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Alteration in Respiratory Function Jan Bazner-Chandler RN, MSN, CNS, CPNP.

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Presentation on theme: "Alteration in Respiratory Function Jan Bazner-Chandler RN, MSN, CNS, CPNP."— Presentation transcript:

1 Alteration in Respiratory Function Jan Bazner-Chandler RN, MSN, CNS, CPNP

2 Allergic Rhinitis

3 Assessment Itching of nose, eyes, and throat Sneezing and stuffiness Watery nasal discharge / post nasal drip Watery eyes Swelling around the eyes

4 Assessment Allergic Shiner Allergic Salute

5 Rhinitis Interdisciplinary Interventions Avoid offending allergen – smoke / pets Pharmacologic management: Oral or nasal antihistamines - Benadryl Leukotriene modifiers - Singulair Mast cell stabilizers – cromylin – nasal / ophthalmic / inhaled Allergen-specific immunotherapy Do not use combination OTC medications especially those that contain pseudoephedrine No OTC Antihistamines for children under 6 years of age.

6 Sinusitis

7 Assessment Fever Purulent rhinorrhea Nasal congestion Pain in facial area Malodorous breath Chronic night-time cough Children more prone to sinusitis: children with asthma and cystic fibrosis.

8 Interdisciplinary Interventions Normal saline nose drops Warm pack to face Acetaminophen for pain Increase po fluid intake Antibiotics Recent studies question their effectiveness

9 Tonsillitis Tonsils and adenoids are important to the normal development of the bodys immune system. Serve as part of the bodys defense against infection Can become the site of acute or chronic infection Repeated acute infections cause the tonsil tissue to swell Enlarged tonsils and adenoids impinge on the pharyngeal opening of the eustachian tube

10 Assessment Child may refuse to drink Fever Reddened pharynx and tonsils Most common causative agent = group A beta-hemolytic stretococci Chronic tonsillitis may result in snoring due to enlarged tonsils and adenoids

11 Tonsilitis Kissing tonsils occur when the tonsils are so enlarged they touch each other.

12 Interdisciplinary Interventions Throat culture to determine causative agent Antibiotics for ten days if throat culturepositive for beta strep Acetaminophen for pain Cool fluids Saline gargles Antiseptic sprays Viral throat infections will not get better faster with antibiotics.

13 Tonsillectomy Done if childs respiratory status is compromised Post operative care: Side lying position Ice collar Watch for swallowing Cool fluids / soft diet

14 Croup Most common acute respiratory condition seen in early childhood. Highest incidence from 6 months to about 3 years Respiratory symptoms are caused by inflammation of the larynx and upper airway, with resultant narrowing of the airway. Severity depends on the area of the upper airway that is inflamed and narrowed. Most often viral – antibiotics are not needed


16 Assessment Symptoms: Hoarseness Inspiratory stridor Barking cough Afebrile Often worsens at night

17 Interdisciplinary Interventions Home care: Cool mist Fluids Hospital care: Racemic epinephrine inhalant Mist tent – not used much anymore Dexamethasone IV fluids if not taking po fluids

18 Epiglottitis Tripod position Bowden & Greenberg

19 Acute Epiglottitis Acute inflammation of supraglottic structures, the epiglottis and aryepiglottic folds. True pediatric emergency Delayed treatment may result in complete airway obstruction Most often seen in children 2 to 7 years Most common causative agent – H. influenzae type B

20 Assessment Sudden onset High fever – or greater Dysphasia and drooling Agitation, irritability and restlessness Epiglottis is cherry red and swollen Note: Do not look into the mouth – diagnosis often made by presenting symptoms or lateral neck x-ray

21 Interdisciplinary Interventions Keep child quiet in a controlled medical environment with emergency airway equipment readily available. Do not put tongue blade in mouth to look in the throat – may cause epiglottis to spasm and shut Assess respiratory status Give humidified oxygen by mask and keep HOB elevated. Mild sedation may help the child relax

22 Apnea Apnea is cessation of respirations lasting longer than 20 seconds. Monitor in hospital for underlying problems Discharge home with monitor

23 Foreign Body Severe inspiratory stridor Symptoms depend on location Unilateral chest movement Chest x-ray Bronchoscope to remove object

24 Coin in Trachea

25 Teaching No small hard candies, raisins, popcorn or nuts until age 3 or 4 years Cut food into small pieces No running, jumping, or talking with food in mouth Inspect toys for small parts Keep coins, earring, balloons out of reach

26 Influenza Associated with community epidemic Febrile, URI, achy joints Management: Acetaminophen for fever Fluids Keep away from others Watch for signs of pneumonia

27 Bronchiolitis Acute obstruction and inflammation of the bronchioles. Most common causative agent: Respiratory Syncytial Virus (RSV) Bronchioles become narrowed or occluded as a result of inflammatory process, edema, mucus and cellular debris clog alveoli


29 Assessment Harsh dry cough Low grade fever Feeding difficulties Wheezing Respiratory distress with apnea Thick mucus

30 Interdisciplinary Interventions Oxygen to maintain oxygen saturation >than 95% Pulse oximeter Nasal suction as needed Chest percussion to mobilize secretions Inhalation therapy – not sure if it is beneficial Mechanical ventilation as needed if increased work of breathing is seen Increased heart rate, poor peripheral perfusion, apnea, bradycardia and hypercarbia

31 RSV Positive - Isolation Respiratory Syncytial Virus is spread from respiratory secretions through close contact with infected persons or contact with contaminated surfaces or objects. Patient should be on contact and respiratory isolation Can be placed with other RSV + patients

32 Pneumonia An inflammatory condition of the lungs in which alveoli fill with fluid or blood resulting in poor oxygenation and air exchange. Can be primary illness or develop as a complication of another illness. Incidence: 34 to 40 cases per 1000 children younger than 5 years Most likely to develop when the body is unable to defend against infectious agents.


34 Typical X-ray

35 Assessment High fever Thick green, yellow, or blood tinged secretions Grunting respirations Rales, crackles, diminished breath sounds Cough and cyanosis Diagnostic tests: Infiltrate seen on x-ray

36 Interdisciplinary Interventions Assess for respiratory distress NPO (respiratory rate > 60 = high risk for aspiration) IV fluids for hydration Supplemental Oxygen to keep oxygen saturation equal to or > 92% Chest percussion Nasal suctioning as needed Acetaminophen for fever Antibiotics – ampicillin and an aminoglycoside (Gentamicin)

37 Pneumonia Isolation Respiratory isolation May be taken off isolation if RSV negative and on antibiotics for 24 hours.

38 Cystic Fibrosis Inherited autosomal recessive disorder of the exocrine glands Gene responsible for CF is located on chromosome 7 Life span is about 37 years Complex disease requiring a holistic approach

39 CFTR Gene Mutation of the CFTR gene disrupts the function of the chloride channels, preventing them from regulating the flow of chloride ions and water across cell membranes. As a result cells that line the passage ways of the lungs, pancreas and other organs produce mucus that is thick and sticky

40 Cystic Fibrosis


42 Assessment History of Meconium ileus at birth Foul smelling, greasy, bulky stools / constipation Voracious appetite with poor weight gain Recurrent respiratory infections Persistent chronic cough Salty tasting skin

43 Diagnosis Positive sweat test – Gold standard Genetic marker

44 Medications Pancreatic enzymes to help digest food Inhaled antibiotics – antimicrobial for lung treatment Aerosol bronchodilators to open airways Mucolytic enzyme – to thin mucus H2 blocker – alters gastrointestinal acidic environment Tagamet Prokinetic agents – enhances gastrointestinal motility Reglan Vitamin C to improve absorption of other meds Vitamins E, A, D, K / fat soluble vitamins Oral and IV antibiotics – S. aureus, H. influenzae, P aeruginosa

45 Long Term Complications Nasal polyps Sinusitis Rectal polyps / rectal prolapse Hyperglycemia / diabetes Infertility - male

46 Asthma Asthma is a chronic, inflammatory lung disease involving recurrent breathing problems. Third leading cause of hospitalization among children younger than 15 years. Most common, chronic health problem in children

47 Pathophysiology Reversible changes in airway that lead to bronchoconstriction, airway hyper-responsiveness and airway edema. At the cellular level mast cells release histamine causing smooth muscle contraction and bronchoconstriction. Increased mucous secretion by goblet cells causes epithelial damage Increased mucus secretion results in airway edema, mucus hypersecretion and plugging, airway narrowing, leading to airway obstruction

48 Assessment Wheezing Cough Tightness of chest Prolonged expiratory phase

49 Assessment Hypoxemia – universal in child with moderate to severe symptoms Hypercarbia – carbon dioxide retention from air trapping in the alveoli and ventilation – perfusion mismatch Monitor blood gases – PaCO2 level more than 50 mm Hg indicated ventilatory failure Diagnostics: chest x-ray = hyper-expansion of lungs

50 Asthma Attack

51 Interdisciplinary Interventions High fowlers position / bed rest Pulse oximetry Nebulized albuterol – beta 2 agonist Chest percussion to mobilize secretions Methylprednisone / Solu-medrol IV IV fluids Oxygen to keep oxygen sats > 95%

52 Home Management Peak flow spirometer Identify triggers Maximize lung function Optimal physical growth Optimal psycho-social state Maximum participation

53 Peak Flow Meter Peak flow meters are used to measure PEFR and are designed for monitoring purposes rather than diagnosis of asthma.

54 Home Medications Rescue drugs: short acting albuterol beta 2 agonist – used as a quick-relief agent for acute bronchospasm and for prevention of exercise induced bronchospasm. Anti-inflammatory or preventative: low-dose inhaled corticosteroid: inhaled or oral prednisone Allergy: leukotrines such as Singulair

55 Bronchodilators Bronchodilators rapidly relax the airway smooth muscle cells, thus reversing the bronchospasm until anti- inflammatory effect of steroids is attained. Aerosols Via mouth piece 3 years and older Via facial mask for less than 3 years

56 Spacer mdi pediatrics

57 Nebulizer pediatrics

58 Corticosteroids Steroids reduce the inflammatory component of bronchial obstruction, decrease mucus production and mediator release, as well as the late phase (cellular) inflammatory process. Methyl prednisone IV in severe cases May need histamine H2 receptor antagonists (cimetadine or ranitidine) if experiencing GI upset PO prednisone – always give with food to decrease GI upset

59 Inhaled Corticosteroids Inhaled corticosteroids: Pulmicort, AeroBid, Flovent Infant: mask should fit firmly to prevent cataracts Older child: rinse and spit after treatment to prevent thrush

60 Family Teaching Teach how to use medication When to use and how often No OTC drugs Increase fluid intake Signs and symptoms of respiratory distress

61 Neonate Disorders Pediatric Nursing January/February 1999 Bronchopulmonary Dysplasia

62 History It occurs in newborns who are born prematurely and or have a variety of pulmonary disorders and who require ventilatory support with high pressure and oxygen in the first 2 weeks of life.

63 Pathophysiology Fibrosis of airways and marked hyperplasia of the bronchial epithelium Increased fluid in the lungs, as a result of disruption of the alveolar-capillary membrane Over distention due to damage to alveolar supporting structures resulting in air trapping Fibrosis, airway edema, and broncho-constriction

64 BPD Assessment Persistent respiratory distress Dependent on supplemental oxygen Failure to thrive Gastro-esophageal reflux Pulmonary hypertension

65 Long-term Outcomes Oxygen dependent Visual problems Feeding difficulties Developmental delay Learning difficulties

66 Long Term Management Supplemental oxygen CPT Bronchodilators Diuretics (pulmonary hypertension) Anti-inflammatory medication Nutritional support: po formula + NG supplement Gastrostomy tube (GER) Bicarbonate in formula due to chronic state of acidosis

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