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Heterochromatin protein 1 is recruited to various types of DNA damage Martijn S. Luijsterburg, Christoffel Dinant, Hannes Lans, Jan Stap, Elzbieta Wiernasz, Saskia Lagerwerf, Daniël O. Warmerdam, Michael Lindh, Maartje C. Brink, Jurek W. Dobrucki, Jacob A. Aten, Maria I. Fousteri, Gert Jansen, Nico P. Dantuma, Wim Vermeulen, Leon H.F. Mullenders, Adriaan B. Houtsmuller, Pernette J. Verschure, and Roel van Driel J. Cell Biol. Volume 185(4): May 2, 2009 © 2009 Luijsterburg et.al.
Figure 1. Recruitment of HP1s to UV damage depends on the CSD Luijsterburg et al. J. Cell Biol. 2009:185: © 2009 Luijsterburg et.al.
Figure 2. Recruitment of HP1 in NER-deficient cells Luijsterburg et al. J. Cell Biol. 2009:185: © 2009 Luijsterburg et.al.
Figure 3. Long-term accumulation of HP1-β in repair-proficient and repair-deficient cells Luijsterburg et al. J. Cell Biol. 2009:185: © 2009 Luijsterburg et.al.
Figure 4. Recruitment of HP1-β to DSBs Luijsterburg et al. J. Cell Biol. 2009:185: © 2009 Luijsterburg et.al.
Figure 5. Survival of C. elegans HP1 knockout worms upon UV irradiation and IR Luijsterburg et al. J. Cell Biol. 2009:185: © 2009 Luijsterburg et.al.
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Jordan A. Mays. Key Points: 8 XP family proteins repair DNA damaged by sunlight through Nucleotide Excision Repair. XPF-ERCC1 complex is a structure-specific.
INTRODUCTION It is known that neurodegenerative disorders such as Alzheimer’s disease and Parkinson's disease are caused by protein accumulation within.
A lower risk of recurrent venous thrombosis in women compared with men is explained by sex-specific risk factors at time of first venous thrombosis in.
A Molecular Switch for Initiating Cell Differentiation in Arabidopsis Maite Sanmartín, Michael Sauer, Alfonso Muñoz, Jan Zouhar, Angel Ordóñez, Wilhelmina.
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