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Syndrome X Insulin Resistance Metabolic Syndrome Healthy Wednesday Club 20 th February, 2008 with Richard Hill & Sue Davis.

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Presentation on theme: "Syndrome X Insulin Resistance Metabolic Syndrome Healthy Wednesday Club 20 th February, 2008 with Richard Hill & Sue Davis."— Presentation transcript:

1 Syndrome X Insulin Resistance Metabolic Syndrome Healthy Wednesday Club 20 th February, 2008 with Richard Hill & Sue Davis

2 Uh-oh

3 Bad, Scary News Sugar is an addiction like tobacco and alcohol Obesity is now the main association with poor health (overtaking tobacco) Teenagers have insulin resistance Half of children born this century will have diabetes II by the age of 30

4 Insulin (from pancreas) helps glucose (broken down from carbohydrates) to enter the cell to give the cell energy Insulin does this by attaching to a receptor on the outer wall of the cell Insulin converts the excess Glucose into a starch – glycogen for storage in the muscles and liver for later use. Normal

5 Insulin Resistance Too much glucose, especially from refined carbohydrates causes the pancreas to produce lots of insulin the cells are overwhelmed and shut down receptors Insulin cant get the glucose out of the blood and into cells so it turns the glucose into fat instead (triglycerides)

6

7 Visceral Fat

8

9 Professor Gerald Raven

10 NutritionInactivityStressSmokingSleep DISEASE Gastro-Int Osteopathic ReproductiveSensory Metabolic Dermatological. Psychological Pulmonary Cancers O B E S I T Y Inflammatory cytokines, adipokines, myokines etc. Low Grade Systemic Inflammation Insulin Resistance Risk Factors Lifestyle-Based Causes of Disease

11 More glucose from too much refined carbohydrate, too often Pancreas produces more insulin to cope High glucose AND high insulin pre-diabetic syndrome Pancreas finally gives up Diabetes II – late onset diabetes

12 X. CENTRAL OBESITY: Defined according to ethnicity: >94 for Europid men >80 for Europid women(10cm less for Asians/Indians etc) XX. PLUS ANY TWO OF THE FOLLOWING: A.Raised TG: (>1.7mmol/L) B.Decreased HDL: (M<1.0 mmol/L; F<1.3 mmol/L) C.Raised Blood Pressure: Systolic BP >130mmHG Diastolic BP >85mmHG D. Raised Fasting Plasma Glucose: >5.6 mmol/L IDF Criteria for Defining Metabolic Syndrome

13 Fatigue Agitated, jittery, moody, headaches Intestinal bloating – gas Sleepiness Weight gain Depression Also –

14 What to do STOP THIS

15 DO THIS EXERCISE

16

17 And do this Healthy, low GI, whole foods

18 Glucaemic Index GI is the effect that food has to raise blood glucose over 2 hours

19 GI 3 tbsp glucose = 50 grams pure carbohydrate = 200 gms of spagetti = 5 oranges Carbohydrates are how many calories food contains 1gm = 4 calories 1 tbsp honey = 17g carb (= 68 cals) - GI 58 ½ cup Lima Beans = 17g carb (= 68 cals) - GI 32

20 Low GI – less than 55 Moderate GI – 56 to 70 High GI – greater than 70 On Packaging

21 Vegetables - GI High GI – potato, carrots, pumpkin, corn, sweet potato High Carbs, Low GI – beetroot, peas Normal servings of carrots, pumpkin and potatoes eaten with protein will lower GI effect

22 Bread – GI (per slice) Low GI – Chapati (Indian bread made from chickpea flour) GI – 27 Pumpernickel GI – 52 Sourdough GI – 52 Stone ground GI – 53 Standard white GI - 70

23 Fruits GI Low: Grapefruit – 25 Cherries – 22 Apples - 38 Pears - 38 Dried apricots - 31 Med - High: Oranges - 44 Grapes - 46 Mango - 55 Apricots - 57 Watermelon - 72

24 Gatorade (1 cup) - 78 Approx 3 cups per bottle

25 AND DO THIS Good quality Supplements Especially antioxidants

26 A meal replacement program That encourages you eat

27 Medical Journal of Australia Volume 84 No 2 January 16, 2006 Professor Gary Eggar - SCU Are meal replacements and effective clinical tool for weight loss? Clinical trials show partial meal replacement products to be safe, acceptable and effective when used as part of an overall low energy diet.

28 Side effects of weight loss Homocystine levels Oxidation – free radicals Energy loss Depression Medications – weight gain Starvation

29 AND THIS Romance, Beauty and Joy mind – body – soul connection

30

31 NutritionInactivityStressSmokingSleep DISEASE Gastro-Int Osteopathic ReproductiveSensory Metabolic Dermatological. Psychological Pulmonary Cancers O B E S I T Y Inflammatory cytokines, adipokines, myokines etc. Low Grade Systemic Inflammation Insulin Resistance Risk Factors Lifestyle-Based Causes of Disease

32 Biological Pathology Ecological Pathology Risk factors/ Markers Proximal Causes Medial Causes Distal Causes Dis-ease Industrialization Population growth Economic Growth -Diabetes -CVD -Cancers -Climate change -Sea level rise -Extinctions Energy use Over-production Industrial waste (Lifestyle) Over-nutrition Inactivity etc. Atmospheric pollution Ocean acidity etc. Ecoflammation Carbon resistance Global warming etc. Metaflammation Insulin resistance Obesity etc. Energy use Social pressure Peer pressure (Lifestyle) Individual Global Metaflammation, Ecoflammmation, health & the environment

33 A Lifestyle Screening Questionnaire

34 X. CENTRAL OBESITY: Defined according to ethnicity: >94 for Europid men >80 for Europid women(10cm less for Asians/Indians etc) XX. PLUS ANY TWO OF THE FOLLOWING: A.Raised TG: (>1.7mmol/L) B.Decreased HDL: (M<1.0 mmol/L; F<1.3 mmol/L) C.Raised Blood Pressure: Systolic BP >130mmHG Diastolic BP >85mmHG D. Raised Fasting Plasma Glucose: >5.6 mmol/L IDF Criteria for Defining Metabolic Syndrome

35 Indicators of Insulin Resistance


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