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NOISE-INDUCED HEARING LOSS Dr. Supreet Singh Nayyar, AFMC For more presentations, visit 14-Jul-12www.nayyarENT.com1.

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Presentation on theme: "NOISE-INDUCED HEARING LOSS Dr. Supreet Singh Nayyar, AFMC For more presentations, visit 14-Jul-12www.nayyarENT.com1."— Presentation transcript:

1 NOISE-INDUCED HEARING LOSS Dr. Supreet Singh Nayyar, AFMC For more presentations, visit 14-Jul-12www.nayyarENT.com1

2 LAYOUT Historical perspective Noise & effects of noise on hearing Mechanism & clinical features of NIHL Early detection and prevention of NIHL Noise and the armed forces Recent advances Legal and social issues in relation to NIHL 2

3 Historical perspective Bronze age 1713, Ramazzini found hearing loss in coppersmiths who hammered copper for their living. 1886 the first epidemiological survey of NIHL was conducted by Thomas Barr 1890 Habermann described the histology of NIHL in organ of corti. 1928 Fowler observed the typical dip at 4 KHz due to NIHL 1939 Bunch published the first audiometric feature of NIHL demonstrating the typical high frequency SNHL 3

4 Noise Noise is unwanted sound which may damage A persons hearing Temporal pattern of environmental noise can be continuous (steady state), fluctuating, impulsive or intermittent 4

5 Assessing Noise Levels Duration, frequency and sound pressure levels Sound Level Meter (SLM) Noise Dose Meter (NDM) The meter is designed to measure a frequency weighted and time-weighted value of the sound pressure level 5

6 General Aim Of A Noise Assessment •Identify •Obtain information •Effectiveness of measures taken •Choose appropriate personal hearing protectors •Define hearing protection areas at work 6

7 Excessive Noise A heavy truck is about 85 db, A jet taking off is about 120 db. Suction and drill during ear surgery 91-108 dB. MRI units 90 dB. Peak sound level in entertainment clubs 140dB 7

8 Effects Of Noise On Hearing Either reversible or permanent inner ear damage Auditory or non-auditory. Effects can happen with both high and low level noise Tinnitus Loss of hearing as well as health effects 8

9 Auditory Effects Of Noise Auditory adaptation Noise induced temporary threshold shift (NITTS) Noise induced Permanent threshold shift (NIPTS) Acoustic trauma 9

10 Adaptation An immediate physiological phenomenon that occurs whenever the ear is stimulated by sound. For sounds up to 70 db spl, recovery occurs in less than half a second. 10

11 Acoustic trauma A condition when there is a sudden damage to the ear due to intense short term exposure or even a single exposure to a very high intensity noise Usually occurs from fireworks, small arms fire, gun fire and explosion. This not only results in some damage to inner ear but also can damage the tympanic membrane and ear ossicles 11

12 Noise Induced Temporary Threshold Shift (NITTS) Exposure to moderately intense sounds A short term elevation of hearing thresholds Associated with other auditory symptoms Measured in minutes and days The amount of TTS is directly proportional to the intensity of sound and duration of exposure 12

13 Noise Induced Permanent Threshold Shift (NIPTS) Permanent elevation of hearing threshold due to chronic exposure to moderately intense noise Permanent structural damage to the critical elements of the cochlea 13

14 Factors Affecting The Development Of NIPTS Physical factors- intensity, duration of noise. Biological factors – individual susceptibility, age, sex, genetic and social predisposition, acoustic reflex, pre exposure threshold Pathological factors –drugs and chemicals, other co existing ear diseases. 14

15 Intensity of exposure Most studies have found exponential increase in hearing losses with increasing intensities Damage risk criteria regarding safe level duration of exposure have been established 15

16 Duration Of Exposure An average work place exposure of 90-94 dB everyday over a period 10 years, NIHL reaches its maximum and remains constant thereafter. Hearing loss in the higher frequencies will stop progressing but it will spread gradually to lower frequencies. Hearing loss occurs early in the first 2-3 years 16

17 Individual susceptibility Certain biologic characters unique to the individual Potentially important variables have been investigated No evidence to suggest that there is any relationship between age and susceptibility to NIHL 17

18 Role of Acoustic Reflex Variability in fatiguability of acoustic reflex may be one of the factors in individual susceptibility to noise trauma Documented that subjects with poor acoustic reflex recorded a large TTS after exposure to noise 18

19 Effect Of Melanin Melanin in stria vascularis of cochlea may have a protective effect against noise People with blue and light colored iris may be more susceptible to NIHL 19

20 Drugs And Chemicals Combination of noise and aminoglycosides Additional hearing loss may take place when humans are treated with aspirin and other NSAIDs and exposed to high intensity noise concomitantly. Environmental pollutants Chemical pollutants or chemical intermediaries in industries like toluene, hexa methyl mercury and lead acetate 20

21 Sociacusis Urban setting transportation is the main cause A worker who is exposed to sound levels of 88 dB for 8 hours, who is then exposed to 94 dBA sounds while commuting to and from work is at risk of developing NIHL due to additive effects of sound Recreational noise, personal stereos and CD players can also be potentially harmful 21

22 Medical noise MRI units may produce sound levels at patients head in excess of 90 dB Sound levels produced by drills and suction units during ear surgery are high enough to produce NIHL, ranging from 91- 108 dB Amplified noise in powerful hearing aids 22

23 Mechanisms for NIHL - theories : Mechanical/structural damage, ranging from disturbance of the delicate stereocilia to tearing of the organ of corti and eventually permanent hair- cell loss, caused by severe motion of basilar membrane due to excessive noise 23

24 Mechanisms for NIHL - theories : Exposure to extremely intense noise (130dB) results in necrotic-cell death process Metabolic exhaustion (overstimulation) of hair cells may lead to excessive release of glutamate, which may contribute to NIHL by causing swelling of auditory nerve terminals 24

25 Mechanisms for NIHL - theories : Severe vascular narrowing and ischemia of cochlear microvasculature due to excessive noise exposure Ionic imbalance and cellular damage due to disruption of ionic gradients of cochlear structures 25

26 Specific features of NIHL Permanent SNHL with damage predominantly to outer hair cells (OHCs). History of long term exposure to dangerous noise levels (> 85 dB for 8 hours per day) Gradual loss of hearing over 5-10 years of exposure Hearing loss initially involving higher frequencies 3-8 kHz before involving frequencies below 2KHz and below Speech recognition scores that are consistent with audiometric pattern Hearing stabilizes once the noise exposure is terminated 26

27 Audiometric configuration- acoustic notch 27

28 Otoacoustic emission (OAE) in NIHL The fact that OAEs are primarily generated by OHCs assumes great significance as noise primarily destroys the OHCs of cochlea in NIHL Therefore this investigative modality offers exciting prospects for early diagnosis of NIHL and possible detection of susceptible individuals 28

29 Diagnosis Diagnosis, which is circumstantial, is largely based on a careful history, physical examination and appropriate audiometric evaluation, and is frequently made by exclusion Noise exposure has been adequate, and there is an appropriate hearing loss, it is customary to attribute the loss to that cause 29

30 Damage Risk Factors Intensity of sound and duration of exposure. Equal amounts of sound exposure produce equal amounts of damage whether sound exposure is spread over a short or long period. Important to know that with a 90 dBA sound exposure for 8 hours a day, 5 days a week, 15% of the population is at risk for signicant hearing loss after 10 years of exposure, and that for 85 dBA exposure 8 hours a day, 5 days a week, after 10 years, only 7% of the population is at risk 30

31 Noise and Armed forces Personnel of the armed forces are exposed to very high intensity noise produced as a result of the weapon that they use, the mechanical transport, aircraft and ships Nature of their occupation exposes them to noise levels that can jeopardize their hearing Exposure to a combination of steady state noise and impulse noise of very high intensities and their unprotected ears are vulnerable to extensive hearing damage 31

32 Noise and Armed forces In armed forces, personnel serving in certain branches and trades are more vulnerable. In the army the infantry, artillery, armoured corps and engineers are at higher risk of developing NIHL In the air force the pilots, air handlers and air craft maintenance personnel are at higher risk. In the navy, engine room sailors, gunnery crew, air craft carrier personnel and divers and submariners are at higher risk of developing NIHL due to nature of their jobs 32

33 Noise in shooting ranges Nature of firing a firearm: produces very high peak sound pressure levels even with small calibers These peak levels cause impulsive sound to reach the inner ear and because of the characteristics of impulsive type sounds, our ears are more prone to hearing damage than with sounds emitted from operating machines 33

34 Typical noise levels from firearms Outdoor range pistol cal. 22 @ 10 m to the side 127 - 129dB peak(C) rifle cal 7.62 mm @ 2 m to the side 154 – 158dB peak(C) rifle cal 5.56 mm @ ear level 156 - 157dB peak(C) revolver cal.38 @ ear level 149 – 153dB peak(C) Indoor range pistol cal.38 @ approx. 1.5 m behind 145 – 147dB peak(C) pistol 9 mm @ approx. 1.5 m behind 147 - 149dB peak(C) 12 gauge pump action shotgun 150 – 151dB peak(C) 34

35 Treatment NIHL unfortunately cannot be cured but it probably is the single largest cause of preventable deafness all over the world Experimental treatment modalities like antioxidant therapy need further evaluation Rehabilitation with hearing aids is an excellent option in improving the communication status of people suffering from NIHL Advanced digital and programmable hearing aids offer very good quality of hearing improvement 35

36 Prevention Noise hazard identification Engineering controls Personal hearing protection Monitoring audiology Record keeping Health education Enforcement Programme evaluation 36

37 Dealing With Noise Hazards Carry out a risk assessment to determine the risks from noise exposure •Develop a noise control policy and a hearing conservation program •Provide workers with information and training on noise, the risks from noise exposure Control measures put in place at the workplace 37

38 Personal Hearing Protectors A device, or pair of devices, designed to be worn over (ear muffs) or inserted in the ears (earplugs) of a person to protect hearing Vary considerably in effectiveness and is highly frequency dependent Earplugs reduces the noise reaching the middle ear by 15 to 30 db and work best for the mid to higher frequency region (i.e. 2- 5 kHz) Earmuffs attenuate the noise by 30 to 40 db. 38

39 Personal Hearing Protectors Personal hearing protectors are most vital for prevention of NIHL The most important aspect of personal hearing protectors is the regularity of use Most important aspect of choosing a hearing protector device is worker comfort and confidence of the worker using it A large variety of personal hearing protectors like ear plugs, ear muffs and canal caps are available with varying degrees of attenuation 39

40 WHO Recommendation National programme for prevention of noise-induced hearing loss should be established in all countries and integrated with primary health care. This should include environmental and medical surveillance, noise reduction, effective legislation, inspection, enforcement, health promotion and education, hearing conservation, compensation and training Prevention of NIHL must be appropriate, adequate, acceptable and affordable Most of the population of developing countries is ignorant of the hazards of excessive noise exposure. Awareness must be increased about the harmful effects of noise and about its prevention and control of NIHL 40

41 WHO Recommendation There is acute shortage of reliable epidemiological data on prevalence, risk factors and costs of NIHL from developing countries. There is an urgent need of structured and controlled studies in this regard. Research needs to be focused on pathophysiology, technical measures for noise reduction, improving personal hearing protectors and low cost medications for prevention Communication and collaboration should be strengthened between developed and developing countries to facilitate research and development in this field 41

42 Legislations: Noise control policy An important step in managing noise in the workplace is the development of a noise control policy which should cover the following issues: – •Goals for noise exposure and peak noise levels – Design goals for new workplaces and plant – •Selection and purchase of quiet plant – •Noise controls for temporary work areas and situations – •Agreements with contractors for the responsibility of noise control and provision of information – •Audiometric testing and availability of records – •Funding for a hearing conservation program – •Period of review for the hearing conservation program 42

43 Permitted hours of exposure at work place Hrs permitted LeqLOSHA 16 8 4 2 1 0.5 0.25 87 dB 90 db 93 dB 96 dB 99 dB 102 dB 105 dB 85 dB 90 dB 95 dB 100 dB 105 dB 110 dB 115 dB 43

44 Ambient Noise standards and Guidelines in India Area codeCategory of areaLimits in dB(A) Leq Day Night ABCDABCD Industrial area Commercial area Residential area Silence Zone 75 70 65 55 55 45 50 40 Ambient noise standards published in Gazette of India no 643 dt 26.12.89, Succeeded by the Noise pollution (Regulation and control ) rules, 2000(Gazette of India, vide SOI 123(E) dated 14-12-2000 and subsequently amended vide SO 1046 (E) dated 22-11-2000. 44

45 Noise standards for Domestic appliances and construction equipments in India CategoryNoise limit dB(A) Window air-conditioners 1 ton & above Air coolers Refrigerators Diesel generators foe domestic purposes Compact rollers, front loaders, concrete mixers, cranes, vibrators and saws 68 60 46 85-90 75 Noise standards for Domestic appliances and construction equipment as notified under Environment (Protection) Rules, 1986, by G.S.R 742 (E) dated 30-8-90 and subsequently amended by GSR 422 (E) dated 19 May 1993 45

46 Noise limits for vehicles in India Type of vehicleNoiseLimits dB(A) Two wheeler displacement up to 80 cm3 Displacement more than 80 cm3 but less than 175 cm3 Displacement more than 175 cm3 Three wheeler Displacement up to 175 cm3 Displacement more than 175 cm3 Passenger car Passenger or commercial vehicle Gross weight up to 4 tonne Gross vehicle weight more than 4 tonne but up to 12 tonne Gross vehicle weight more than 12 tonne 75 77 80 77 80 75 77 80 82 Noise limits for vehicles from Jan 2003 as notified under Environment (Protection) Rules, 1996, by GSR 742(E) dated 25 Sep 2000 46

47 Research on NIHL Hair cell regeneration Genetic and molecular basis for NIHL Protection from conditioning the cochlear-efferent system Antioxidant therapy for NIHL and acoustic trauma 47

48 Hair cell regeneration Recent results using in vitro cultures of neonatal mouse cochleas showed that over expression of mammalian atonal homolog 1 or Math 1, a basic helix-loop-helix transcription factor known to be necessary for hair cell differentiation during development, leads to an increase in the production of extra numerary hair cells. It appears that certain cells in the mammalian organ of Corti can, at least in young animals, be redirected towards a hair cell fate by the over expression of Math 1 48

49 Hair cell regeneration Recent studies discovered that new hair cells can be grown in a mature mammalian ear using the Math 1 gene. Math 1 gene can direct hair cell differentiation in mature nonsensory cells and that adenoviral gene therapy will some day likely lead to a treatment of NIHL. 49

50 Genetic and molecular basis for NIHL Effort being made to find out if there are genetic factor in the susceptibility to NIHL It has been seen that some strains of inbred mice are more susceptible to NIHL than others Efforts to isolate a NIHL gene to a chromosomal loci A recessive gene that is responsible for premature age related hearing loss has been shown to be related to excessive susceptibility to NIHL 50

51 Protection from conditioning exposure experience pre exposure of low to moderate intensity sounds toughen or condition the ear and offer protection against subsequent harmful effects of high intensity noise Recent studies have demonstrated conditioning effect or the development of resistance in human subjects 51

52 Antioxidant therapy for NIHL N-L-acetylcysteine Acetyl-L-carnitine Carbanathione Glutathione ( GSH ) Allopurinol GSH peroxidase 52

53 Non-auditory Effects Of Noise Interference with communication Interference with efficiency and work output Psychological effects like irritability and annoyance Disturbance to sleep, rest and thereby contributing to fatigue Hypertension, peptic ulcer etc and other systemic illnesses 53

54 Role of the Otolaryngologist To identify the cause and extent of NIHL Educate the patient Preventive measures Aural rehabilitation Counseling the patient Teach the patient to guard against further loss Expert witness 54

55 Conclusion NIHL is the single most important cause for preventable hearing loss in this world today. It is to be appreciated that it is practically impossible to reduce noise levels in industry and in our cities to safe enough levels for infinite exposure. Educating people about adverse effects of noise and its prevention and the use of personal hearing protective devices are the major strategies against NIHL There is an urgent need to hasten research on the fundamental mechanisms involved in NIHL so that preventive and curative measures to reduce or mitigate the permanent hearing damage due noise are evolved 55 14-Jul-12

56 References Scott-Browns Otorhinolaryngology, editor Michael Gleeson, 7 th edition. Otolaryngology Head and Neck Surgery, editor Charles W. Cummings, 4 th edition. Ballengers Otorhinolaryngology Head and Neck Surgery, editor James B. Snow Jr, John Jacob Ballenger, 16 th edition. 56

57 Thank You For more ENT presentations, visit 14-Jul-12www.nayyarENT.com57

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