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Fig. 6. VitabridC12 inhibited cutaneous inflammation via the nuclear factor-κB signaling pathway. Protein extracts were prepared, and western blot analysis.

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Presentation on theme: "Fig. 6. VitabridC12 inhibited cutaneous inflammation via the nuclear factor-κB signaling pathway. Protein extracts were prepared, and western blot analysis."— Presentation transcript:

1 Fig. 6. VitabridC12 inhibited cutaneous inflammation via the nuclear factor-κB signaling pathway. Protein extracts were prepared, and western blot analysis was performed with antibodies specific for the molecules indicated. pERK, p-p38, pMEK, p-p65, and COX-2 expression levels increased in psoriatic skin. The protein levels of pERK (44, 48 kDa), p-p38 (38 kDa), pMEK (44, 45 kDa), p-p65 (65 kDa), and COX-2 (75 kDa) decreased after treatment with VitabridC12. ERK: extracellular signal-regulated kinase, MEK: mitogen-activated protein kinase/ERK kinase, GAPDH: glyceraldehyde-3-phosphate dehydrogenase. Fig. 6. VitabridC12 inhibited cutaneous inflammation via the nuclear factor-κB signaling pathway. Protein extracts were prepared, and western blot analysis was performed with antibodies specific for the molecules indicated. pERK, p-p38, pMEK, p-p65, and COX-2 expression levels increased in psoriatic skin. The protein levels of pERK (44, 48 kDa), p-p38 (38 kDa), pMEK… Ann Dermatol Oct;29(5):

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