej frequency Cough, blood streaked sputum, undocumented fever Dyspnea at rest Consulted: CXR (unrecalled result); unrecalled antibiotics & other meds for 7 days Relief of blood streaked sputum, fever, dyspnea at rest

5 History of Present Illness
Persistence of exertional dyspnea (3 blocks), productive cough of whitish / greenish to grayish sputum & dyspnea at rest (3-5 times/year); Tx: Procaterol 25mcg/tab prn Interim Dyspnea, prod cough 2 weeks PTA Cough with increased production of grayish sputum 2 pillow orthopnea (-) PND, chest pain, fever, weight loss, night sweats, malaise, anorexia No medications or consultations were done 1 day PTA Persistence of symptoms Dyspnea at rest Dyspnea while walking about 3 blocks away from home and climbing the stairs of 4-5 steps. This was accompanied by productive cough of grayish sputum amounting to 2 teaspoonfuls. Patient denied any presence of fever or vomiting. No consult was done or medications taken. dyspnea progressed now experienced even at rest. Still, no consult done or medications taken. Few hours PTA, there was recurrence of above symptom. Patient took Procaterol HCl (Meptin) 25 mcg/tab previously prescribed by a pulmonologist which he usually take as needed for difficulty of breathing. Patient denied any relief from the said medication. Persistence of the symptom prompted consult and subsequent admission. Few hours PTA Persistence of symptoms Self medicated with Procaterol 25mcg/tab  no relief Consulted at UST ERCD Admission

6 Past Medical History Medical
(+) Hypertension (2005) UBP 130/80, HBP 180/100 unrecalled drug, non-compliant (+) DM Type 2 (2007) unrecalled drug, non-compliant (-) PTB, (-) thyroid disease, (-) asthma, (-) cancer Surgical none Allergies, Blood transfusion

7 Family History (+) HPN-father, mother (+) DM- father
(+) Heart disease- mother (+) PTB-sister (-) asthma, thyroid disease, cancer

8 Personal and Social History
Smoker-168 pack years; (stopped April, 2009) Alcoholic drinker (1 bottle of gin/day since 16 years old) Denies illicit drug use Mixed diet more of meat and fish No tattoo Alcohol content

9 Review of System (-) pallor, (-) fatigue
(-) jaundice, (-) hypo/hyperpigmentation (+) blurring of vision, (-) lacrimation, (-) eye pain, redness, (-) itchiness (-) deafness, discharge, tenderness (-) colds, (-) discharge (-) epistaxis (-) sore throat (-) sores, fissures, bleeding gums (-) neck stiffness, limitation of movement, masses (-) constipation, (-) abdominal pain, (-) diarrhea, (-) hematochezia, (-) melena, (-) nausea, (-) vomiting

10 Review of System (-) oliguria, (-) hematuria, (-) dysuria (-) urgency (-) frequency (-) discharge (-) muscle pain (-) palpitation, (-) polydipsia, (-) polyuria, (-) polyphagia, (-) heat-cold intolerance (-) Poor wound healing, (-) easy bruisability (-) Sensory deficit, (-) seizures (-) depression, (-) hallucinations

11 Physical Examination Conscious, coherent, wheelchair borne, purse-lip breathing, speaks in phrases BP 130/80mmHg (sitting) 120/80mmHg (lying), PR 82, regular, RR 26 regular, cpm, T 37.3°C Warm moist skin, no active dermatoses Pink palpebral conjunctiva, anicteric sclera, no ptosis, isocoric at 2-3mm ERTL, (+) ROR, distinct margins, no edema, 2:3 A:V ratio OU; (+) dot blot hemorrhage on OS

12 Physical Examination Nasal septum midline, turbinates not congested, no alar flaring; No tragal tenderness, no aural discharge, R and L tympanic membrane intact; (-) central cyanosis, moist buccal mucosa, non- hyperemic PPW, tonsils not enlarged; (+) tense sternocleidomastoid, trachea deviated to the left, supple neck, thyroid not enlarged, non- palpable cervical lymphadenopathies; (-) carotid bruit, neck veins not distended

13 Physical Examination Symmetrical chest expansion, (+) supraclavicular and intercostal retractions, (+) barrel chest, (+) I:E 1:4, (-) abdominal paradox, decreased tactile fremiti on the right (T8 down), dull on percussion on the right (T8 down), decreased vocal fremiti and breath sounds on the right (T8 down), (+) egophony at the right (T8 down); (+) rales on right lower lung field (+) wheezes on all lung fields

14 Physical Examination Adynamic precordium, JVP 3 cm at 30o, AB 6th LICS AAL, not diffused or sustained, (-) heaves, lifts, thrills, S1 > S2 at the apex, S2 > S1 at the base, (-) S3, murmurs Flat abdomen, normoactive bowel sounds, soft, (-) tenderness, (-) masses, tympanitic in all quadrants, Liver span 8cm MCL, liver and spleen not palpable, traube’s space not obliterated, (-) CVA tenderness, (-) hepatojugular reflux Pulses full and equal, ABI 1, (-) cyanosis (-) edema (-) clubbing

15 Physical Examination Conscious, coherent, oriented to 3 spheres ; GCS 15 EOMs full and equal, V1V2V3 intact; can frown, can raise eyebrows, can smile; gross hearing intact, uvula midline, can shrug shoulders against resistance, can turn head against resistance, tongue midline on protrusion No muscle atrophy/hypertrophy, no fasciculation, MMT 5/5 all extremities Can do APST and FTNT with ease DTRs +2 on all extremities No Sensory deficits No babinski, no signs of meningeal irritation

16 Salient Features 69 y/o, M Chronic cough with sputum Dyspnea
Subjective Objective 69 y/o, M Chronic cough with sputum Resolve but recur Dyspnea 2 pillow orthopnea Hypertensive (2005) uncontrolled Diabetic (2007) uncontrolled (+) FH for HPN, DM and PTB 168 pack years Alcoholic beverage drinker In respiratory distress Tracheal deviation to the Left Barrel chest Prolonged I:E ratio Decreased tactile fremiti, vocal fremiti, breath sounds and dullness on percussion on the Right, T8 down Rales on the RLL Wheezes on all lung fields Dot-blot hemorrhage OS

17 Assessment Obstructive lung disease, probably Chronic Obstructive Pulmonary Disease (COPD), in acute exacerbation probably secondary to Community acquired pneumonia (CAP), in patient, non ICU setting t/c pleural effusion, right Systemic arterial Hypertension (SAH) stage 2 Diabetes Mellitus, Type 2 t/c Diabetic Retinopathy


19 Chronic Obstructive Pulmonary Disease
a disease state characterized by the presence of airflow obstruction due to chronic bronchitis or emphysema. the airflow obstruction is generally progressive.

20 Differential Diagnosis of COPD

21 Pathology of COPD CENTRAL airways: Enlarged mucus secreting glands and an increase in the number of goblet cells are associated with mucus hypersecretion. PERIPHERAL airways: chronic inflammation leads to repeated cycles of injury and repair of the airway wall  structural remodeling of the airway wall, with increasing collagen content and scar tissue formation, that narrows the lumen and produces fixed airways obstruction.

22 Pathophysiology Pathological changes in the lungs lead to corresponding physiological changes characteristic of the disease, including: (in order over the course) mucus hypersecretion, ciliary dysfunction, airflow limitation, pulmonary hyperinflation, gas exchange abnormalities, pulmonary hypertension, and cor pulmonale. Mucus hypersecretion and ciliary dysfunction lead to chronic cough and sputum production. These symptoms can be present for many years before other symptoms or physiological abnormalities develop.

23 Clinical Features of COPD

24 Physical Examination of COPD
Early: slowed expiration and wheezing on forced expiration. Obstruction progresses: hyperinflation becomes evident, and the AP diameter of the chest increases. The diaphragm becomes limited in its motion. Breath sounds are decreased, expiration is prolonged, and heart sounds often become distant. Coarse crackles may be heard at the lung bases. Wheezes are frequently heard, especially on forced expiration. End-stage COPD: Tri-pod position, use of accessory respiratory muscles of the neck and shoulder girdle, expiration through pursed lips and paradoxical indrawing of the lower interspaces is often evident. Cyanosis may be present. An enlarged, tender liver indicates heart failure Neck vein distention, especially during expiration  due to increased intrathoracic pressure. Asterixis may be seen with severe hypercapnia.

25 GOLD Classification of Stable COPD

26 GOLD Classification of Stable COPD
Patient usually not aware of abnormal lung function

27 GOLD Classification of Stable COPD
Worsening of airflow limitation, progression of symptoms w/ SOB typically on exertion

28 GOLD Classification of Stable COPD
Further worsening of airflow limitation, increased SOB and frequent exacerbations that impact the QOL of the patient

29 Acute Exacerbation of COPD
Sustained worsening of the patient’s symptoms from the usual stable state that is beyond normal day to day variation Onset usually acute (1-3 days)

30 Symptoms of COPD Exacerbation

31 Community Acquired Pneumonia
Acute infection of pulmonary parenchyma Symptoms of acute infection Respiratory or general Maybe less prominent in the elderly Acute infiltrates on CXR Clinical findings such as localized rales No hospitalization within previous 14 days Excludes residents in long term care facilities

32 Etiologies of CAP

33 Typical vs Atypical Pneumonia

34 CURB 65

35 JNC 7

36 Diabetes Mellitus RBS > 200 + symptoms of diabetes FBS < 126
2 hr OGTT > 200

37 Diabetic Retinopathy Affects the circulatory system of the retina.
Earliest phase: non-proliferative / background diabetic retinopathy.  arteries in the retina become weakened and leak, forming small, dot- like hemorrhages.  These leaking vessels often lead to swelling or edema in the retina and decreased vision. Next stage: Proliferative diabetic retinopathy.  circulation problems oxygen-deprivation or ischemic  new, fragile, vessels develop as the circulatory system attempts to maintain adequate oxygen levels within the retina (neovascularization)   hemorrhage leak into the retina and vitreous, causing spots or floaters, along with decreased vision.  Later phases: continued abnormal vessel growth and scar tissue may cause serious problems such as retinal detachment and glaucoma

38 Diabetic Retinopathy

39 ABI index resting ankle-brachial index of less than 1 is abnormal. If the ABI is: Less than 0.95, significant narrowing of one or more blood vessels in the legs is indicated. Less than 0.8, pain in the foot, leg, or buttock may occur during exercise (intermittent claudication). Less than 0.4, symptoms may occur when at rest. 0.25 or below, severe limb-threatening PAD is probably present.

40 Thank you for listening 


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