1. The natural history of endothelial structure and function in arterialized vein grafts 
Harry L. Bush, M.D., Joseph A. Jakubowski, Ph.D., G.Richard Curl, M.D., Daniel Deykin, M.D., Donald C. Nabseth, M.D. 
Journal of Vascular Surgery 
Volume 3, Issue 2, Pages (February 1986)
DOI: / (86)
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions

2. Fig. 1
Luminal production of prostacyclin by autogenous vein grafts (groups I, II, and III) before and after arterialization. Results expressed as mean ± SEM; n = 10 grafts for each data point.
Journal of Vascular Surgery 1986 3, DOI: ( / (86) )
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions

3. Fig. 2
Luminal production of thromboxane A2 by autogenous vein grafts (groups I, II, and III) before and after arterialization. Results expressed as mean ± SEM; n = 10 grafts for each data point.
Journal of Vascular Surgery 1986 3, DOI: ( / (86) )
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions

4. Fig. 3
Prearterialization luminal morphology. A, Group 1 shows normal endothelial morphology and tight intercellular junctions (original magnification, × 2000). B, Group II shows similar luminal morphology (original magnification, × 2000). C, Group III shows extensive loss of endothelium with exposure of subendothelial structures (original magnification, × 1000).
Journal of Vascular Surgery 1986 3, DOI: ( / (86) )
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions

5. Fig. 4
Relationship of intraluminal perfusion pressure and prostacyclin release by lumen of healthy canine external jugular veins. Results expressed as mean ± SEM; n = 10 vein segments for each data point.
Journal of Vascular Surgery 1986 3, DOI: ( / (86) )
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions

6. Fig. 5
Arterialized in situ vein grafts (group I). A, Twenty-four hours after surgery: moderate areas of endothelial loss and separation plus extensive leukocyte adherence to denuded areas and migration under intact endothelial cells; platelets were rarely noted (original magnification, × 1000). B, One week: improved endothelial coverage with leukocytes and fibrin bridging areas of endothelial loss (original magnification, × 1500). C, Four weeks: persistence of leukocytes in areas of endothelial injury and continued absence of platelets (original magnification, × 2000). D, Six weeks: normal endothelial monolayer (original magnification, × 1000).
Journal of Vascular Surgery 1986 3, DOI: ( / (86) )
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions

7. Fig. 6
Arterialized reversed vein grafts (group II, normothermic whole blood). A, Twenty-four hours: extensive endothelial loss and separation plus dense platelet and leukocyte adherence (original magnification, × 1000). B, One week: area of healing endothelial surface with primarily leukocyte adherence on surface (original magnification, × 1000). C, Four weeks: persistent areas of endothelial cell loss with adherent platelets (original magnification, × 1000). D, Six weeks: healed endothelial monolayer predominates (original magnification, × 1000).
Journal of Vascular Surgery 1986 3, DOI: ( / (86) )
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions

8. Fig. 7
Arterialized reversed vein grafts (group III, hypothermic normal saline solution). A, Twenty-four hours: extensive cellular adherence on and under residual endothelium (original magnification, × 1000). B, One week: fibrinous, cellular coating of areas with extensive endothelial cell loss (original magnification, × 1000). C, Three weeks: example of activated leukocytes and platelets bridging gaps between healing endothelial cells (original magnification, × 2000.) D, Four weeks: area where endothelial cells have covered the gross defects; however, there is still significant separation of the endothelial cell junctions (original magnification, × 1000).
Journal of Vascular Surgery 1986 3, DOI: ( / (86) )
Copyright © 1986 Society for Vascular Surgery and North American Chapter, International Society for Cardiovascular Surgery Terms and Conditions