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Figure 1. Brain MRI follow-up of Sjögren syndrome–associated type II mixed cryoglobulinemic cerebral vasculitis treated with rituximab Brain MRI follow-up.

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Presentation on theme: "Figure 1. Brain MRI follow-up of Sjögren syndrome–associated type II mixed cryoglobulinemic cerebral vasculitis treated with rituximab Brain MRI follow-up."— Presentation transcript:

1 Figure 1. Brain MRI follow-up of Sjögren syndrome–associated type II mixed cryoglobulinemic cerebral vasculitis treated with rituximab Brain MRI follow-up of Sjögren syndrome–associated type II mixed cryoglobulinemic cerebral vasculitis treated with rituximab In case 1, axial T2-weighted image (A) demonstrates multifocal subcortical/deep white matter hyperintensities and residual parenchymal hematoma in the right frontal lobe, whereas MR angiography (B) shows segmental areas of arterial narrowing (arrows) of the cortical branches of the left middle cerebral artery. Three months after rituximab infusion, MR angiography (C) shows normalization of arterial calibre of the cortical branches of the left middle cerebral artery (arrows). In case 2, MR angiography (D) shows multifocal stenosis of the pericallosal artery (arrows). Three years after rituximab infusion, MR angiography (E) shows persistent stenosis (white arrows) and a mild dilatation of the previous narrow arterial segment (black arrow). MR = magnetic resonance. Bertrand Lioger et al. Neurol Neuroimmunol Neuroinflamm 2016;3:e253 © 2016 American Academy of Neurology

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Date of download: 7/2/2016 Copyright © 2016 American Medical Association. All rights reserved. From: Stroke in a Healthy 46-Year-Old Man JAMA. 2001;285(21):2757-2762.

Date of download: 7/2/2016 Copyright © 2016 American Medical Association. All rights reserved. From: Stroke in a Healthy 46-Year-Old Man JAMA. 2001;285(21):
Copyright © 2015 by the American Osteopathic Association.

Copyright © 2015 by the American Osteopathic Association.
Figure 1 Initial brain imaging (A–C) patient 1; (D–F) patient 2; (G–I) patient 3; (J–L) patient 4; and (M) patient 2. Initial brain imaging (A–C) patient.

Figure 1 Initial brain imaging (A–C) patient 1; (D–F) patient 2; (G–I) patient 3; (J–L) patient 4; and (M) patient 2. Initial brain imaging (A–C) patient.
Nat. Rev. Neurol. doi: /nrneurol

Nat. Rev. Neurol. doi: /nrneurol
Case One serial imaging.

Case One serial imaging.
Figure 3 Brain MRI findings in patients with MOG-Ab Extensive brain lesions with large diameter (A and B), posterior reversible encephalopathy–like lesions.

Figure 3 Brain MRI findings in patients with MOG-Ab Extensive brain lesions with large diameter (A and B), posterior reversible encephalopathy–like lesions.
Figure 1 Brain MRI findings in the present case

Figure 1 Brain MRI findings in the present case
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Figure 2 Evolution of MRI abnormalities in faciobrachial dystonic seizures Axial fluid- attenuated inversion recovery (FLAIR)-weighted images from patient.
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Figure 2 Association of serum IgG reactivity with MRI measures of disease severity Association of serum IgG reactivity with MRI measures of disease severity.
Figure 2 Spinal cord lesions

Figure 2 Spinal cord lesions
Figure 1 Coronal MRI images showing the evolution of white matter abnormality and atrophy of patient 1 Coronal MRI images showing the evolution of white.

Figure 1 Coronal MRI images showing the evolution of white matter abnormality and atrophy of patient 1 Coronal MRI images showing the evolution of white.
Figure Facial photograph during headache attack and brain and upper cervical cord MRI Facial photograph during headache attack and brain and upper cervical.

Figure Facial photograph during headache attack and brain and upper cervical cord MRI Facial photograph during headache attack and brain and upper cervical.
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Figure Brain MRI of the patient throughout the disease course(A) Brain MRI at the time of cerebral toxoplasmosis diagnosis (a) and after 1 month of toxoplasmosis.
Axial magnetic resonance (MR) imaging

Axial magnetic resonance (MR) imaging
Figure 3 Example of venous narrowing

Figure 3 Example of venous narrowing
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Figure Immune checkpoint inhibitor–induced encephalitis before and after treatment with natalizumab Immune checkpoint inhibitor–induced encephalitis before.
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Figure 1 MRI head in faciobrachial dystonic seizures (A) Axial fluid-attenuated inversion recovery image from patient 3 in table 2 shows T2-weighted hyperintensity.
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Figure Radiographic and histopathologic findings (A) Brain MRI at presentation shows multiple areas of T2 hyperintensity in the mesial temporal lobes,
Figure 1. Prebiopsy and postbiopsy MRI

Figure 1. Prebiopsy and postbiopsy MRI
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