Presentation on theme: "Free OnDemand Webinar “At Natural Medicine Seminars, we would like to continually encourage and challenge our students in becoming the best diagnosticians."— Presentation transcript:
1Free OnDemand Webinar“At Natural Medicine Seminars, we would like to continually encourage and challenge our students in becoming the best diagnosticians through advanced training.Please enjoy this complimentary webinar.”Dr. Patrick GarrettThis is part one of a two part series. Part one – HeartPart two – Lungs
2Advanced Diagnostics Introduction The Cardiac Exam The Pulmonary Exam Normal Heart SoundsAbnormal Heart SoundsThe Pulmonary ExamNormal Lung SoundsAbnormal Lung Sounds
3Introduction Why bother listening to the heart? Auscultation of the heart is an essential part of even a cursory cardiac exam. By listening to the heart you can gather information about the: 1) Rate of the heart2) Rhythm of the heart3) Valvular function (e.g. stenosis, regurgitation, insufficiency)4) Anatomical defects (e.g. atrial septal defects -ASD, ventricular septal defect -VSD, ventricular hypertrophy)
4The Cardiac Exam Why bother listening to the heart? Auscultation of the heart is an essential part of even a cursory cardiac exam. By listening to the heart you can gather information about the: 1) Rate of the heart2) Rhythm of the heart3) Valvular function (e.g. stenosis, regurgitation, insufficiency)4) Anatomical defects (e.g. atrial septal defects -ASD, ventricular septal defect -VSD, ventricular hypertrophy)
5The Cardiac Exam Where to Listen and Why #1 Start with the Tricuspid region located between the 3rd, 4th, 5th, and 6th intercostal spaces at the left sternal border (LLSB)
6The Cardiac Exam Where to Listen and Why #2 Move to the Pulmonary region located between the 2nd and 3rd intercostal spaces at the left sternal border (LUSB)
7The Cardiac Exam Where to Listen and Why #3 Then to the Mitral region is located near the apex of the heard between the 5th and 6th intercostal spaces in the midclavicular line
8The Cardiac Exam Where to Listen and Why #4 End with the Aortic region located between the 2nd and 3rd intercostal spaces at the right sternal border (RUSB)
10The Cardiac ExamHint:Here is a mnemonic to help remember the different valve locations.“A PeT Monkey”Starting from theRUSB = A (Aortic Valve)LUSB = Pet (Pulmonic Valve)LLSB = PeT (Tricuspid Valve)Apex = Monkey (Mitral Valve)Don’t forget both of the carotids!
11The Cardiac ExamHint:Here is a mnemonic to help remember each of the atrial-ventricular valve locations.“LAB RAT”Left AV valve = (Bicuspid)Right AV valve = (Tricuspid)
12Additional Parts to the a complete cardiac examination The Cardiac ExamAdditional Parts to the a complete cardiac examinationAfter the initial examination in the supine positions, several additional maneuvers should be accomplished in the thorough cardiac exam.
13Additional Parts to the a complete cardiac examination The Cardiac ExamAdditional Parts to the a complete cardiac examinationFirst: Instruct the patient to turn onto their left side (left decubitus position) and listen with the bell of the stethoscope at the apex for mitral stenosis (This will be a low pitched diastolic murmur).
14Additional Parts to the a complete cardiac examination The Cardiac ExamAdditional Parts to the a complete cardiac examinationSecond: Instruct the patient to sit upright and re-examine the 4 valvular regions, again with the diaphragm of the stethoscope.
15The Cardiac Exam If a murmur is heard, further exam may be useful ManeuverRt SidedLt SidedTR / PSASMRHypertrophic CardiomyopathyChange with RespirationInspirationIncreasedDecrease or no changeTo decrease flowValsalva ManeuverDecreaseIncreaseSquat to StandVariableTo increase flowLeg ElevationNo DecreaseHand GripStand to Squat
16Additional Parts to the a complete cardiac examination The Cardiac ExamAdditional Parts to the a complete cardiac examinationLast: Instruct the patient to lean forward, exhale, and hold their breath. This Valsalva Manuever will increase the blood pressure and will help identify any underlying murmur.Listen with the diaphragm between the second and third intercostal spaces at the right sternal (aortic) and left sternal (pulmonic) areas for aortic regurgitation.
17Normal Heart SoundsThe normal heart sounds are often described as sounding like “lub dub”.Lub - The first heart sound is caused by vibrations arising from closure of both of the atrial-ventricular valves (mitral [bicuspid] and tricuspid). It coincides with the beginning of ventricular systole and so each first sound comes at the beginning of the pulse wave.Dub - The second heart sound is softer, shorter and of higher frequency and is caused by closure of the semi-lunar valves (aortic and pulmonary). It coincides with the end of ventricular systole and so occurs at the end of each pulse wave.Clinical Pearl: Because of their relationship to the pulse wave it is useful to listen to the heart sounds while feeling the pulse.
18Abnormal SoundsThe presence of a murmur indicates either increased or turbulent blood flow.Turbulent flow may arise because of abnormal flow across a valve or as a result of an abnormal communication between the chambers of the heart / great vessels.
19When are murmurs considered “normal”? Abnormal SoundsWhen are murmurs considered “normal”?Increased flow across a normal valve may occur in high output states, such as pregnancy, severe anemia, or associated with a significant pyrexia.
20Abnormal SoundsIn the event that you hear a murmur, your description and documentation should include these 4 properties of an “abnormal” heart sound:LOCATIONTIMINGGRADEQUALITY
21Abnormal SoundsLOCATION - Knowing the location of the murmur will help determine the most likely etiology, diagnostic testing, and follow up. Note any radiation of the sound. (i.e. Aortic will radiate into the carotids)*Location of maximal intensity (i.e. …between the 2nd and 3rd intercostal spaces at the right sternal border [2-3 ICS / URSB] with radiation into right carotid)
22Location of Maximal Intensity Abnormal SoundsRadiationLocation of Maximal IntensityRadiationTypical for2nd right intercostal spaceRight carotid arteryAortic stenosis5th or 6th left intercostal spaceLeft anterior axillary line, left axillaMitral regurgitation (including mitral regurgitation due to mitral valve prolapse)Left axilla Lower left sternal borderLRSB, Epigastrium, 5th ICS mid left thoraxTricuspid regurgitation5th left intercostal space mid- left thoraxLower left sternal borderHypertrophic cardiomyopathy
23Abnormal SoundsTIMING – Knowing the timing of the heart sound will narrow down the most likely cause.*Note which part of the diastolic systolic phase the sound occurs. (i.e. early diastolic, pan systolic, etc)
24Determining the timing Abnormal SoundsDetermining the timingEarly Systolic – The murmur will over shadow the lub. This will sound like : Murmur-Dub Mid Systolic – The murmur will occur after the lub and prior to the dub. This will sound like : Lub -MurmurDubLate Systolic – The murmur will over shadow the dub. This will sound like : Dub-MurmurHoloSystolic – The murmur will over shadow both of the lub and the dub. This will sound like : Murmur-Murmur
25Abnormal Sounds Murmur Timing and other Descriptions Description Possible DiagnosisSystolic ejection murmurNormal, pulmonic, or aortic stenosisEarly diastolic murmurAortic regurgitationEjection SoundAortic valve diseasePansystolic murmurTricuspid or mitral regurgitationLate diastolic murmurTricuspid or mitral stenosisSystolic click with late systolic murmurMitral valve prolapseOpening snap with diastolic rumble murmurMitral stenosisS3Normal in children and occurs in heart failureS4Physiological and in various diseases
26Abnormal SoundsGRADING – Every murmur should be graded according to its intensity. This will differentiate a light murmur from a harsh murmur.
27Abnormal Sounds Murmur Grades Grade Volume Thrill 1 Sound is very faint, only heard in ideal circumstancesNo2Sound is loud enough to be generally heard3Sound is louder then grade 2 but no thrill4Sound is louder then grade 3 with a thrillYes5Sound is heard with stethoscope partially off chest6Sound is heard with stethoscope entirely off chest
28Abnormal SoundsThrills – Typically a murmur that is loud enough to be graded a 4, 5, or 6 will be associated with a palpable thrill.To check for a thrill, place the palm of your right left hand across the base of the heart. You should be able to feel the vibration transfer through the chest wall.
29Abnormal Sounds QUALITY – The quality and pitch of the heart sound. *This may include terms such as harsh, musical, rumble, blowing. Use the modifier – High, Medium, Low and note any changes in body position or with respiration.
30Abnormal Sounds Quality If there is a musical “honk” or “coo”, it is usually due to an Aortic Stenosis.If the murmur creates a “harsh” sound it is also usually due to an Aortic Stenosis.
31Clinical ExamplesIf the sound is normal, document the rate and rhythm and the absence of an audible murmur.If the sound is abnormal, document rate, rhythm, location of murmur, timing, grade, and quality. If the sound radiates, make sure to note where.
32Etiology of Pediatric Murmurs 30% Innocent Murmur 20% Ventricular Septal Defect, VSD 11% Aortic Stenosis AS /AI 9% Mitral Valve Prolapse MVP/Mitral Regurgitation MR 7% Sub Aortic Stenosis (HOCM) 7% Pulmonary Stenosis, PS 5% Tetralogy of Fallot, ToF 9% otherFinley et al. Assessing children's heart sounds at a distance with digital recordings Pediatrics Dec;118(6):
33Normal Heart SoundClick on the speaker to hear the heart soundNote the Lub Dub soundNow go to the next page and listen to the most common abnormal sounds you will hear in adults.
34Mitral Regurgitation Mitral Stenosis Aortic Regurgitation Click on the speaker to hear the heart soundMitral StenosisAortic RegurgitationAortic StenosisPulmonary Stenosis
37Normal Heart Sounds S1S1 - The first heart tone, or S1, forms the "lub" of "lub-dub" and is composed of components Mitral and Tricuspid valves. It is caused by the sudden block of reverse blood flow due to closure of the atrioventricular valves at the beginning of ventricular contraction, or systole.When the pressure in the ventricles rises above the pressure in the atria, venous blood flow entering the ventricles is pushed back toward the atria, catching the valve leaflets, closing the inlet valves and preventing regurgitation of blood from the ventricles back into the atria. The S1 sound results from reverberation within the blood associated with the sudden block of flow reversal by the valves.
38Normal Heart Sounds S2S2 - The second heart tone, or S2, forms the "dub" of "lub-dub" and is composed of components Aortic and Pulmonic valves. It is caused by the sudden block of reversing blood flow due to closure of the aortic valve and pulmonary valve at the end of ventricular systole, i.e beginning of ventricular diastole. As the left ventricle empties, its pressure falls below the pressure in the aorta, aortic blood flow quickly reverses back toward the left ventricle, catching the aortic valve leaflets and is stopped by aortic (outlet) valve closure. Similarly, as the pressure in the right ventricle falls below the pressure in the pulmonary artery, the pulmonary (outlet) valve closes. The S2 sound results from reverberation within the blood associated with the sudden block of flow reversal.
39Mitral RegurgitationMitral regurgitation (MR), also known as mitral insufficiency or mitral incompetence, is a valvular heart disease which consists of abnormal leaking of blood through the mitral valve, from the left ventricle into the left atrium of the heart
40Mitral StenosisMitral Stenosis is a valvular heart disease characterized by the narrowing of the orifice of the mitral valve of the heart.With mitral stenosis, the valve does not open completely, and to transport the same amount of blood the left atrium needs a higher pressure than normal to overcome the increased gradient.
41Aortic RegurgitationAortic regurgitation (AR), also known as aortic insufficiency or aortic incompetence, is a valvular heart disease which consists of abnormal leaking of blood through the aortic valve, from the aorta into the right ventricle of the heart
42Aortic StenosisAortic Stenosis is a valvular heart disease characterized by the narrowing of the orifice of the aortic semilunar valve of the heart.With Aortic stenosis, the valve does not open completely, and to transport the same amount of blood the right ventricle needs a higher pressure than normal to overcome the increased gradient.This will eventually lead to right ventricular hypertrophy.
43Pulmonary StenosisPulmonary Stenosis is a valvular heart disease characterized by the narrowing of the orifice of the pulmonic semilunar valve of the heart.With pulmonary stenosis, the valve does not open completely, and to transport the same amount of blood the left ventricle needs a higher pressure than normal to overcome the increased gradient.This will eventually lead to left ventricular hypertrophy.
44Tricuspid Regurgitation Tricuspid regurgitation (TR), also known as tricuspid insufficiency or tricuspid incompetence, is a valvular heart disease which consists of abnormal leaking of blood through the tricuspid valve, from the right ventricle into the right atrium of the heart
45VSDA ventricular septal defect (VSD) is a defect in the ventricular septum, the wall dividing the left and right ventricles of the heart.This allows oxygenated from the left ventricle to mix with de-oxygenated blood from the right ventricle.
46PericarditisPericarditis is an inflammation of the pericardium (the fibrous sac surrounding the heart).Chest pain, radiating to the back and relieved by sitting up forward and worsened by lying down, is the classical presentation.Other symptoms of pericarditis may include dry cough, fever, fatigue, and anxiety. Pericarditis can be misdiagnosed as myocardial infarction (heart attack), and vice versa.
47Mid-Systolic ClickMid-systolic clicks are due to blood flow through the semilunar valves.They occur at the start of blood ejection — which starts after S1 — and ends with the cessation of the blood flow — which is before S2. Therefore, the onset of a midsystolic ejection murmur is separated from S1 by the isovolumic contraction phase; the cessation of the murmur and the S2 interval is the aortic or pulmonary hangout time. The resultant configuration of this murmur is a crescendo-decrescendo murmur.Causes of midsystolic ejection murmurs include outflow obstruction, increased flow through normal semilunar valves, dilation of aortic root or pulmonary trunk, or structural changes in the semilunar valves without obstruction.
48Opening SnapThe Opening Snap in addition to a typical murmur indicates the murmur is due to mitral stenosis and not a flow rumble across a non-stenotic valve.The timing of the Opening Snap has been suggested as a gauge of the severity of the stenosis.
49Third Heart Sound (S3)Rarely, there may be a third heart sound also called a protodiastolic gallop, ventricular gallop, or informally the "Kentucky" gallop as an onomatopoeic reference to the rhythm and stress of S1, S2, and S3 together (S1=ken; S2=tuc; S3=ky).It occurs at the beginning of diastole after S2 and is lower in pitch than S1 or S2 as it is not of valvular origin.The third heart sound is benign in youth and some trained athletes, but if it re-emerges later in life it may signal cardiac problems like a failing left ventricle as in dilated congestive heart failure (CHF).S3 is thought to be caused by the oscillation of blood back and forth between the walls of the ventricles initiated by inrushing blood from the atria.The reason the third heart sound does not occur until the middle third of diastole is probably because during the early part of diastole, the ventricles are not filled sufficiently to create enough tension for reverberation. It may also be a result of tensing of the chordae tendineae during rapid filling and expansion of the ventricle.
50Tumor PlopA third heart sound can also be simulated by a myxoma of the left atrium. This type of third heart sound is often referred to as a "tumor plop".The "tumor plop" is caused by the gelatinous tumor, which is attached by a stalk to the atrial septum, plunging into the ventricle and pushing the blood ahead of it like a syringe plunger.
51Fourth Heart Sound (S4)The rare fourth heart sound is sometimes audible in healthy children and again in trained athletes, but when audible in an adult is called a presystolic gallop or atrial gallop.This gallop is produced by the sound of blood being forced into a stiff / hypertrophic ventricle.It is a sign of a pathologic state, usually a failing left ventricle.The sound occurs just after atrial contraction ("atrial kick") at the end of diastole and immediately before S1, producing a rhythm sometimes referred to as the “Tennessee” gallop where S4 represents the "tenn-" syllable. The combined presence of S3 and S4 is a quadruple gallop. At rapid heart rates, S3 and S4 may merge to produce a summation gallop.
52Physiological SplitDuring inspiration, the increased negative intrathorasic pressure which allows lung expansion also induces both increased blood return from the body into the right ventricle and simultaneous reduced blood volume return from the lungs into the left ventricle. Because of the increased blood volume in the right atrium, the pulmonary valve stays open longer during ventricular systole whereas the aortic valve closes slightly earlier due to slightly reduced left ventricular volume.Thus the Pulmonary Valve component of S2 is delayed relative to the Aortic valve component. This delay in P2 versus A2 is heard as a slight broadening or even "splitting" of the second heart sound, though usually only in the pulmonic area of the chest because the P2 is soft and not heard in other areas.During expiration, the less negative (than during inspiration) intrathoracic pressure no longer increases blood return to the right ventricle versus the left ventricle, the right ventricle volume in no longer increased, the pulmonary valve closes earlier, Pulmonic valve occurs earlier and overlaps Aortic valve.It is physiological to hear a "splitting" of the second heart tone in younger people, during inspiration and in the "pulmonary area", i.e. the 2nd ICS (intercostal space) at the left edge of the sternum. During expiration the interval between the two components normally shortens and the S2 sounds become merged.
53Wide SplitA bundle branch block will produce continuous splitting but the degree of splitting will still vary with respiration.If splitting does not vary with inspiration, it is termed a "fixed split S2" and is usually due to an atrial septal defect (ASD) or ventricular septal defect (VSD). The ASD or VSD creates a left to right shunt that increases the blood flow to the right side of the heart, thereby causing the pulmonic valve to close later than the aortic valve independent of inspiration/expiration.Reverse splitting indicates pathology. Aortic stenosis, hypertrophic cardiomyopathy, left bundle branch block, and a ventricular pacemaker could all cause a reverse splitting of the second heart sound.
54Diagnosing Murmurs Accuracy in Diagnosis of Systolic Murmurs Maneuvers This study looks at the sensitivity, specificity and predictive value of physical exam maneuvers that historically have been used in diagnosing systolic murmurs.Two cardiologist independently examined 50 patients age 6 to 85 years of age ( mean age 45) with a systolic murmur I / VI or greater. The cardiologists were separated from the patients by a partition. An independent examiner instructed the patients in the maneuvers and supervised the implementation of them.Sapira, JD. The art and science of bedside diagnosis. (Urban & Schwarzenberg, Baltimore-Munich) 1990.
55Results for Systolic Murmur Maneuvers--result in increased murmurMurmurSens(%)Spec(%)LR+LR-InspirationRight-sided100888.3ValsalvaHCM659616.250.36Squat to stand95845.90.06HandgripMitral regurgitation, VSD68928.50.35Transient arterial occlusion78-Maneuvers--result in decreased murmurstand to squatLeg elevation859110.630.16753.40.2
57Cardiac References"The Cardiovascular System." Bates, B. A Guide to Physical Examination and History Taking. 9h EdAronow, WS, and I Kronzon. Correlation of prevalence and severity of valvular aortic stenosis determined by continuous-wave Doppler echocardiography with physical signs of aortic stenosis in patients 62 to 100 years with aortic systolic ejection murmurs. Am J Cardiol (4):Badgett RG, CR Lucey, CD Mulrow, DL Simel, D Rennie. Can the clinical examination diagnose left sided heart failure in adults? JAMA 1997; 277:"Choudhry NK, Etchells EE. Does This Patient Have Aortic Regurgitation? JAMA. 1999; 281:2231.Etchells E, C Bell, K Robb, DL Simel, and D Rennie. Does this patient have an abnormal systolic murmur? JAMA 1997; 277:Fye, WB. Disorders of the heartbeat: A historical overview from antiquity to the mid-20th century. Am J Cardiol 1993; 72:Gaeschke, R, GH Guyatt, DL Sackett for the Evidence-Based Medicine Working Group. Users' guides to the medical literature. VI. How to use an article about a diagnostic test. A: Are the results of the study valid? JAMA :McGee S. Evidence based physical diagnosis. WB Saunders CoMcMichael, J. History of atrial fibrillation : Harvey - de Senac - Laennec. Br Heart J 1982; 48:Sapira, JD. The art and science of bedside diagnosis. (Urban & Schwarzenberg, Baltimore-Munich) 1990.
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