Presentation is loading. Please wait.

Presentation is loading. Please wait.

Cardiac Arrhythmias A Guide For Medical Students William Beaumont Hospital Department of Emergency Medicine A Guide For Medical Students William Beaumont.

Similar presentations


Presentation on theme: "Cardiac Arrhythmias A Guide For Medical Students William Beaumont Hospital Department of Emergency Medicine A Guide For Medical Students William Beaumont."— Presentation transcript:

1 Cardiac Arrhythmias A Guide For Medical Students William Beaumont Hospital Department of Emergency Medicine A Guide For Medical Students William Beaumont Hospital Department of Emergency Medicine

2 In evaluating arrhythmias - Rate - Is it fast or is it slow If slow – is there group to group beating Rhythm - Is it regular, irregular or irregularly irregular? P waves - Are they present? QRS - Is it narrow or wide? Rate - Is it fast or is it slow If slow – is there group to group beating Rhythm - Is it regular, irregular or irregularly irregular? P waves - Are they present? QRS - Is it narrow or wide?

3 Sinus Bradycardia What is it? What causes it? When do you treat it? How do you treat it? What is it? What causes it? When do you treat it? How do you treat it?

4 Sinus Bradycardia A sinus rhythm with normal intervals and a rate less than 60 bpm Normal variant, beta blocker overdose, dig, hypothermia, hypothyroidism, brady-tachy syndrome, and SA node ischemia Requires treatment only if there is evidence of hypoperfusion Two treatment options Pacing: transvenous or transcutaneous Atropine 0.5 mg IVP A sinus rhythm with normal intervals and a rate less than 60 bpm Normal variant, beta blocker overdose, dig, hypothermia, hypothyroidism, brady-tachy syndrome, and SA node ischemia Requires treatment only if there is evidence of hypoperfusion Two treatment options Pacing: transvenous or transcutaneous Atropine 0.5 mg IVP

5 Sinus Tachycardia A sinus rhythm faster than 100 bpm Etiology - usually a physiologic response to a stressor Volume depletion / low stroke volume Hypoxia Systemic pathology: fever, anemia, hyperthyroidism Drugs Treatment - treat the underlying cause A sinus rhythm faster than 100 bpm Etiology - usually a physiologic response to a stressor Volume depletion / low stroke volume Hypoxia Systemic pathology: fever, anemia, hyperthyroidism Drugs Treatment - treat the underlying cause

6 Atrial Arrhythmias PACs MAT Atrial Fibrillation, atrial flutter SVT PACs MAT Atrial Fibrillation, atrial flutter SVT

7 Multifocal Atrial Tachycardia Diagnosis requires the presence of three distinct p waves in a narrow complex tachycardia Almost always associated with pulmonary disease Less often due to hypokalemia or hypomagnesemia Treat the underlying disorder – usually hypoxia Unlike the other atrial tachyarrhythmias, cardioversion is of no value in MAT Diagnosis requires the presence of three distinct p waves in a narrow complex tachycardia Almost always associated with pulmonary disease Less often due to hypokalemia or hypomagnesemia Treat the underlying disorder – usually hypoxia Unlike the other atrial tachyarrhythmias, cardioversion is of no value in MAT

8 MAT Rule of Threes 3 different p waves, 3 different pr intervals and 3 different r to r intervals

9 Causes of A-fib Cardiovascular - CAD, HTN, CHF, myopathy, myo-, endo- and pericarditis, infiltrative disease, valvular, congenital Metabolic - thyroid, electrolytes Pulmonary - pulmonary HTN, PE Toxic - cocaine, ETOH (holiday heart), beta agonists Sepsis Idiopathic Cardiovascular - CAD, HTN, CHF, myopathy, myo-, endo- and pericarditis, infiltrative disease, valvular, congenital Metabolic - thyroid, electrolytes Pulmonary - pulmonary HTN, PE Toxic - cocaine, ETOH (holiday heart), beta agonists Sepsis Idiopathic

10 ECG Rules for A-fib Regularity - irregularly irregular Rate - atrial rate usually > 350 Controlled - ventricular rate < 100 RVR - ventricular rate > 100 P wave - none discernable, may be f waves QRS - less that 0.12 seconds (easy dx). If > 0.12 sec must rule out VT (which is usually more regular) Regularity - irregularly irregular Rate - atrial rate usually > 350 Controlled - ventricular rate < 100 RVR - ventricular rate > 100 P wave - none discernable, may be f waves QRS - less that 0.12 seconds (easy dx). If > 0.12 sec must rule out VT (which is usually more regular)

11 A-fib with RVR A fib with ventricular rate > than bpm Patients usually symptomatic requiring rapid tx Unstable – cardioversion Stable - control rate with calcium channel blockers, beta blockers or digitalis A fib with ventricular rate > than bpm Patients usually symptomatic requiring rapid tx Unstable – cardioversion Stable - control rate with calcium channel blockers, beta blockers or digitalis

12 A-fib treatment Recognize the underlying cause A rate under 120 in an asymptomatic patient generally requires no emergent treatment Unstable patients with acute rapid a-fib should receive synchronized cardioversion with J Treatment otherwise depends on the duration Recognize the underlying cause A rate under 120 in an asymptomatic patient generally requires no emergent treatment Unstable patients with acute rapid a-fib should receive synchronized cardioversion with J Treatment otherwise depends on the duration

13 Treatment of A-fib Less than 48 hours duration Cardioversion is indicated in any unstable patient, synchronized if possible, with J May also be used electively in symptomatic but stable patients Pharmacologic cardioversion may be attempted with procainamide, amiodarone or ibutilide Less than 48 hours duration Cardioversion is indicated in any unstable patient, synchronized if possible, with J May also be used electively in symptomatic but stable patients Pharmacologic cardioversion may be attempted with procainamide, amiodarone or ibutilide

14 Treatment of A-fib > 48 hours Longer duration predisposes the patient to atrial clot formation and failure of conversion Rate control with diltiazem, beta blockers or digitalis Do not attempt cardioversion unless emergently indicated Anticoagulation and arrangement for echo Longer duration predisposes the patient to atrial clot formation and failure of conversion Rate control with diltiazem, beta blockers or digitalis Do not attempt cardioversion unless emergently indicated Anticoagulation and arrangement for echo

15 Atrial Flutter Patients usually with cardiac or pulmonary dz Conduction through the AV node may be at a 2:1, 3:1, 4:1 or 5:1 rate Whenever you see a ventricular rate close to 150 you should consider a flutter Frequently is a transient rhythm which may degenerate into a-fib or convert to sinus Patients usually with cardiac or pulmonary dz Conduction through the AV node may be at a 2:1, 3:1, 4:1 or 5:1 rate Whenever you see a ventricular rate close to 150 you should consider a flutter Frequently is a transient rhythm which may degenerate into a-fib or convert to sinus

16 Treatment of A-flutter Hemodynamically unstable - immediate synchronized cardioversion Hemodynamically stable Vagal manuevers – if no carotid bruits Adenosine - will not terminate the atrial tachycardia, but may allow flutter waves to become more apparent Dig, beta blockers or calcium channel blockers for AV nodal blockade to slow the ventricular rate Hemodynamically unstable - immediate synchronized cardioversion Hemodynamically stable Vagal manuevers – if no carotid bruits Adenosine - will not terminate the atrial tachycardia, but may allow flutter waves to become more apparent Dig, beta blockers or calcium channel blockers for AV nodal blockade to slow the ventricular rate

17 SVT AV nodal reentrant tachycardia Usually you see a regular, narrow complex tachycardia without p waves Treatment – adenosine, beta blockers, calcium channel blockers, digoxin AV nodal reentrant tachycardia Usually you see a regular, narrow complex tachycardia without p waves Treatment – adenosine, beta blockers, calcium channel blockers, digoxin

18 SVT – HR around 150s Is it SVT, afib, aflutter, sinus tach?

19 Preexcitation syndrome Wolf-Parkinson-White syndrome AV re-entrant tachycardia (accessory pathway) Short PR interval, delta waves Treat like PSVT if the QRS is narrow If the QRS is wide or if afib is present, use amiodarone or procainamide (slow the atrial rate and increase conduction through the AV node) Avoid ABCD – adenosine, beta blockers, calcium channel blockers, dig – if WIDE QRS Wolf-Parkinson-White syndrome AV re-entrant tachycardia (accessory pathway) Short PR interval, delta waves Treat like PSVT if the QRS is narrow If the QRS is wide or if afib is present, use amiodarone or procainamide (slow the atrial rate and increase conduction through the AV node) Avoid ABCD – adenosine, beta blockers, calcium channel blockers, dig – if WIDE QRS

20 Narrow complex WPW

21 Wide complex WPW

22 Atrioventricular Blocks First Degree Second Degree - Type I Second Degree - Type II Third Degree First Degree Second Degree - Type I Second Degree - Type II Third Degree

23 Second Degree AV Blocks Group to group beating Second degree blocks are partial blocks Two types Type I, Mobitz I or Wenckebach - transient Type II, Mobitz II or Classic - often degenerates into 3 rd degree heart block Group to group beating Second degree blocks are partial blocks Two types Type I, Mobitz I or Wenckebach - transient Type II, Mobitz II or Classic - often degenerates into 3 rd degree heart block

24 Second Degree Type I Decremental Conduction: Grouped beats with progressively longer PR intervals until an impulse is not conducted (a p without a QRS) Usually narrow QRS Generally requires no treatment – atropine, temporary pacing if symptomatic May be associated with inferior MI Decremental Conduction: Grouped beats with progressively longer PR intervals until an impulse is not conducted (a p without a QRS) Usually narrow QRS Generally requires no treatment – atropine, temporary pacing if symptomatic May be associated with inferior MI

25 Second Degree Type II Conduction fails suddenly, without a change in the PR interval This is not a benign rhythm, is chronic and often progresses to a complete heart block Is associated with anteroseptal MI May have wide QRS Conduction fails suddenly, without a change in the PR interval This is not a benign rhythm, is chronic and often progresses to a complete heart block Is associated with anteroseptal MI May have wide QRS

26 Second Degree Type II No pharmacologic treatment – atropine has no effect on the His-Purkinje system so not helpful and may worsen the conduction ratio Emergency treatment - transcutaneous or transvenous pacing No pharmacologic treatment – atropine has no effect on the His-Purkinje system so not helpful and may worsen the conduction ratio Emergency treatment - transcutaneous or transvenous pacing

27 Third Degree Block Complete block - there is total AV Dissociation None of the atrial impulses are conducted through to the ventricles (P and QRS are independent, P-P and R-R intervals constant An escape rhythm from a focus below the block will drive the ventricles If the escape rhythm originates in the AV junction, the ventricular rate will be in the range of and the QRS less than 0.12 If the escape is generated from the ventricles, the rate will be in the range of with a wide QRS Complete block - there is total AV Dissociation None of the atrial impulses are conducted through to the ventricles (P and QRS are independent, P-P and R-R intervals constant An escape rhythm from a focus below the block will drive the ventricles If the escape rhythm originates in the AV junction, the ventricular rate will be in the range of and the QRS less than 0.12 If the escape is generated from the ventricles, the rate will be in the range of with a wide QRS

28 Third Degree Block Although patients may be asymptomatic, transcutaneous or transvenous pacing is warranted Autonomic drugs such as atropine will have no effect on ventricular rate Type I antiarrhythmics should be avoided (they may suppress the escape rhythm) Although patients may be asymptomatic, transcutaneous or transvenous pacing is warranted Autonomic drugs such as atropine will have no effect on ventricular rate Type I antiarrhythmics should be avoided (they may suppress the escape rhythm)

29 Ventricular Arrhythmias PVCs V tach V fib PVCs V tach V fib

30 Premature Ventricular Contractions Generally benign, but may be a consequence of a pathology, esp if multifocal More concerning causes include hypoxia, ischemia, MI, toxins/drugs, acidosis or alkalosis, hypokalemia Generally benign, but may be a consequence of a pathology, esp if multifocal More concerning causes include hypoxia, ischemia, MI, toxins/drugs, acidosis or alkalosis, hypokalemia

31 Ventricular Tachycardia Results from a dysrhythmia originating at or below the bundle of His Has a wide QRS complex (>0.12 second) May be monomorphic or polymorphic Results from a dysrhythmia originating at or below the bundle of His Has a wide QRS complex (>0.12 second) May be monomorphic or polymorphic

32 Monomorphic V-tach Morphologically consistent QRS complexes Most common form of v-tach Seen primarily with cardiac ischemia Also seen in cardiomyopathy, valvular disease, electrolyte imbalance, myocarditis Morphologically consistent QRS complexes Most common form of v-tach Seen primarily with cardiac ischemia Also seen in cardiomyopathy, valvular disease, electrolyte imbalance, myocarditis

33 Polymorphic V-tach QRS complexes vary in structure and amplitude Predominantly caused by CAD Associated with more severe disease QRS complexes vary in structure and amplitude Predominantly caused by CAD Associated with more severe disease

34 Torsades de pointes A specific form of polymorphic v-tach Associated with prolonged QT May be due to drugs (tricyclics), electrolyte imbalance (hypo K, Mg or Ca), or subarachnoid hemorrhage A specific form of polymorphic v-tach Associated with prolonged QT May be due to drugs (tricyclics), electrolyte imbalance (hypo K, Mg or Ca), or subarachnoid hemorrhage

35 Treatment of Ventricular Tachycardias Unstable - immediate cardioversion 100 – 200 – 300 – 360 Stable - amiodorone 150 mg IVP or lidocaine 1 mg/kg and prepare for elective cardioversion If torsades de pointes – magnesium 1-2g IV Unstable - immediate cardioversion 100 – 200 – 300 – 360 Stable - amiodorone 150 mg IVP or lidocaine 1 mg/kg and prepare for elective cardioversion If torsades de pointes – magnesium 1-2g IV

36 Ventricular Fibrillation An irregularly irregular rhythm with no p waves or definite QRS complexes

37 Treatment of V Fib Defibrillate Adult 360/360/360 joules Children 2 joules/kg Epinephrine 1 mg IVP q 3-5 min (0.01 mg/kg) Amiodarone, Lidocaine, Magnesium Defibrillate Adult 360/360/360 joules Children 2 joules/kg Epinephrine 1 mg IVP q 3-5 min (0.01 mg/kg) Amiodarone, Lidocaine, Magnesium

38 Osborne Waves Not a true arrhythmia, but an EKG abnormality suggestive of underlying pathology Seen primarily in hypothermia, < 35.6 degrees May also be seen in other conditions, such as hypercalcemia or brain injury Also called J-waves, Camel backs, hathooks Not a true arrhythmia, but an EKG abnormality suggestive of underlying pathology Seen primarily in hypothermia, < 35.6 degrees May also be seen in other conditions, such as hypercalcemia or brain injury Also called J-waves, Camel backs, hathooks

39 Osborne Waves - Hypothermia

40 Osborne Waves - Hypercalcemia

41 Brugada Syndrome Genetic disease - autosomal dominant Mutation in the gene that controls the Na channel Characteristic ECG – ST segment elevation V1-V3 no signs of ischemia short QT interval Most common cause of sudden death in young males with no underlying cardiac disease Prevalence for Asians Cause of death – polymorphic V tach or V fib Treatment – no medicine will prevent AICD to abort lethal arhythmias

42 Diagnostic Criteria Type I is the only ECG criterion that is diagnostic of Brugada syndrome. (see figure). Definitive diagnosis - type 1 ST-segment is observed in >1 right precordial lead (V1 to V3) and one of the following: documented ventricular fibrillation (VF) polymorphic ventricular tachycardia (VT) a family history of sudden cardiac death at <45 years old coved-type ECGs in family members inducibility of VT with programmed electrical stimulation syncope nocturnal agonal respiration. Type I is the only ECG criterion that is diagnostic of Brugada syndrome. (see figure). Definitive diagnosis - type 1 ST-segment is observed in >1 right precordial lead (V1 to V3) and one of the following: documented ventricular fibrillation (VF) polymorphic ventricular tachycardia (VT) a family history of sudden cardiac death at <45 years old coved-type ECGs in family members inducibility of VT with programmed electrical stimulation syncope nocturnal agonal respiration.

43 Brugada Syndrome

44 Now for some cases…

45 82 yo male with hx HTN c/o weakness dizziness SOB BP 100/50 HR 155 What does the EKG show? What is the treatment?

46 35 yo female c/o palpitations, near syncope, occasional episodes of rapid HR What does the EKG show What medicines should be avoided in this patient?

47 65 yo male with COPD c/o SOB, wheezing and pedal edema What does the EKG show? What is the treatment?

48 91 yo female c/o weakness and syncope. What does the EKG show? What is the treatment?

49 26 yo medical student presents with N,V altered mental status. She has not been eating well. She is on Erythromycin for a URI. Her K=2.1 Mg=1.0 (nl= ) She collapses in the ER and is placed on a monitor. What does the monitor show? What is the treatment? What does the monitor show? What is the treatment?

50 48 y/o F presents lightheaded after walking. She just started metoprolol. What does the ecg show? What is the treatment?

51 42 y/o F presents with palpitations and DIB? What does the ecg show? How could you differentiate the rhythm?

52 You are working in the EC. The nurses come get you for this rhythm. It appeared 45 min after giving thrombolytics for AMI. Previously it was NSR. What is this? What should you do?

53 86 y/o M c/o weakness and DIB. What rhythm is this on the monitor? What should you worry about? Are you going to treat this? What rhythm is this on the monitor? What should you worry about? Are you going to treat this?

54 61 y/o M with CP, palpitations, and dizziness. What is the ecg show? What do you want to do?

55 So you cardioverted the previous rhythm and now this is on the monitor… What is this? What should you check? What should you do (besides soil yourself)? What is this? What should you check? What should you do (besides soil yourself)?

56 24 y/o pregnant F presents with 3 days of vomiting… What does the ecg show? What is the treatment of choice? What does the ecg show? What is the treatment of choice?

57 52 y/o M who is homeless and found sleeping in an alley. What does the ecg show? What is the treatment?

58 45 y/o M with CKD on HD presents with palpitations and DIB after missing dialysis. What does this ecg show? What should you do to treat this? What does this ecg show? What should you do to treat this?

59 Had enough yet? The End


Download ppt "Cardiac Arrhythmias A Guide For Medical Students William Beaumont Hospital Department of Emergency Medicine A Guide For Medical Students William Beaumont."

Similar presentations


Ads by Google